The novel mechanism of anticancer effect on gastric cancer through inducing G0/G1 cell cycle arrest and caspase-dependent apoptosis in vitro and in vivo by methionine enkephalin

DOI: 10.2147/CMAR.S178343

Keywords: MENK, GC cells, OGFr, cell cycle, apoptosis

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Abstract:

Gastric cancer (GC) is the second cause of cancer-related deaths. Methionine enkephalin (MENK), an endogenous opioid peptide, has immunological and antitumor activity

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