Myles-Worsley M, Ord L, Blailes F, et al. P50 sensory gating in adolescents from a Pacific island isolate with elevated risk for schizophrenia[J]. Biol Psychiatry, 2004, 55(7): 663-667.
[2]
Duncan CC, Barry RJ, Connolly JF, et al. Event-related potentials in clinical research: guidelines for eliciting, recording, and quantifying mismatch negativity, P300, and N400[J]. Clin Neurophysiol, 2009, 120(11): 1883-1908.
[3]
Lee SY, Namkoong K, Cho HH, et al. Reduced visual P300 amplitudes in individuals at ultra-high risk for psychosis and first-episode schizophrenia[J]. Neurosci Lett, 2010, 486(3): 156-160.
[4]
Higuchi Y, Sumiyoshi T, Seo T, et al. Mismatch negativity and cognitive performance for the prediction of psychosis in subjects with At-Risk mental state[J]. PLoS One, 2013, 8(1): 1-10.
[5]
Lin YT, Liu CM, Chiu MJ, et al. Differentiation of schizophrenia patients from healthy subjects by mismatch negativity and neuropsychological tests[J]. PLoS One, 2012, 7(4): 1-9.
[6]
Ziermans TB, Schothorst PF, Sprong MA, et al. Reduced prepulse inhibition as an early vulnerability marker of the psychosis prodrome in adolescence[J]. Schizophr Res, 2012, 134(1): 10-15.
[7]
Polich J. P300 clinical utility and control of variability[J]. J Clin Neurophysiol, 1998, 15(1): 14-33.
[8]
van der Stelt O, Frye J, Lieberman JA, et al. Impaired P3 generation reflects high-level and progressive neurocognitive dysfunction in schizophrenia[J]. Arch Gen Psychiatry, 2004, 61(3): 237-248.
[9]
Yeap S, Kelly SP, Sehatpour P, et al. Visual sensory processing deficits in schizophrenia and their relationship to disease state[J]. Eur Arch Psychiatry Clin Neurosci, 2008, 258(5): 305-316.
[10]
Yeap S, Kelly SP, Sehatpour P, et al. Early visual sensory deficits as endophenotypes for schizophrenia: high-density electrical mapping in clinically unaffected first-degree relatives[J]. Arch Gen Psychiatry, 2006, 63(11): 1180-1188.
[11]
Koychev I, El-Deredy W, Haenschel C, et al. Visual information processing deficits as biomarkers of vulnerability to schizophrenia: an event-related potential study in schizotypy[J]. Neuropsychologia, 2010, 48(7): 2205-2214.
[12]
Schwartzman D, Maravic K, Kranczioch C, et al. Altered early visual processing components in hallucination-prone individuals[J]. Neuroreport, 2008, 19(9): 933-937.
[13]
Yeap S, Kelly SP, Thakore JH, et al. Visual sensory processing deficits in first-episode patients with schizophrenia[J]. Schizophr Res, 2008, 102(1/2/3): 340-343.
[14]
Koychev I, El-Deredy W, Deakin JF. New visual information processing abnormality biomarker for the diagnosis of schizophrenia[J]. Expert Opin Med Diagn, 2011, 5(4): 357-368.
[15]
Bramon E, Shaikh M, Broome M, et al. Abnormal P300 in people with high risk of developing psychosis[J]. Neuroimage, 2008, 41(2): 553-560.
[16]
Raux G, Bonnet-Brilhault F, Louchart S, et al. The-2 bp deletion in exon 6 of the “alpha 7-like” nicotinic receptor subunit gene is a risk factor for the P50 sensory gating deficit[J]. Mol Psychiatry, 2002, 7(9): 1006-1011.
[17]
Freedman R, Olincy A, Ross RG, et al. The genetics of sensory gating deficits in schizophrenia[J]. Curr Psychiatry Rep, 2003, 5(2): 155-161.
[18]
Bramon E, Rabe-Hesketh S, Sham P, et al. Meta-analysis of the P300 and P50 waveforms in schizophrenia[J]. Schizophr Res, 2004, 70(2/3): 315-329.
[19]
Freedman R, Ross R, Leonard S, et al. Early biomarkers of psychosis[J]. Dialogues Clin Neurosci, 2005, 7(1): 17-29.
[20]
Clementz BA. Psychophysiological measures of (dis)inhibition as liability indicators for schizophrenia[J]. Psychophysiology, 1998, 35(6): 648-668.
[21]
Brockhaus-Dumke A, Schultze-Lutter F, Mueller R, et al. Sensory gating in schizophrenia: P50 and N100 gating in antipsychotic-free subjects at risk, first-episode, and chronic patients[J]. Biol Psychiatry, 2008, 64(5): 376-384.
[22]
[ 1 ] Yung AR, Nelson B, Stanford C, et al. Validation of “prodromal” criteria to detect individuals at ultra high risk of psychosis: 2 year follow-up[J]. Schizophr Res, 2008, 105(1/2/3): 10-17.
[23]
[ 2 ] Tretter F, Gebicke-Haerter PJ. Systems biology in psychiatric research: from complex data sets over wiring diagrams to computer simulations[J]. Methods Mol Biol, 2012, 829: 567-592.
[24]
[ 3 ] Nieman D, Becker H, Van DR, et al. Antisaccade task performance in patients at ultra high risk for developing psychosis[J]. Schizophr Res, 2007, 95(1/3): 54-60.
