The anemia improved spontaneously dependent on the favism in patient with G6PD deficiency

Glucose-6-phosphate dehydrogenase (G6PD) enzyme deficiency is the most common enzyme deficiency and the G6PD enzyme is a key enzyme catalyzing the first step of the pentose phosphate pathway. Clinically, G6PD enzyme deficiency causes a series of disorders such as newborn hyperbilirubinemia, acute or chronic hemolysis. The G6PD enzyme catalyzes the first step of the pentose phosphate pathway and leading to the formation of antioxidants that protect cells from oxidative damage. In patients with G6PD enzyme deficiency some drugs, some metabolic conditions and some infections cause oxidative stress-related hemolysis in red blood cells. In this article, we presented a case in which the anemia spontaneously regresses after the exposure of the pine to the anemia due to haemolysis after the eating of the pod in our case with G6PD enzyme deficiency and sickle cell anemia carrier