Riljak V, Milotová M, Jandová K, et al. Repeated kainic acid administration and hippocampal neuronal degeneration[J]. Prague Med Rep, 2005, 106(1): 75-78.
[2]
Chuang YC, Lin JW, Chen SD, et al. Preservation of mitochondrial integrity and energy metabolism during experimental status epilepticus leads to neuronal apoptotic cell death in the hippocampus of the rat[J]. Seizure, 2009, 18(6): 420-428.
[3]
Tian FF, Zeng C, Guo TH, et al. Mossy fiber sprouting, hippocampal damage and spontaneous recurrent seizures in pentylenetetrazole kindling rat model[J]. Acta Neurol Belg, 2009, 109(4): 298-304.
[4]
Wang Y, Majors A, Najm I, et al. Postictal alteration of sodium content and apparent diffusion coefficient in epileptic rat brain induced by kainic acid[J]. Epilepsia, 1996, 37(10): 1000-1006.
[5]
Engelhorn T, Hufnagel A, Weise J, et al. Monitoring of acute generalized status epilepticus using multilocal diffusion MR imaging: early prediction of regional neuronal damage[J]. Am J Neuroradiol, 2007, 28(2): 321-327.
[6]
Di Bonaventura C, Bonini F, Fattouch J, et al. Diffusion-weighted magnetic resonance imaging in patients with partial status epilepticus[J]. Epilepsia, 2009, 50(Suppl 1): 45-52.
[7]
Huang YC, Weng HH, Tsai YT, et al. Periictal magnetic resonance imaging in status epilepticus[J]. Epilepsy Res, 2009, 86(1): 72-81.
[8]
Sakakibara T, Nakagawa E, Saito Y, et al. Hemiconvulsion-hemiplegia syndrome in a patient with severe myoclonic epilepsy in infancy[J]. Epilepsia, 2009, 50(9): 2158-2162.
[9]
Lee SK, Lee SY, Yun CH, et al. Ictal SPECT in neocortical epilepsies: clinical usefulness and factors affecting the pattern of hyperperfusion[J]. Neuroradiology, 2006, 48(9): 678-684.
[10]
Szabo K, Poepel A, Pohlmann-Eden B, et al. Diffusion-weighted and perfusion MRI demonstrates parenchymal changes in complex partial status epilepticus[J]. Brain, 2005, 128(6): 1369-1376.
[11]
Mathews MS, Smith WS, Wintermark M, et al. Local cortical hypoperfusion imaged with CT perfusion during postictal Todd’s paresis[J]. Neuroradiology, 2008, 50(5): 397-401.
[12]
Marchi N, Angelov L, Masaryk T, et al. Seizure-promoting effect of blood-brain barrier disruption[J]. Epilepsia, 2007, 48(4): 732-742.
[13]
Wasterlain CG, Fujikawa DG, Penix LR, et al. Pathophysiological mechanisms of brain damage from status epilepticus[J]. Epilepsia, 1993, 34(Suppl 1): 137-153.
[14]
Fujikawa DG. The temporal evolution of neuronal damage from pilocarpin-induced status epilepticus[J]. Brain Res, 1996, 725(1): 11-22.
[15]
Lansberg MG, O’Brien MW, Norbash AM, et al. MRI abnormalities associated with partial status epilepticus[J]. Neurology, 1999, 52(5): 1021-1027.
[16]
Men S, Lee DH, Barron JR, et al. Selective Neuronal Necrosis associated with status epilepticus: MR findings[J]. Am J Neuroradiol, 2000, 21(10): 1837-1840.
[17]
Senn P, L？vblad KO, Zutter D, et al. Changes on diffusion-weighted MRI with focal motor status epilepticus: case report[J]. Neuroradiology, 2003, 45(4): 246-249.
[18]
Toledo M, Munuera J, Sueiras M, et al. MRI findings in aphasic status epilepticus[J]. Epilepsia, 2008, 49(8): 1465-1469.
[19]
Ampuero E, Dagnino-Subiabre A, Sandoval R, et al. Status epilepticus induces region-specific changes in dendritic spines, dendritic length and TrkB protein content of rat brain cortex[J]. Brain Res, 2007, 1150: 225-238.
