Not all arrestins are created equal: Therapeutic implications of the functional diversity of the β-arrestins in the heart

DOI: 10.4330/wjc.v11.i2.47

Keywords: Adverse remodeling, β-arrestin, Biased signaling, Cardiac myocyte, Cardiac fibroblast, contractility, Functional divergence, G protein-coupled receptor, Heart failure, Hormone, Myocardial infarction, Signal transducer

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