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- 2016
蛋白酶体活性升高在1-BP致大鼠周围神经损伤中的作用
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Abstract:
[1] | Kabashi E, Agar JN, Strong MJ, et al. Impaired proteasome function in sporadic amyotrophic lateral sclerosis[J]. Amyotroph Lateral Scler, 2012, 13(4):367-371. |
[2] | Donohue TM Jr,Thomes PG. Ethanol-induced oxidant stress modulates hepatic autophagy and proteasome activity [J]. Redox Biol, 2014, 3:29-39. doi:10.1016/j.redox.2014.10.006. Epub 2014 Oct 31. |
[3] | Zhong Z, Zeng T, Xie K, et al. Elevation of 4-hydroxynonenal and malondialdehyde modified protein levels in cerebral cortex with cognitive dysfunction in rats exposed to 1-bromopropane[J]. Toxicology, 2013, 306:16-23. doi:10.1016/j.tox.2013.01.022. Epub 2013 Feb 8. |
[4] | LoPachin RM, Gavin T, Petersen DR, et al. Molecular mechanisms of 4-hydroxy-2-nonenal and acrolein toxicity:nucleophilic targets and adduct formation [J]. Chem Res Toxicol, 2009, 22(9):1499-1508. |
[5] | Cipierre C, Hays S, Maucort-Boulch D, et al. Malondialdehyde adduct to hemoglobin:a new marker of oxidative stress suitable for full-term and preterm neonates[J]. Oxid Med Cell Longev, 2013, 2013:694014. doi:10.1155/2013/694014. Epub 2013 Jun 13. |
[6] | Sulistio YA, Heese K. The Ubiquitin-proteasome system and molecular chaperone deregulation in alzheimers disease [J]. Mol Neurobiol, 2015. [Epub ahead of print]. |
[7] | Chang TL, Lin SW, Wu SL, et al. Regulation of ubiquitin and 26S proteasome mediated by phenolic compounds during oxidative stress[J]. J Nutr Biochem, 2013, 24(11):1970-1981. |
[8] | Majersik JJ, Caravati EM, and Steffens JD. Severe neurotoxicity associated with exposure to the solvent 1-bromopropane(n-propyl bromide)[J]. Clin Toxicol(Phila), 2007, 45(3):270-276. |
[9] | 王清华, 钟志霞, 陈晶晶, 等. 1-溴丙烷经口染毒致大鼠周围神经病模型的建立[J]. 中华劳动卫生职业病杂志, 2012, 30(10):751-755. WANG Qinghua, ZHONG Zhixia, CHEN Jingjing, et al. Development of peripheral neuropathy rat model induced by 1-bromopropane[J]. Chinese Journal of Industrial Hygiene and Occupational Diseases, 2012, 30(10):751-755. |
[10] | Schreier SM, Muellner MK, Steinkellner H, et al. Hydrogen sulfide scavenges the cytotoxic lipid oxidation product 4-HNE [J]. Neurotox Res, 2010, 17(3):249-256. |
[11] | Ichihara G, Li W, Ding X, et al. A survey on exposure level, health status, and biomarkers in workers exposed to 1-bromopropane [J]. Am J Ind Med, 2004, 45(1):63-75. |
[12] | Dal Vechio FH, Cerqueira F, Augusto O, et al. Peptides that activate the 20S proteasome by gate opening increased oxidized protein removal and reduced protein aggregation[J]. Free Radic Biol Med, 2014, 67:304-313. doi:10.1016/j.freeradbiomed.2013.11.017. Epub 2013 Nov 27. |
[13] | Sharma M, Burré J, Südhof TC. Proteasome inhibition alleviates SNARE-dependent neurodegeneration [J]. Sci Transl Med, 2012, 4(147):147ra113. doi:10.1126/scitranslmed.3004028. |
[14] | Zhai Q, Wang J, Kim A, et al. Involvement of the ubiquitin-proteasome system in the early stages of wallerian degeneration [J]. Neuron, 2003, 39(2):217-225. |
[15] | Ciechanover A, Orian A, Schwartz AL. Ubiquitin-mediated proteolysis:biological regulation via destruction[J]. Bioessays, 2000, 22(5):442-451. |
[16] | Moussa CE. Parkin is dispensable for mitochondrial function, but its ubiquitin ligase activity is critical for macroautophagy and neurotransmitters:therapeutic potential beyond parkinsons disease[J]. Neurodegener Dis, 2015, 15(5):259-270. |
[17] | Carbone DL, Doorn JA, Petersen DR. 4-Hydroxynonenal regulates 26S proteasomal degradation of alcohol dehydrogenase [J]. Free Radical Biol Med, 2004, 37:1430-1439. |
[18] | 唐珍,罗蓉. 泛素-蛋白酶体通路与疾病发生的研究进展[J].兰州大学学报(医学版), 2014(3):71-75. TANG Zhen, LUO Rong. Research progress in ubiquitin-proteasome path and disease occurrence[J]. Journal of Lanzhou University(Medical Sciences), 2014(3):71-75. |