The PERK Branch of the Unfolded Protein Response Promotes DLL4 Expression by Activating an Alternative Translation Mechanism

DOI: 10.3390/cancers11020142

Keywords: DLL4 (delta like ligand 4), angiogenesis, IRES (internal ribosome entry site), hypoxia, endoplasmic reticulum stress, UPR (unfolded protein response), PERK (PKR-Like endoplasmic reticulum kinase)

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