Jin L, Carpenter D, Moerdyk-Schauwecker M, et al. Cellular FLIP can substitute for the herpes simplex virus type 1 latency-associated transcript gene to support a wild-type virus reactivation phenotype in mice[J]. Neurovirol, 2008, 14(5)：389-400.
[2]
Gondi CS, Rao JS. Concepts in in vivo siRNA？delivery？for？cancer therapy[J]. Cell Physiol, 2009, 220(2)：285-291.
[3]
Kopp SJ, Storti CS, Muller WJ. Herpes simplex virus-2 glycoprotein interaction with HVEM influences virus-specific recall cellular responses at the Mucosa[J]. Clin Dev Immunol, 2012, 2012：284104.
[4]
Paddison PJ, Caudy AA, Bernstein E, et al. Short hairpin RNAs(shRNAs)induce sequence-specific silencing in mammalian cells[J]. Genes Dev, 2002, 16(8)：948-958.
[5]
Paul CP, Good PD, Winer I, et al. Effective expression of small interfering RNA in human cells[J]. Nat Biotechno, 2002, 20：505-508.
[6]
Brummelkamp TR, Bemards R, Agami R. A system for stable expression of short interfering RNAs in mammalian cells[J]. Science, 2002, 296(5567)：550-553.
[7]
Hafner M, Landthaler L, Burger M, et al. Transcriptome-wide identification of RNA-binding protein and microRNA binding sites by PAR-CLIP[J]. Cell, 2010, 141(1)：129-141.
[8]
Jackson AL, Bartz SR, Schelter J, et al. Expression profiling reveals off-target gene regulation by RNAi[J]. Nature Biotech, 2003, 21(6)：635-637.
[9]
Zhang T, Cheng T, Wei L, et al. Efficient inhibition of HIV-1 replication by an artificial polycistronic miRNA construct[J]. Virol, 2012, 9：118.
[10]
Anesti AM, Peeters PJ, Royaux I, et al. Efficient delivery of RNA interference to peripheral neurons in vivo using herpes simplex virus[J]. Nucleic Acids Res, 2008, 36(14)：e86.
Muppirala UK, Honavar VG, Dobbs D. Predicting RNA-protein interactions using only sequence information[J]. BMC Bioinformatics, 2011, 12：489.
[13]
Makarova KS, Grishin NV, Shabalina SA, et al. A putative RNA-interference-based immune system in prokaryotes：computational analysis of the predicted enzymatic machinery functional analogies with eukaryotic RNAi, and hypothetical mechanisms of action[J]. Biol Direct, 2006, 1：7.
[14]
Jackson AL, Bartz SR, Schelter J, et al. Expression profiling reveals off-target gene regulation by RNAi[J]. Nat Biotechnol, 2003, 21(6)：635-637.
[15]
Chew GT, Watts GF. HSV-2 and atherosclerosis：adding to the alp-habet soup of coronary risk in HIV infection[J]. Atherosclerosis, 2012, 223(2)：278-279.
[16]
McMaIlus MT, Petersen CP, Hailles BB, et al. Gene silencillg using micro-RNA designed hairpins[J]. RNA, 2002, 8(6)：842-850.
[17]
Hoshino Y, Pesnicak L, Straus SE, et al. Impairment in reactivation of a latency associated transcript(LAT)-deficient HSV-2 is not solely dependent on the latent viral load or the number of CD8(+)T cells infiltrating the ganglia[J]. Virology, 2009, 387(1)：193-199.
