ASHKENAZI A, DIXIT V M. Death receptors: signaling and modulation[J]. Science, 1998, 281:1305-1308.
[2]
NAGATA S. Apoptosis by death factor[J]. Cell, 1997,88(3):355-365.
[3]
ALLEN R G, TRESINI M. Oxidative stress and gene regulation[J]. Free Radical Biology and Medicine 2000, 28(3):463-499.
[4]
HATOUM O A, OTTERSON M F, KOPELMAN D, et al. Radiation induces endothelial dysfunction in murine intestinal arterioles via enhanced production of reactive oxygen species[J]. Arteriosclerosis, Thrombosis and Vascular Biology, 2006, 26(2):287-294.
[5]
STELLER H. Mechanisms and genes of cellular suicide[J]. Science, 1995, 267:1445-1449.
[6]
MEIER P, FINCH A, EVAN G. Apoptosis in development[J]. Nature, 2000, 407:796-801.
[7]
WYLLIE A H. Apoptosis: an overview[J]. British Medical Bulletin, 1997, 53(3):451-465.
[8]
KUMAR S, VAUX D L. Apoptosis A cinderella caspase takes center stage[J]. Science, 2002, 297:1290-1291.
[9]
THORNBERRY N A, LAZEBNIK Y. Caspases: enemies within[J]. Science, 1998, 281:1312-1316.
[10]
ZOU H, HENZEL W J, LIU X, et al. Apaf-1, a human protein homologous to C. elegans CED-4, participates in cytochrome c-dependent activation of caspase-3[J]. Cell, 1997, 90(3):405-413.
[11]
LI P, NIJHAWAN D, BUDIHARDJO I, et al. Cytochrome c and dATP-dependent formation of Apaf-1/caspase-9 complex initiates an apoptotic protease cascade[J]. Cell, 1997, 91(4):479-489.
[12]
LAKHANI S A, MASUD A, KUIDA K, et al. Caspases 3 and 7: key mediators of mitochondrial events of apoptosis[J]. Science, 2006, 311:847-851.
[13]
SCORRANO L, OAKES S A, OPERFRMAN J T, et al. BAX and BAK regulation of endoplasmic reticulum Ca2+: a control point for apoptosis[J]. Science, 2003, 300:135-139.
[14]
MAJUMDAR A P, DU J, YU Y, et al. Cell cycle and apoptosis regulatory protein-1: a novel regulator of apoptosis in the colonic mucosa during aging[J]. American Journal of Physiology, 2007, 293(6):G1215-G1222.
[15]
JIMBO A, FUJITA E, KOUROKU Y, et al. ER stress induces caspase-8 activation, stimulating cytochrome c release and caspase-9 activation[J]. Experimental Cell Research, 2003, 283(2):156-166.
[16]
FERRI K F, KROEMER G. Organelle-specific initiation of cell death pathways[J]. Nature Cell Biology, 2001, 3(11):E255-E263.
[17]
DI SANO F, FERRARO E, TUFI R, et al. Endoplasmic reticulum stress induces apoptosis by an apoptosome-dependent but caspase 12-independent mechanism[J]. The Journal of Biological Chemistry, 2006, 281(5):2693-2700.
[18]
FUJIMOTO K, IWAKIRI R, WU B, et al. Homeostasis in the small intestinal mucosa balanced between cell proliferation and apoptosis is regulated partly by the central nervous system[J]. Journal of Gastroenterology, 2002,37(Suppl. 14):139-144.
[19]
ZABIELSKI R, GODLEWSKI M M, GUILLOTEAU P. Control of development of gastrointestinal system in neonates[J]. Journal of Physiology and Pharmacology, 2008, 59(Suppl. 1):35-54.
[20]
BOUDRY G, PéRON V, HU？ROU-LURON I L, et al. Weaning induces both transient and long-lasting modifications of absorptive, secretory, and barrier properties of piglet intestine[J]. The Journal of Nutrition, 2004, 134(9):2256-2262.
[21]
HENGARTNER M O. The biochemistry of apoptosis[J]. Nature, 2000, 407:770-776.
[22]
WU B, IWAKIRI R, TSUNADA S, et al. iNOS enhances rat intestinal apoptosis after ischemia-reperfusion[J]. Free Radical Biology & Medicine, 2002, 33(5):649-658.
[23]
GRANGER D N. Role of xanthine oxidase and granulocytes in ischemia-reperfusion injury[J]. The American Journal of Physiology, 1988, 255:H1269-H1275.
[24]
ABRAHAM C, CHO J H. IL-23 and autoimmunity: new insights into the pathogenesis of inflammatory bowel disease[J]. Annual Review of Medicine, 2009, 60:97-110.
[25]
LEVINE A D. Apoptosis: implications for inflammatory bowel disease[J]. Inflammatory Bowel Diseases, 2000, 6(3):191-205.
[26]
KOUNTOURAS J, KOUKLAKIS G, ZAVOS C, et al. Apoptosis, inflammatory bowel disease and carcinogenesis: overview of international and Greek experiences[J]. Canadian Journal of Gastroenterology, 2003, 17(4):249-258.
[27]
VELTKAMP C, ANSTAETT M, WAHL K, et al. Apoptosis of regulatory T lymphocytes is increased in chronic inflammatory bowel disease and reversed by anti-TNFα treatment[J]. Gut, doi:10.1136/gut.2010.217117.
[28]
INA K, ITOH J, FUKUSHIMA K, et al. Resistance of Crohno’s disease T cells to multiple apoptotic signals is associated with a Bcl-2/Bax mucosal imbalance[J]. Journal of Immunology, 1999, 163(2):1081-1090.
[29]
CHIPUK J E, KUWANA T, BOUCHIER-HAYES L, et al. Direct activation of Bax by p53 mediates mitochondrial membrane permeabilization and apoptosis[J]. Science, 2004, 303: 1010-1014.
[30]
HAUSMANN M. How bacteria-induced apoptosis of intestinal epithelial cells contributes to mucosal inflammation[J]. International Journal of Inflammation, doi:10.4061/2010/574568
PANEE J, LIU W, NAKAMURA K, et al. The responses of HT22 cells to the blockade of mitochondrial complexes and potential protective effect of selenium supplementation[J]. International Journal of Biological Sciences, 2007, 3(5):335-341.
CERA K R, MAHAN D C, CROSS R F, et al. Effect of age, weaning and postweaning diet on small intestinal growth and jejunal morphology in young swine[J]. Journal of Animal Science, 1988, 66(2):574-584.
