Zeng QY, Chen R, Darmawan J, et al. Rheumatic diseases in China [J]. Arthritis Res Ther, 2008, 10:R17.
[2]
Braun J, Sieper J. Ankylosing spondylitis[J]. Lancet 2007, 369:1379？90.
[3]
Brewerton DA, Hart FD, Caffrey M, et al. Ankylosing spondylitis and HLA-B27[J]. Lancet,1973,1:904-907.
[4]
Goie The HS,Steven MM,van der Linden SM,et al. Evaluation of diagnostic criteria for ankylosing spondylitis:A comparison ofthe Rome, New York and modified New York criteria in patients with a positive clinical history screening test for ankylosing spondylitis[J].Br J Rheumatol, 1985, 24(3): 242-249.
Gundry CN, Dobrowolski SF, Martin YR, et al.Base-pair neutral homozygotes can be discriminated by calibrated high-resolution melting of small amplicons [J]. Nucleic Acids Res,2008,36:3401.
Wendling D, Cedoz JP, Racadot E.Serum and synovial fluid levels of p40 IL12/23 in spondyloarthropathy patients[J].Clin Rheumatol,2009,28(2):187-190.
[9]
Iwakura Y, Ishigame H. The IL23/IL17 axis in inflammation [J]. J Clin Invest, 2006, 116(5):1218-1222.
[10]
Duerr RH, Taylor KD, Brant SR, et al. A genome-wide association study identifies IL23R as an inflammatory bowel disease gene [J]. Science, 2006, 314:1461-1463.
[11]
Einarsdottir E, Koskinen LL, Dukes E, et al. IL23R in the Swedish, Finnish, Hungarian and Italian populations: association with IBD and psoriasis, and linkage to celiac disease [J]. BMC Med Genet, 2009, 10:8.
[12]
Wellcome Trust Case Control Consortium, Australo-Anglo-American Spondylitis Consortium(TASC), Burton PR, et al. Association scan of 14,500 nonsynonymous SNPs in four diseases identifies autoimmunity variants [J]. Nat Genet, 2007, 39: 1329-1337.
[13]
Sáfrány E, Pazár B, Cs？ngei V, et al. Variants of the IL23R gene are associated with ankylosing spondylitis but not with Sj？gren syndrome in Hungarian population samples. Scand J Immunol [J]. 2009, 70(1):68-74.
[14]
Rahman P, Inman RD, Gladman DD, et al. Association of interleukin-23 receptor variants with ankylosing spondylitis [J]. Arthritis Rheum, 2008, 58:1020-1025.
Pimentel-Santos FM, Ligeiro D, Matos M, et al. Association of IL23R and ERAP1 genes with ankylosing spondylitis in a Portuguese population[J].Clin Exp Rheumatol, 2009, 27:800-806.
[17]
Sung IH, Kim TH, Bang SY, et al. IL-23R polymorphisms in patients with ankylosing spondylitis in Korea [J]. J Rheumatol, 2009, 36:1003-1005.
[18]
Karaderi T, Harvey D, Farrar C, et al. Association between the interleukin 23 receptor and ankylosing spondylitis is confirmed by a new UK case-control study and meta analysis of published series[J]. Rheumatology, 2009, 48: 386-389.
[19]
Davidson SI, Wu X, Liu Y, et al. Association of ERAP1, but not IL23R, with ankylosing spondylitis in a Han Chinese population [J]. Arthritis Rheum, 2009, 60: 3263-3268.
[20]
Brown MA, Laval SH, Brophy S, et al. Recurrence risk modelling of the genetic susceptibility to ankylosing spondylitis [J]. Ann Rheum Dis, 2000, 59:883- 886.
[21]
Brown MA, Kennedy LG, MacGregor AJ, et al. Susceptibility to ankylosing spondylitis in twins: the role of genes, HLA and the environment[J]. Arthritis Rheum,1997,40:1823-1828.
