1. ?Drummond GS, Kappas A. Chemoprevention of neonatal jaundice: potency of tin-protoporphyrin in an animal model. Science, 1982, 217(4566): 1250-1252.
3. Cisowski J, Loboda A, Jozkowicz A, et al. Role of hemeoxygenase-1 in hydrogen peroxide-induced VEGF synthesis: effect of HO-1 knockout. Biochem Biophys Res Commun, 2005, 326(3): 670-676.
[4]
4. Ferrando M, Gueron G, Elguero B, Giudice J, et al. Hemeoxygenase-1(HO-1) challenges the angiogenic switch in prostate cancer. Angiogenesis, 2011, 14(4): 467-479.
[5]
5. Wu M, Huang J, Xu S, et al. Hemeoxygenase-1 delays programmed cell death in wheat aleurone layers by modulation of hydrogen peroxide metabolism. J Exp Bot, 2011, 62(1): 235-248.
[6]
6. Bao W, Song F, Li X, et al. Plasma heme oxygenase-1 concentration is elevated in individuals with type 2 diabetes mellitus. PLoS One, 2010, 5(8): e12371.
[7]
7. Miyake M, Fujimoto K, Anai S, et al. Heme oxygenase-1promotes angiogenesis in urothelial carcinoma of the urinary bladder. Oncol Rep, 2011, 25(3): 653-660.
[8]
8. Jee SH, AOhrr H, Sull JW, et al. Fasting serum glucose level and cancer risk in Korean men and women. JAMA, 2005, 293(2): 194-202.
[9]
10. Mateo I, Infante J, Sanchez-Juan P, et al. Serum heme oxygenase-1 levels are increased in Parkinson’s disease but not in Alzheimer’s disease. Acta Neurol Scand, 2010, 121(2): 136-138.
[10]
11. Miyazaki T, Kirino Y, Takeno M, et al. Serum HO-1 is useful to make differential diagnosis of secondary hemophagocytic syndrome from other similar hematological conditions. Int J Hematol, 2010, 91(2): 229-237.
[11]
12. Susztak K, Raff AC, Schiffer M, et al. Glucose-induced reactive oxygen species cause apoptosis of podocytes and podocyte depletion at the onset of diabetic nephropathy. Diabetes, 2006, 55(1): 225-233.
[12]
15. Zuo L, Youtz DJ, Wold LE. Particulate matter exposure exacerbates high glucose-induced cardiomyocyte dysfunction through ROS generation. PLoS One, 2011, 6(8): e23116.
19. Singh R, Barden A, Mori T, et al. Advanced glycation endproducts: a review. Diabetologia, 2001, 44(2): 129-146.
[15]
9. Sato T, Takeno M, Honma K, et al. Heme oxygenase-1, a potential biomarker of chronic silicosis, attenuates silica-induced lung injury. Am J Respir Crit Care Med, 2006, 174(8): 906-914.
[16]
13. Wu WS. The signaling mechanism of ROS in tumor progression. Cancer Metastasis Rev, 2006, 25(4): 695-705.
[17]
14. Li H, van Berlo D, Shi T, et al. Curcumin protects against cytotoxic and inflammatory effects of quartz particles but causes oxidative DNA damage in a rat lung epithelial cell line. Toxicol Appl Pharmacol, 2008, 227(1): 115-124.
[18]
17. Bonnefont-Rousselot D, Bastard JP, Jaudon MC, et al. Consequences of the diabetic status on the oxidant/antioxidant balance. Diabetes Metab, 2000, 26(3): 163-176.
[19]
18. Inoguchi T, Li P, Umeda F, et al. High glucose level and free fatty acid stimulate reactive oxygen species production throughprotein kinase C-dependent activation of NAD(P)H oxidase in cultured vascular cells. Diabetes, 2000, 49(11): 1939-1945.
[20]
20. Alisson-Silva F, Freire-de-Lima L, Donadio JL, et al. Increase of O-glycosylated oncofetal fibronectin in high glucose-induced epithelial-mesenchymal transition of cultured human epithelial cells. PLoS One, 2013, 8(4): e60471.
22. Li W, Ma Q, Li J, et al. Hyperglycemia enhances the invasive and migratory activity of pancreatic cancer cells via hydrogen peroxide. Oncol Rep, 2011, 25(5): 1279-1287.