Buckwalter J A. The role of mechanical forces in the initiation and progression of osteoarthritis[J]. HSS J, 2012, 8(1): 37-38. [2]Felson D T. Osteoarthritis as a disease of mechanics[J]. Osteoarthritis Cartilage,2013, 21(1): 10-15. [3]Griffin T M, Guilak F. The role of mechanical loading in the onset and progression of osteoarthritis[J]. Exerc Sport Sci Rev, 2005, 33(4): 195-200. [4]Guilak F. Biomechanical factors in osteoarthritis[J]. Best Pract Res Clin Rheumatol, 2011,25(6): 815-823. [5]Trickey W R, Lee G M, Guilak F. Viscoelastic properties of chondrocytes from normal and osteoarthritic human cartilage[J]. J Orthop Res, 2000, 18(6): 891-898. [6]Blain E J, Gilbert S J, Hayes A J, et al. Disassembly of the vimentin cytoskeleton disrupts articular cartilage chondrocyte homeostasis[J]. Matrix Biol, 2006, 25(7): 398-408. [7]Duan W, Wei L, Cao X, et al. Effect of the disruption of three cytoskeleton components on chondrocyte metabolism in rabbit knee cartilage[J]. Chin Med J (Engl), 2014, 127(21): 3764-3770. [8]Trickey W R, Vail T P, Guilak F. The role of the cytoskeleton in the viscoelastic properties of human articular chondrocytes[J]. J Orthop Res, 2004, 22(1):? 131-139. [9]Haudenschild D R, Chen J, Pang N, et al. Vimentin contributes to changes in chondrocyte stiffness in osteoarthritis[J]. J Orthop Res, 2011,29(1): 20-25. [10]Chahine N O, Blanchette C, Thomas C B, et al. Effect of age and cytoskeletal elements on the indentation-dependent mechanical properties of chondrocytes[J]. PLoS One, 2013, 8(4): e61651. [11]Lambrecht S, Verbruggen G, Verdonk P C, et al. Differential proteome analysis of normal and osteoarthritic chondrocytes reveals distortion of vimentin network in osteoarthritis[J]. Osteoarthritis Cartilage, 2008,16(2): 163-173. [12]Capin-Gutierrez N, Talamas-Rohana P, Gonzalez-Robles A, et al. Cytoskeleton disruption in chondrocytes from a rat osteoarthrosic (OA)-induced model:? its potential role in OA pathogenesis[J]. Histol Histopathol, 2004, 19(4): 1125-1132. [13]Chen J L, Duan L, Zhu W, et al. Extracellular matrix production ?in vitro? in cartilage tissue engineering[J]. J Transl Med, 2014, 12: 88. [14]Demoor M, Ollitrault D, Gomez-Leduc T, et al. Cartilage tissue engineering:? molecular control of chondrocyte differentiation for proper cartilage matrix reconstruction[J]. Biochim Biophys Acta, 2014, 1840(8):? 2414-2440. [15]谭洪波,段小军,杨柳,等.透明软骨材料经脱细胞处理后细胞外基质成分变化的比较[J].第三军医大学学报, 2013, 35(19): 2028-2032. [16]Shen B, Wei A, Tao H, et al. BMP-2 enhances TGF-beta3-mediated chondrogenic differentiation of human bone marrow multipotent mesenchymal stromal cells in alginate bead culture[J]. Tissue Eng Part A, 2009, 15(6):? 1311-1320. [17]Haleem A M, Chu C R. Advances in tissue engineering techniques for articular cartilage repair[J]. Oper Tech Orthop, 2010,20(2): 76-89.
[2]
吕伟宏,田怀军.低氧对大鼠睾丸支持细胞形态结构与存活率的影响[J].第三军医大学学报,2007,29(01):65. LU Wei-hong,TIAN Huai-jun.Effects of hypoxia on morphology and survival of rat Sertoli cells[J].J Third Mil Med Univ,2007,29(21):65. [2]罗果,保玉心,李晋.不同固定液对细胞骨架荧光染色标记效果的影响[J].第三军医大学学报,2011,33(07):753.