Sharov VS,Scheneich C.Oxidative Modification of Ca2+Channels,Ryanodine Receptors,and the Sarco/Endoplasmic Reticulum Ca2+-ATPase[J].Free Radical Effects on Membranes,2008,61:113-130.
[2]
Dilly KW,Rossow CF,Votaw VS,et al.Mechanisms underlying variations in excitation-contraction coupling across the mouse left ventricular free wall[J].J Physiol,2006,572:227-241.
[3]
Viola HM,Hool LC.Qo site of mitochondrial complex III is the source of increased superoxide after transient exposure to hydrogen peroxide[J].J Mol Cell Cardiol,2010,49(5):875-885.
[4]
Oda T,Yang Y,Uchinoumi H,et al.Oxidation of ryanodine receptor(RyR)and calmodulin enhance Ca release and pathologically alter,RyR structure and calmodulin affinity[J].J Mol Cell Cardiol,2015,85:240-248.
Lüscher TF.Ageing,inflammation,and oxidative stress:final common pathways of cardiovascular disease[J].Eur Heart J,2015,36(48):3 381-3 383.
[7]
Liao R,Podesser BK,Lim CC.The continuing evolution of the Langendorff and ejecting murine heart:new advances in cardiac phenotyping[J].Am J Physiol Heart Circ Physiol,2012,303(2):H156-H167.
[8]
Yan Y,Liu J,Wei CL.Bidirectional regulation of Ca2+sparks by mitochondria-derived reactive oxygen species in cardiac myocytes[J].Cardiovasc Res,2008,77(2):432-441.
[9]
Kuster GM,Lancel S,Zhang J.Redox-mediated reciprocal regulation of SERCA and Na+-Ca2+exchanger contributes to sarcoplasmic reticulum Ca2+depletion in cardiac myocytes[J].Free Radic Biol Med,2010,48(9):1 182-1 187.
[10]
Li W,Wang Y,Gao L.Resveratrol protects rabbit ventricular myocytes against oxidative stress-induced arrhythmogenic activity and Ca2+overload[J].Acta Pharmacol Sin,2013,34:1 164-1 173.