Shastry S, Ingram AJ, Scholey JW, et al. Homocysteine induces mesangial cell apoptosis via activation of p38-mitogen-activated protein kinase [J]. Kidney Int, 2007, 71(4):304-311.
[2]
Yan SK, Chang T, Wang H, et al. Effects of hydrogen sulfide on homocysteine- induced oxidative stress in vascμlar smooth muscle cells[J]. Biochem Biophys Res Commun,2006,351(2):485-491.
[3]
Schnyder G, Roffi M, Flammer Y, et al. Association of plasma homocysteine with restenosis after percutaneous coronary angioplasty [J]. Eur Heart J,2002, 23(9):726-733.
Cooke JP. The endothelium: a new target for therapy[J]. VASC MED, 2000, 5:49-53.
[6]
Ross R.The pathogenesis of atherosclerosisanupolate[J].N Eng J Med, 1985,314:488-493.
[7]
Wilcken DE, Wilcken B.The pathogenesis of coronary artery disease. A possible role for methionine metabolism[J]. J Clin Invest,1976,57(4):1079-1082.
[8]
Tsai JC, Perrella MA, Yoshizum iM. Promotion of vascular smooth muscle cell growth by homocysteine: a link to atherosclerosis [J].Proc Natl Acad Sci USA, 1994, 91(14):6369-6373.
[9]
Blundell G, Rose FA, Tudball N.Homocysteine induced endothelial cell toxicity and its protection[J].Biochem Soc Trans,1994,22(3):341S.
[10]
Jones BG, Rose FA, Tudball N.Lipid peroxidation and homocysteine induced toxicity[J].Atherosclerosis,1994,105(2):165-170.
[11]
Taban-shomal O, KILTER H, Wagnet A, et al. The cardiac effects of prolonged vitamin B12 and folate deficiency in rats [J]. Cardiovasc Toxicol, 2009, 9(2): 95-102.
[12]
Zhou J, Austin RC. Contributions of hyperhomocysteinemia to atherosclerosis: Causal relationship and potential mechanisms [J]. Biofactors, 2009, 35(2): 120-129.
