心肌肥厚和心力衰竭时左心室收缩力及钙瞬变的变化 Changes of the Contraction and Calcium Transients in Myocytes of Left Ventricle in Patients with Cardiac Hypertrophy and Heart Failure
Lopez-Lopez JR,Shacklock PS,Balke CW,et al.Local,stochastic release of Ca2+in voltage-clamped rat heart cells:Visualization with confocal microscopy[J].J Physiol,1994,480(Pt 1):21-29.
[2]
Yao A,Matsui H,Spitzer KW,et al.Sarcoplasmic reticulum and Na+/Ca2+exchanger function during early and late relaxation in ventricular myocytes[J].Am J Physiol,1997,273(6Pt 2):H2 765-H2 773.
[3]
Rebbeck RT,Karunasekara Y,Board PG,et al.Skeletal muscle excitation-contraction coupling:Who are the dancing partners[J]?Int J Biochem Cell Biol,2014,48:28-38.
[4]
Horiuchi-Hirose M,Kashihara T,Nakada T,et al.Decrease in the density of t-tubular l-type Ca2+channel currents in failing ventricular myocytes[J].Am J Physiol Heart Circ Physiol,2011,300(3):H978-H988.
[5]
Hu ST,Liu GS,Shen YF,et al.Defective Ca2+handling proteins regulation during heart failure[J].Physiol Res,2011,60(1):27-37.
Curran J,Tang L,Roof SR,et al.Nitric oxide-dependent activation of camkii increases diastolic sarcoplasmic reticulum calcium release in cardiac myocytes in response to adrenergic stimulation[J].PLoS One,2014,9(2):e87495.
[9]
Clay SA,Domeier TL,Hanft LM,et al.Elevated Ca2+transients and increased myofibrillar power generation cause cardiac hypercontractility in a model of noonan syndrome with multiple lentigines[J].Am J Physiol Heart Circ Physiol,2015,308(9):H1 086-H1 095.
[10]
Sullivan MJ,Knight JD,Higginbotham MB,et al.Relation between central and peripheral hemodynamics during exercise in patients with chronic heart failure.Muscle blood flow is reduced with maintenance of arterial perfusion pressure[J].Circulation,1989,80(4):769-781.
[11]
Ibrahim M,Navaratnarajah M,Siedlecka U,et al.Mechanical unloading reverses transverse tubule remodelling and normalises local calcium-induced calcium release in a rodent model of heart failure[J].Eur J Heart Fail,2012,14(6):571-580.