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非肽类Ang(1-7)受体激动剂AVE0991对大鼠糖尿病肾病的保护作用
Evaluation of the protective effect of AVE0991 on streptozotocin-induced diabetic nephropathy rats

DOI: 10.6040/j.issn.1671-7554.0.2015.1184

Keywords: 血管紧张素(1-7)受体激动剂,糖尿病肾病,AVE0991,血管紧张素1-7,Wistar,大鼠,
AVE0991
,Ang(1-7)receptor agonist,Diabetes nephropathy,Rats, Wistar,Ang(1-7)

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[1]  吴宏力, 鲁军, 佘星星, 等. 血管紧张素转换酶2-血管紧张素(1-7)-Mas轴的血压调控作用研究进展[J]. 中华高血压杂志, 2014, 22(6):528-532.
[2]  Liu CX, Hu Q, Wang Y, et al. Angiotensin-converting enzyme(ACE)2 overexpression ameliorates glomerular injury in a rat model of diabetic nephropathy: a comparison with ACE inhibition[J]. Mol Med, 2011, 17(1-2):59-69.
[3]  Mizuiri S, Aoki T, Hemmi H, et al. Urinary angiotensin-converting enzyme 2 in patients with CKD[J]. Nephrology(Carlton), 2011, 16(6):567-572.
[4]  Mori J, Patel VB, Ramprasath T, et al. Angiotensin 1-7 mediates renoprotection against diabetic nephropathy by reducing oxidative stress, inflammation, and lipotoxicity [J]. Am J Physiol Renal Physiol, 2014, 306(8):F812-821.
[5]  Wada J, Makino H. Inflammation and the pathogenesis of diabetic nephropathy [J]. Clin Sci(Lond), 2013, 124(3):139-152.
[6]  Tesch GH. Macrophages and diabetic nephropathy [J]. Semin Nephrol, 2010, 30(3):290-301.
[7]  Liu RM, Gaston Pravia KA. Oxidative stress and glutathione in TGFbeta-mediated fibrogenesis[J]. Free Radic Biol Med, 2010, 48(1):1-15.
[8]  Grace JA, Klein S, Herath CB, et al. Activation of the MAS receptor by angiotensin-(1-7)in the renin-angiotensin system mediates mesenteric vasodilatation in cirrhosis[J]. Gastroenterology, 2013, 145(4):874-884.
[9]  Kamiyama M, Urushihara M, Morikawa T, et al. Oxidative stress angiotensinogen renin-angiotensin system axis in patients with diabetic nephropathy [J]. Int J Mol Sci, 2013, 14(11):23045-23062.
[10]  Barroso LC, Silveira KD, Lima CX, et al. Renoprotective effects of AVE0991, a nonpeptide mas receptor agonist, in experimental acute renal injury[J]. Int J Hypertens, 2012, 2012:808726. doi: 10.1155/2012/808726. Epub 2012 Jan 29.
[11]  Passos-Silva DG, Verano-Braga T, Santos RA, et al. Angiotensin-(1-7): beyond the cardio-renal actions[J]. Clin Sci(Lond), 2013, 124(7):443-456.
[12]  Nguyen D, Ping F, Mu W, et al. Macrophage accumulation in human progressive diabetic nephropathy [J]. Nephrology(Carlton), 2006, 11(3):226-231.
[13]  Giani JF, Burghi V, Veiras LC, et al. Angiotensin-(1-7)attenuates diabetic nephropathyin Zucker diabetic fatty rats[J]. Am J Physiol Renal, 2012, 302(12):F1606-1615.
[14]  Liu GC, Oudit GY, Fang F, et al. Angiotensin-(1-7)-induced activation of ERK1/2 is cAMP/protein kinase A-dependent in glomerular mesangial cells [J]. Am J Physiol Renal Physiol, 2012, 302:F784-F790.
[15]  Zhang K, Meng X, Li D, et al. Angiotensin(1-7)attenuates the progression of streptozotocin-induced diabetic renal injury better than angiotensin receptor blockade[J]. Kidney Int, 2015, 87(2):359-369.
[16]  Santos RA, Ferreira AJ. Pharmacological effects of AVE 0991, a nonpeptide angiotensin-(1-7)receptor agonist [J]. Cardiovasc Drμg Rev, 2006, 24(3-4):239-246.
[17]  da Silveira KD, Coelho FM, Vieira AT, et al. Anti-inflammatory effects of the activation of the angiotensin-(1-7)receptor, MAS, in experimental models of arthritis [J]. J Immunol, 2010, 185(9):5569-5576.

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