Plasticity at Thalamo-amygdala Synapses Regulates Cocaine-Cue Memory Formation and Extinction

？ Cocaine-cue memory formation strengthens thalamo-amygdala, not cortico-amygdala synapses ？ Cocaine-cue extinction reduces relapse and depotentiates thalamo-amygdala synapses ？ Medial geniculate thalamic (MGN) projections to amygdala (LA) regulate cocaine memory ？ Optogenetic LTD at MGN-LA synapses is sufficient to block cocaine-cue-induced relaps