GROSCH S,KAINA B. Transcriptional activation of apurin-ic /apyrimidinic endonuclease (Ape, Ref21 ) by oxidativestress requires CREB[J]. Biochem Biophys Res Commun,1999,261(3):859-863.
[3]
JONES D P. Redefining oxidative stress[J]. Antioxid Red-ox Signa,2006,8(9-10):1865-1879.
[4]
FLAHERTY D M,MONICK M M,Carter A B,et al. Oxi-dant-mediated increases in redox factor-1 nuclear proteinand activator protein-1 DNA binding in asbestos-treatedmacrophages[J]. J Immuno 2002,168(11):5675-5681．
[5]
FAN J, WILSON D M. Protein-protein interactions andposttranslational modifications in mammalian base exci-sion repair[J ]. Free Radic Biol Med, 2005, 38 (9):1121-1138.
[6]
VASKOM R, GUO C, KELLEY M R. The multifunctionalDNA repair/redox enzyme Ape1 /Re-f1 promotes survivalof neurons after oxidative stress [J]. DNA Rep, 2005, 4(3):367-379.
[7]
KIM C S, SON S J, KIM E K, et al. Apurinic /apyrimidin-ic endonuclease1 /redox factor-1 inhibits monocyte adhe-sion in endothelial cells [J]. Cardiovasc Res, 2006, 69(3):520-526.
[8]
徐海燕, 辛晓燕. APE /Ref-1 与肿瘤的研究进展[J]. 现代肿瘤医学,2012,20(11):2426-2429
[9]
Katoh K, Asimenos G, Toh H. Multiple alignment of DNAsequences with MAFFT[J]. Methods in Molecular Biolo-gy,2009,537:39-64.
[10]
ARNOLD K, BORDOLI L, Kopp J, et al. The SWISS-MODEL Workspace: A web-based environment for pro-tein structure homology modelling [J]. Bioinformatics,2006,22(2):195-201.
[11]
FRITZ G. HUMAN APE /Ref-1 protein[J]. The Interna-tional Journal of Biochemistry & Cell Biology, 2000, 32(9): 925-929.
Yu E,GAUCHER S P,HADI M Z. Probing conformationalchanges in Ape1 during the progression of base excision re-pair[J]. Biochemistry,2010,49 (18):3786-3796.
[14]
Qu J, Liu G H, HUANG B, et al. Nitric oxide controlsnuclear export of APE1 /Ref-1 through S-nitrosation ofcysteines93 and 310 [J]. Nucleic Acids Res, 2007, 35(8): 2522-2532.
