1. Lozano R, Naghavi M, Foreman K. Global and regional mortality from 235 causes of death for 20 age groups in 1990 and 2010: a systematic analysis for the Global Burden of Disease Study 2010. Lancet, 2012, 380(9859): 2095-2128.
3. Vogl T, Leukert N, Barczyk K, et al. Biophysical characterization of S100A8 and S100A9 in the absence and presence of bivalent cations. Biochm Biophys Acta, 2006, 1763(11): 1298-1306.
[4]
4. Roth J, Vogl T, Sorg C, et al. Phagocyte-specific S100 proteins: a novel group of proinflammatory molecules. Trends Immunol, 2003, 24(4): 155-158.
[5]
5. Vogl T, Gharibyan AL, Morozova-Roche LA. Pro-inflammatory S100A8 and S100A9 proteins: self-assembly into multifunctional native and amyloid complexes. Int J Mol Sci, 2012, 13 (3): 2893-2917.
[6]
6. Halayko AJ, Sharma P. S100A8/A9 as a Pro-inflammatory Cytokine in Obstructive Airway Disease Via the Multi-Ligand Receptor, RAGE. Anti-Inflammatory and Anti-Allergy Agents in Medicinal Chemistry, 2009, 8(4): 306-317.
[7]
7. Rammes A, Roth J, Goebeler M, et al. Myeloid-related protein (MRP) 8 and MRP14, calcium-binding proteins of the S100 family, are secreted by activated monocytes via a novel, tubulin-dependent pathway. J Biol Chem, 1997, 272(14): 9496-9502.
[8]
8. Merkel D, Rist W, Seither P, et al. Proteomic study of human bronchoalveolar lavage fluids from smokers with chronic obstructive pulmonary disease by combining surface-enhanced laser desorption/ionization-mass spectrometry profiling with mass spectrometric protein identification. Proteomics, 2005, 5(11): 2972-2980.
[9]
9. RivaM, Kallberg E, Bjork P, et al. Induction of nuclear factorkappaB responses by the S100A9 protein is Toll-like receptor-4- dependent.Immunology, 2012, 137(2): 172-182.
