GOFF J P.Major advances in our understanding of nutritional influences on bovine health[J].Journal of Dairy Science, 2006, 89(4):1292-1301.
[2]
SORDILLO L M, AITKEN S L.Impact of oxidative stress on the health and immune function of dairy cattle[J].Veterinary Immunology and Immunopathology, 2009, 128(1/2/3):104-109.
[3]
SORDILLO L M.Selenium-dependent regulation of oxidative stress and immunity in periparturient dairy cattle[J].Veterinary Medicine International, 2013, 2013:154045, doi:10.1155/2013/154045.
[4]
HALLIWELL B, GUTTERIDEGE J M C.Free radicals in biology and medicine[M].4th ed.Oxford:Oxford University Press, 2007.
[5]
WEBER P S D, MADSEN S A, SMITH G W, et al.Pre-translational regulation of neutrophil L-selectin in glucocorticoid-challenged cattle[J].Veterinary Immunology and Immunopathology, 2001, 83(3/4):213-240.
[6]
BURTON J L, ERSKINE R J.Immunity and mastitis.Some new ideas for an old disease[J].Veterinary Clinics of North America:Food Animal Practice, 2003, 19(1):1-45.
[7]
KEHRLI M E, Jr, NONNECKE B J, ROTH J A.Alterations in bovine neutrophil function during the periparturient period[J].American Journal of Veterinary Research, 1989, 50(2):207-214.
[8]
SORDILLO L M.Factors affecting mammary gland immunity and mastitis susceptibility[J].Livestock Production Science, 2005, 98(1/2):89-99.
[9]
SHAFER-WEAVER K A, CORL C M, SORDILLO L M.Shifts in bovine CD4+ subpopulations increase TH-2 compared to TH-1 effector cells during the postpartum period[J].Journal of Dairy Science, 1999, 82(8):1696-1706.
[10]
SHAFER-WEAVER K A, SORDILLO L M.Bovine CD8+ suppressor lymphocytes alter immune responsiveness during the postpartum period[J].Veterinary Immunology and Immunopathology, 1997, 56(1/2):53-64.
[11]
SORDILLO L M, O''BOYLE N, GAUD J C, et al.Shifts in thioredoxin reductase activity and oxidant status in mononuclear cells obtained from transition dairy cattle[J].Journal of Dairy Science, 2006, 90(3):1186-1192.
[12]
D''ROURKE O.Nutrition and udder health in dairy cows:a review[J].Irish Veterinary Journal, 2009, 62(Suppl.4):S15-S20.
[13]
LARSEN H J.Relations between selenium and immunity[J].Norwegian Journal of Agricultural Science, 1993, 11:105-119.
[14]
WEISS W P, HOGAN J S, SMITH K L, et al.Relationships among selenium, vitamin E, and mammary gland health in commercial dairy herds[J].Journal of Dairy Science, 1990, 73(2):381-390.
[15]
HOGAN J S, SMITH K L, WEISS W P, et al.Relationships among vitamin E, selenium, and bovine blood neutrophils[J].Journal of Dairy Science, 1990, 73(9):2372-2378.
[16]
ARTHUR J R, MCKENZIE R C, BECKETT G J.Selenium in the immune system[J].Journal of Nutrition, 2003, 133(5):1457-1459.
[17]
NDIWENI N, FINCH J M.Effects of in vitro supplementation with alpha-tocopherol and selenium on bovine neutrophil functions:implications for resistance to mastitis[J].Veterinary Immunology and Immunopathology, 1996, 51(1/2):67-78.
[18]
NDIWENI N, FINCH J M.Effects of in vitro supplementation of bovine mammary gland macrophages and peripheral blood lymphocytes with alpha-tocopherol and sodium selenite:implications for udder defences[J].Veterinary Immunology and Immunopathology, 1995, 47(1/2):111-121.
[19]
PARNHAM M J, BITTNER C, WINKELMANN J.Chemi luminescence from mouse resident macrophages characterization and modulation by arachidonate metabolites[J].Immunopharmacology, 1983, 5:277-291.
[20]
KIM S H, JOHNSON V J, SHIN T Y, et al.Selenium attenuates lipopolysaccharide-induced oxidative stress responses through modulation of p38 MAPK and NF-κB signaling pathways[J].Experimental Biology and Medicine, 2004, 229(2):203-213.
[21]
SURAI P F.Selenium in nutrition and health[M].United Kingdom:Nottingham University Press, 2006.
[22]
CAO Y Z, MADDOX J F, MASTRO A M, et al.Selenium deficiency alters the lipoxygenase pathway and mitogenic response in bovine lymphocytes[J].The Journal of Nutrition, 1992, 122(11):2121-2127.
[23]
LARSEN H J, MOKSNES K, OVERNES G.Influence of selenium on antibody production in sheep[J].Research in Veterinary Science, 1988, 45(1):4-10.
[24]
ROY M, KIREMIDJIANSCHUMACHER L, WISHE H I, et al.Supplementation with selenium and human immune cell functions:effect on lymphocyte-proliferation and interleukin-2 receptor expression[J].Biological Trace Element Research, 1994, 41(1/2):103-114.
[25]
PAVLATA L, PRASEK J, FILIPEK J, et al.Influence of parenteral administration of selenium and vitamin E during pregnancy on selected metabolic parameters and colostrum quality in dairy cows at parturition[J].Veterinarni Medicina, 2004, 49(5):149-155.
[26]
AWADEH F T, KINCAID R L, JOHNSON K A.Effect of level and source of dietary selenium on concentrations of thyroid hormones and immunoglobulins in beef cows and calves[J].Journal of Animal Science, 1998, 76(4):1204-1215.
STABEL J R, REINHARDT T A, NONNECKE B J, et.Effect of selenium and reducing agents on in vitro immunoglobulin M synthesis by bovine lymphocytes[J].Journal of Dairy Science, 1991, 74(8):2501-2506.
[29]
LEYAN V, WITTWER F, CONTRERAS P A, et al.Serum and colostrum immunoglobulin concentrations from selenium deficient cows and in the blood of their calves[J].Archivos De Medicina Veterinaria, 2004, 36(2):155-162.
[30]
HAMPEL G, WATANABE K, WEKSLER B B, et al.Selenium deficiency inhibits prostacyclin release and enhances production of platelet activating factor by human endothelial cells[J].Biochimica et Biophysica Acta:Lipids and Lipid Metabolism, 1989, 1006(2):151-158.
[31]
WEAVER J A, MADDOX J F, CAO Y Z, et al.Increased 15-HPETE production decreases prostacyclin synthase activity during oxidant stress in aortic endothelial cells[J].Free Radical Biology and Medicine, 2001, 30(3):299-308.
[32]
CAO Y Z, REDDY C C, SORDILLO L M.Altered eicosanoid biosynthesis in selenium-deficient endothelial cells[J].Free Radical Biology and Medicine, 2000, 28(3):381-389.
[33]
SOGA M, MATSUZAWA A, ICHIJO H.Oxidative stress-induced diseases via the ASK1 signaling pathway[J].International Journal of Cell Biology, 2012, doi:10.1155/2012/439587.
[34]
SHI Y Q, CHEN X, DAI J, et al.Selenium pretreatment attenuates formaldehyde-induced genotoxicity in A549 cell lines[J].Toxicology and Industrial Health, 2012, doi:10.1177/0748233712466129.
[35]
YING X Z, CHEN X W, CHENG S W, et al.SeMet Inhibits IL-1β-induced rheumatoid fibroblast-like synoviocytes proliferation and the production of inflammatory mediators[J].Biological Trace Element Research, 2013, 153(1/2/3):437-445.
