高铁酸盐氧化法降解四溴双酚A及生物毒性控制

为考察高铁酸盐氧化法对四溴双酚A的降解规律、毒性控制效能和降解机理，利用烧杯试验研究了氧化剂投加量、溶液pH对四溴双酚A降解的影响，利用发光细菌考察急、慢性生物毒性的变化及控制效果，并提出可能性降解机理.结果表明，高铁酸盐可高效地降解四溴双酚A，当两者质量浓度均为0.15 mg/L时，在不同pH条件下(6.0~9.0)四溴双酚A均能被完全降解.更高毒性有机中间产物的积累导致反应初期水样的急、慢性毒性有所增强，但随着反应时间延长高铁酸盐氧化法对急、慢性毒性均有良好的控制效果，最佳反应pH为8.0，此时急、慢性毒性当量分别降低至0.02和10.5 TU.机理分析表明，四溴双酚A的降解途径主要包括加成反应、β位断裂反应、去质子化反应和脱溴反应.
The work is to investigate the degradation of TBBPA and its toxicity controlling by ferrate(Ⅵ) oxidizing technology. The effects of oxidant dosage and solution pH on TBBPA degradation was evaluated through the beaker tests. The variation and controlling of the acute and chronic toxicity was investigated, and the possible degradation mechanism was proposed. The results of the experiments showed that, TBBPA could be effectively degraded by ferrate(Ⅵ) oxidation. When the concentrations of ferrate(Ⅵ) and TBBPA were both 0.15 mg/L, TBBPA could be thoroughly degraded under different pH (6.0-9.0) conditions. The acute and chronic toxicity of water samples, enhanced at the initial reaction stage due to the accumulation of more toxic organic intermediates, could be impressively controlled by ferrate(Ⅵ) oxidizing technology with the prolonging of the reaction time. The optimum pH was 8.0, under which condition the acute and chronic toxicity were decreased to 0.02 TU and 10.5 TU respectively. The analysis of mechanism showed that the degradation pathway of TBBPA mainly included the addition reaction, the beta scission, the deprotonating reaction and the debromination reaction.