Onat A, Uyarel H, Hergenc G, et al. Serum uric acid is a determinant of metabolic syndrome in a population based study[J]. AM J Hypertens, 2006, 19(10): 1055-1062.
[ 6 ] Duvnjak M, Leroti I, Barsi N, et al. Pathogenesis and management issues for non-alcoholic fatty liver disease[J]. World J Gastroenterol, 2007, 13(34): 4539-4550.
[8]
[ 7 ] Lonardo A, Caldwell S, Loria P. Clinical physiology of NAFLD: a critical overview of pathogenesis and treatment.Expert Review of Endocrinology & Metabolism, 2010, 5(3): 403-423.
[9]
[ 8 ] Rashid S, Watanabe T, Sakaue T, et al. Mechanisms of HDL lowering in insulin resistant,hypertriglyceridemic states: the combine deffect of HDL triglyceride enrichment and elevated hepatic lipase activity[J]. Clin Biochem, 2003, 36(6): 421-429.
[10]
[ 9 ] Luyckx FH， Lefebvre PJ， Scheen AJ. Non-alconolic steatohepatitis: association with obesity and insulin resistance,and influence of weight loss[J]. Diabetes Metab, 2000, 26(2): 98-106.
HuangHL, LinWY, Lee LT, et al. Metabolic syndrome is related to nonalcoholic steatoh epatitis in severely obese subjects [J]. Obes Surg, 2007, 17(11): 1457-1463.
