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Abstract Recent puberty-timing measurements have confirmed the continuation of accelerated sexual development know as precocious puberty [8, 4]. This shift in reproductive health has unknown health consequences. Current literature speculates that negative consequences of precocious puberty exist in a wide range from social angst to an increased risk of tumor development and cancers. Onset age of puberty is multifactorial, but there is strong evidence to suggest that an external factor may be impacting reproductive timing. Genetics, the environment, dietary choices, obesity, and estrogen disrupting compounds have all been purposed as potential causal agents. The rationale behind and practically of each of these potential determinants was evaluated to better understand and illustrate the connection to external sources and the problem of precocious puberty. Recognizing the presence and dangers of these different factors is critical in implementing methods and shifting behavioral patterns to decrease the risk of precocious puberty