抑郁症患者血清hs-CRP水平变化及其对抗抑郁药疗效的预测作用
Serum level of hs-CRP in patients with depression and its predictive role for the efficiency of antidepressants

DOI: 10.6040/j.issn.1671-7554.0.2017.261

Keywords: 超敏C反应蛋白,度洛西汀,抑郁症,炎症,舍曲林,
Depression,High sensitivity C reactive protein,Duloxetine,Sertraline,Inflammation

Full-Text   Cite this paper   Add to My Lib

Abstract:

References

[1]	Valkanova V, Ebmeier KP, Allan CL. CRP, IL-6 and depression: a systematic review and meta-analysis of longitudinal studies[J]. J Affect Disord, 2013, 150(3): 736-744.
[2]	Copeland WE, Shanahan L, Worthman C, et al. Cumulative depression episodes predict later C-reactive protein levels: a prospective analysis[J]. Biol Psychiatry, 2012, 71(1): 15-21.
[3]	Mowla A, Dastgheib SA, Razeghian Jahromi L. Comparing the effects of sertraline with duloxetine for depression severity and symptoms: a double-blind, randomized controlled trial[J]. Clin Drug Investig, 2016, 36(7): 539-543.
[4]	Vogelzangs N, Duivis HE, Beekman AT, et al. Association of depressive disorders, depression characteristics and antidepressant medication with inflammation[J]. Transl Psychiatry, 2012, 2(2): e79. doi: 10.1038/tp.2012.8.
[5]	Liu Y, Ho RC, Mak A. Interleukin(IL)-6, tumour necrosis factor alpha(TNF-α)and soluble interleukin-2 receptors(sIL-2R)are elevated in patients with major depressive disorder: a meta-analysis and meta-regression[J]. J Affect Disord, 2012, 139(3): 230-239.
[6]	Papakostas GI, Shelton RC, Kinrys G, et al. Assessment of a multi-assay, serum-based biological diagnostic test for major depressive disorder: a pilot and replication study[J]. Mol Psychiatry, 2013, 18(3): 332-339.
[7]	Lopresti AL, Maker GL, Hood SD, et al. A review of peripheral biomarkers in major depression: the potential of inflammatory and oxidative stress biomarkers[J]. Prog Neuropsychopharmacol Biol Psychiatry, 2014, 48(1433): 102-111.
[8]	Dieperink E, Willenbring M, Ho SB. Neuropsychiatric symptoms associated with hepatitis C and interferon alpha: a review[J]. Am J Psychiatry, 2000, 157(6): 867-876.
[9]	Raison CL, Rutherford RE, Woolwine BJ, et al. A randomized controlled trail of the tumor necrosis factor antagonist infliximab for treatment-resistant depression: the role of baseline inflammatory biomarkers[J]. JAMA Psychiatry, 2013, 70(1): 31-41.
[10]	Miller AH, Maletic V, Raison CL. Inflammation and its discontents: the role of cytokines in the pathophysiology of major depression[J]. Biol Psychiatry, 2009, 65(9): 732-741.
[11]	Blank T, Detje CN, Spie？ A, et al. Brain endothelial- and epithelial-specific interferon receptor chain 1 drives virus-induced sickness behavior and cognitive impairment[J]. Immunity, 2016, 44(4): 901-912.
[12]	Shelton RC, Claiborne J, Sidoryk-Wegrzynowicz M, et al. Altered expression of genes involved in inflammation and apoptosis in frontal cortex in major depression[J]. Mol Psychiatry, 2011, 16(7): 751-762.
[13]	Zalli A, Jovanova O, Hoogendijk WJ, et al. Low-grade inflammation predicts persistence of depressive symptoms[J]. Psychopharmacology(Berl), 2016, 233(9): 1669-1678.
[14]	Dowlati Y, Herrmann N, Swardfager W, et al. A meta-analysis of cytokines in major depression[J]. Biol Psychiatry, 2010, 67(5): 446-457.
[15]	张明园, 何燕玲. 精神科评定量表手册[M]. 2版. 长沙: 湖南科学技术出版社, 2015: 142-148.
[16]	Maes M. Evidence for an immune response in major depression: a review and hypothesis[J]. Prog Neuropsychopharmacol Biol Psychiatry, 1995, 19(1): 11-38.
[17]	Uher R, Tansey KE, Dew T, et al. An inflammatory biomarker as a differential predictor of outcome of depression treatment with escitalopram and nortriptyline[J]. Am J Psychiatry, 2014, 171(12): 1278- 1286.
[18]	Vreeburg SA, Hoogendijk WJ, van Pelt J, et al. Major depressive disorder and hypothalamic-pituitary-adrenal axis activity: results from a large cohort study[J]. Arch Gen Psychiatry, 2009, 66(6): 617-626.
[19]	Licht CM, de Geus EJ, Zitman FG,et al. Association between major depressive disorder and heart rate variability in the Netherlands Study of Depression and Anxiety(NESDA)[J]. Arch Gen Psychiatry, 2008, 65(12): 1358-1367.
[20]	Marsland AL, Gianaros PJ, Abramowitch SM, et al. Interleukin-6 covaries inversely with hippocampal grey matter volume in middle-aged adults[J]. Biol Psychiatry, 2008, 64(6): 484-490.
[21]	Khairova RA, Machado-Vieira R, Du J, et al. A potential role for pro-inflammatory cytokines in regulating synaptic plasticity in major depressive disorder[J]. Int J Neuropsychopharmacology, 2009, 12(4): 561.
[22]	Dantzer R, O’Connor JC, Freund GG, et al. From inflammation to sickness and depression: when the immune system subjugates the brain[J]. Nat Rev Neurosci, 2008, 9(1): 46-56.
[23]	Wium-Andersen MK, ？rsted DD, Nielsen SF, et al. Elevated C-reactive protein levels, psychological distress, and depression in 73131 individuals[J]. JAMA Psychiatry, 2013, 70(2): 176-184.
[24]	Almeida OP, Norman PE, Allcock R, et al. Polymorphisms of the CRP gene inhibit inflammatory response and increase susceptibility to depression: the Health in Men Study[J]. Int J Epidemiol, 2009, 38(4): 1049-1059.
[25]	Chavda N, Kantharia ND, Jaykaran. Effects of fluoxetine and escitalopram on C-reactive protein in patients of depression[J]. J Pharmacol Pharmacother, 2011, 2(1): 11-16.
[26]	Felger JC, Li Z, Haroon E, et al. Inflammation is associated with decreased functional connectivity within corticostriatal reward circuitry in depression[J]. Mol Psychiatry, 2016, 21(10): 1358-1365.

Full-Text