[25]
[ 4 ] Olin SC, Mednick SA. Risk factors of psychosis: identifying vulnerable populations premorbidly[J]. Schizophr Bull, 1996, 22(2): 223-240.
[26]
[ 5 ] Mossaheb N, Wiesegger G, Amminger GP, et al. Early recognition and intervention for schizophrenia[J]. Nervenarzt, 2006, 77(1): 32-34.
[27]
[ 6 ] Sprong M, Becker HE, Schothorst PF, et al. Pathways to psychosis: a comparison of the pervasive developmental disorder subtype Multiple Complex Developmental Disorder and the “At Risk Mental State”[J]. Schizophr Res, 2008, 99(1/2/3): 38-47.
[28]
[ 7 ] Haroun N, Dunn L, Haroun A, et al. Risk and protection in prodromal schizophrenia: ethical implications for clinical practice and future research[J]. Schizophr Bull, 2006, 32(1): 166-178.
[29]
[ 8 ] Simon AE, Cattapan-Ludewig K, Zmilacher S, et al. Cognitive functioning in the schizophrenia prodrome[J]. Schizophr Bull, 2007, 33(3): 761-771.
[30]
[ 9 ] Van Tricht MJ, Nieman DH, Koelman JH, et al. Reduced parietal P300 amplitude is associated with an increased risk for a first psychotic episode[J]. Biol Psychiatry, 2010, 68(7): 642-648.
[31]
Van Der Stelt O, Belger A. Application of electroencephalography to the study of cognitive and brain functions in schizophrenia[J]. Schizophr Bull, 2007, 33(4): 955-970.
[32]
Schwarz E, Bahn S. Biomarker discovery in psychiatric disorders[J]. Electrophoresis, 2008, 29(13): 2884-2890.
[33]
Fusar-Poli P, Crossley N, Woolley J, et al. White matter alterations related to P300 abnormalities in individuals at high risk for psychosis: an MRI-EEG study[J]. J Psychiatry Neurosci, 2011, 36(4): 239-248.
[34]
Shutara Y, Koga Y, Fujita K, et al. An event-related potential study on the impairment of automatic processing of auditory input in schizophrenia[J]. Brain Topogr, 1996, 8(3): 285-289.
[35]
Van Tricht MJ, Nieman DH, Koelman JH, et al. Auditory ERP components before and after transition to a first psychotic episode[J]. Biol Psychol, 2011, 87(3): 350-357.
[36]
usar-Poli P, Crossley N, Woolley J, et al. Gray matter alterations related to P300 abnormalities in subjects at high risk for psychosis: longitudinal MRI-EEG study[J]. Neuroimage, 2011, 55(1): 320-328.
[37]
Kujala T, Tervaniemi M, Schr?ger E. The mismatch negativity in cognitive and clinical neuroscience: theoretical and methodological considerations[J]. Biol Psychol, 2007, 74(1): 1-19.
[38]
N??t?nen R. The mismatch negativity: a powerful tool for cognitive neuroscience[J]. Ear Hear, 1995, 16(1): 6-18.
Turetsky BI, Calkins ME, Light GA, et al. Neurophysiological endophenotypes of schizophrenia: the viability of selected candidate measures[J]. Schizophr Bull, 2007, 33(1): 69-94.
[41]
Umbricht D, Krljes S. Mismatch negativity in schizophrenia: a meta-analysis[J]. Schizophr Res, 2005, 76(1): 1-23.
[42]
Hermens DF, Ward PB, Hodge MA, et al. Impaired MMN/P3a complex in first-episode psychosis: cognitive and psychosocial associations[J]. Prog Neuropsychopharmacol Biol Psychiatry, 2010, 34(6): 822-829.
[43]
Brockhaus-Dumke A, Tendolkar I, Pukrop R, et al. Impaired mismatch negativity generation in prodromal subjects and patients with schizophrenia[J]. Schizophr Res, 2005, 73(2/3): 297-310.
[44]
Jahshan C, Cadenhead KS, Rissling AJ, et al. Automatic sensory information processing abnormalities across the illness course of schizophrenia[J]. Psychol Med, 2012, 42(1): 85-97.
[45]
Atkinson RJ, Michie PT, Schall U. Duration mismatch negativity and P3a in first-episode psychosis and individuals at ultra-high risk of psychosis[J]. Biol Psychiatry, 2012, 71(2): 98-104.
[46]
Hsieh MH, Shan JC, Huang WL, et al. Auditory event-related potential of subjects with suspected pre-psychotic state and first-episode psychosis[J]. Schizophr Res, 2012, 140(1/2/3): 243-249.
[47]
Bodatsch M, Ruhrmann S, Wagner M, et al. Prediction of psychosis by mismatch negativity[J]. Schizophr Res, 2010, 117(2/3, SI): 244.
[48]
Shaikh M, Valmaggia L, Broome MR, et al. Reduced mismatch negativity predates the onset of psychosis[J]. Schizophr Res, 2012, 134(1): 42-48.
[49]
Adler LE, Pachtman E, Franks RD, et al. Neurophysiological evidence for a defect in neuronal mechanisms involved in sensory gating in schizophrenia[J]. Biol Psychiatry, 1982, 17(6): 639-654.
[50]
Li Z, Zheng B, Deng W, et al. Multi-components of evoked-brain potentials in deficit and nondeficit schizophrenia[J]. Asia Pac Psychiatry, 2013, 5(2): 69-79.