6. Ohmori Y, Akaishi S, Ogawa R, et al. The analysisof keloid favorite site. The 4th Japan Scar Workshop, Tokyo, Japan, 2009.
[2]
7. Parsons M, Kessler E, Laurent GJ, et al. Mechanical load enhances procollagen processing in dermal fibroblasts by regulating levels of procollagen C-proteinase. Exp Cell Res, 1999, 252(2):319-331.
18. Wang Y, McNiven MA. Invasive matrix degradation at focal adhesions occurs via protease recruitment by a FAK-p130Cas complex. J Cell Biol, 2012, 196(3):375-385.
[5]
19. Jano?tiak R, Brábek J, Auernheimer V, et al. CAS directly interacts with vinculin to control mechanosensing and focal adhesion dynamics. Cell Mol Life Sci, 2014, 71(4):727-744.
[6]
20. Hannafin JA, Attia EA, Henshaw R, et al. Effect of cyclic strain and plating matrix on cell proliferation and integrin expression by ligament fibroblasts. J Orthop Res, 2006, 24(2):149-158.
1. Mustoe TA, Cooter RD, Gold MH, et al. International clinical recommendations on scar management. Plast Reconstr Surg, 2002, 110(2):560-571.
[9]
2. Yagmur C, Akaishi S, Ogawa R, et al. Mechanical receptor-related mechanisms in scar management:a review and hypothesis. Plast Reconstr Surg, 2010, 126(2):426-434.
[10]
3. Derderian CA, Bastidas N, Lerman OZ, et al. Mechanical strain alters gene expression in an in vitro model of hypertrophic scarring. Ann Plast Surg, 2005, 55(1):69-75.
[11]
4. Ogawa R. Keloid and hypertrophic scarring may result from a mechanoreceptor or mechanosensitive nociceptor disorder. Med Hypotheses, 2008, 71(4):493-500.
[12]
5. Ogawa R. Mechanobiology of scarring. Wound Repair Regen, 2011, 19 Suppl 1:s2-9.
11. Syedain ZH, Tranquillo RT. TGF-β1 diminishes collagen production during long-term cyclic stretching of engineered connective tissue:implication of decreased ERK signaling. J Biomech, 2011, 44(5):848-855.
[16]
12. Wipff PJ, Rifkin DB, Meister JJ, et al. Myofibroblast contraction activates latent TGF-beta1 from the extracellular matrix. J Cell Biol, 2007, 179(6):1311-1323.
[17]
13. Wang Z, Fong KD, Phan TT, et al. Increased transcriptional response to mechanical strain in keloid fibroblasts due to increased focal adhesion complex formation. J Cell Physiol, 2006, 206(2):510-517.
[18]
14. Kanazawa Y, Nomura J, Yoshimoto S, et al. Cyclical cell stretching of skin-derived fibroblasts downregulates connective tissue growth factor (CTGF) production. Connect Tissue Res, 2009, 50(5):323-329.
[19]
15. Baker EL, Zaman MH. The biomechanical integrin. J Biomech, 2010, 43(1):38-44.
[20]
17. Cheng M, Guan X, Li H, et al. Shear stress regulates late EPC differentiation via mechanosensitive molecule-mediated cytoskeletal rearrangement. PLoS One, 2013, 8(7):e67675.
[21]
21. Demou ZN. Gene expression profiles in 3D tumor analogs indicate compressive strain differentially enhances metastatic potential. Ann Biomed Eng, 2010, 38(11):3509-3520.