Finney L A, O'Halloran T V. Transition metal speciation in the cell: insights from the chemistry of metal ion receptors[J]. Science, 2003, 300(5621): 931-936
Cross J B, Currier R P, Torraco D J, et al. Killing of bacillus spores by aqueous dissolved oxygen, ascorbic acid, and copper ions[J]. Appl Environ Microbiol, 2003, 69(4): 2245-2252
[4]
Takemura, Y, Satoh M, Satoh K, et al. High dose of ascorbic acid induces cell death in mesothelioma cells[J]. Biochem Biophys Res Commun, 2010, 394(2): 249-253
[5]
Chen Q, Espey M G, Sun A Y, et al. Ascorbate in pharmacologic concentrations selectively generates ascorbate radical and hydrogen peroxide in extracellular fluid in vivo[J]. Proc Natl Acad Sci U S A, 2007, 104(21): 8749-8754
[6]
Clement M V, Ramalingam J, Long L H, et al. The in vitro cytotoxicity of ascorbate depends on the culture medium used to perform the assay and involves hydrogen peroxide[J]. Antioxidants & Redox Signaling, 2001, 3(1): 157-163
[7]
Arakawa N, Nemoto S, Suzuki E, et al. Role of hydrogen peroxide in the inhibitory effect of ascorbate on cell growth[J]. J Nutr Sci Vitaminol (Tokyo), 1994, 40(3): 219-227
[8]
Wang M, Yang G, Feng H, et al. Optimization of Fenton process for decoloration and COD removal in tobacco wastewater and toxicological evaluation of the effluent[J]. Water Sci Technol, 2011, 63(11): 2471-2477
[9]
Klamerth N, Malato S, Aguera A, et al. Treatment of municipal wastewater treatment plant effluents with modified photo-fenton as a tertiary treatment for the degradation of micro pollutants and disinfection[J]. Environ Sci Technol, 2012, 46(5): 2885-2892
Arciello M, Rotilio G, Rossi L. Copper-dependent toxicity in SH-SY5Y neuroblastoma cells involves mitochondrial damage[J]. Biochem Biophys Res Commun, 2005, 327(2): 454-459
[12]
Krumschnabel G, Manzl C, Berger C, et al. Oxidative stress, mitochondrial permeability transition, and cell death in Cu-exposed trout hepatocytes[J]. Toxicol Appl Pharmacol, 2005, 209(1): 62-73
[13]
Rau M A, Whitaker J, Freedman J H, et al. Differential susceptibility of fish and rat liver cells to oxidative stress and cytotoxicity upon exposure to prooxidants[J]. Comparative Biochemistry and Physiology C-Toxicology & Pharmacology, 2004, 137(4): 335-342
[14]
Zastawny T H, Altman S A, Randers-Eichhorn L, et al. DNA base modifications and membrane damage in cultured mammalian cells treated with iron ions[J]. Free Radic Biol Med, 1995, 18(6): 1013-1022
[15]
Valko M, Morris H, Cronin M T. Metals, toxicity and oxidative stress[J]. Curr Med Chem, 2005, 12(10): 1161-1208
[16]
Peebles B, Nagy A, Waldman W J, et al. Fenton activity and cytotoxicity studies of iron-loaded carbon particles[J]. Environ Sci Technol, 2010, 44(17): 6887-6892
[17]
Jomova K, Valko M. Advances in metal-induced oxidative stress and human disease[J]. Toxicology, 2011, 283(2/3): 65-87
[18]
Ozawa T, Hanaki A. Spin-trapping studies on the reactions of Cr(Ⅲ) with hydrogen peroxide in the presence of biological reductants: is Cr(Ⅲ) non-toxic?[J]. Biochem Int, 1990, 22(2): 343-352
[19]
Dehghan M, Akhtar-Danesh N, McMillan C R, et al. Is plasma vitamin C an appropriate biomarker of vitamin C intake? A systematic review and meta-analysis[J]. Nutr J, 2007, 6: 41-53
[20]
Macomber L, Imlay J A. The iron-sulfur clusters of dehydratases are primary intracellular targets of copper toxicity[J]. Proc Natl Acad Sci U S A, 2009, 106(20): 8344-8349
[21]
Banu B S, Ishaq M, Danadevi K, et al. DNA damage in leukocytes of mice treated with copper sulfate[J]. Food and Chemical Toxicology, 2004, 42(12): 1931-1936
