Nanri A, Mizoue T, Takahashi Y,et al. Soy product and isoflavone intakes are associated with a lower risk of type 2 diabetes in overweight Japanese women[J]. J Nutr, 2010, 140(3): 580-586. [2]Valsecchi A E, Franchi S, Panerai A E,et al. The soy isoflavone genistein reverses oxidative and inflammatory state, neuropathic pain, neurotrophic and vasculature deficits in diabetes mouse model[J]. Eur J Pharmacol, 2011, 650(2/3): 694-702. [3]Cho K W, Lee O H, Banz W J,et al. Daidzein and the daidzein metabolite, equol, enhance adipocyte differentiation and PPAR? transcriptional activity[J]. J Nutr Biochem, 2010, 21(9): 841-847. [4]Zhang Y B, Li L N, Zhao X Y,et al. Effect of soy isoflavone crude extract supplementation on high fat diet-induced insulin resistance in ovariectomized rats[J]. Biomed Environ Sci, 2014, 27(1): 49-51. [5]Ferguson J F, Ryan M F, Gibney E R,et al. Dietary isoflavone intake is associated with evoked responses to inflammatory cardiometabolic stimuli and improved glucose homeostasis in healthy volunteers[J]. Nutr Metab Cardiovasc Dis, 2014, 24(9): 996-1003.〖HJ2mm〗 [6]Virk-Baker M K, Barnes S, Krontiras H,et al. S-(-)equol producing status not associated with breast cancer risk among low isoflavone-consuming US postmenopausal women undergoing a physician-recommended breast biopsy[J]. Nutr Res, 2014, 34(2): 116-125. [7]Brown N M, Galandi S L, Summer S S,et al. S-(-)equol production is developmentally regulated and related to early diet composition[J]. Nutr Res, 2014, 34(5): 401-409. [8]Setchell K D, Clerici C. Equol: history, chemistry, and formation[J]. J Nutr, 2010, 140(7): 1355S-1362S. [9]刘凯, 陈卡, 周蕊, 等. S-Equol对高糖培养HepG2细胞胰岛素敏感性及IRS-1表达的影响[J]. 现代生物医学进展, 2013, 13(7): 1210-1213. [10]陈莹晖, 李裕明, 林松挺, 等. 高糖诱导氧化应激对胰岛细胞功能的影响[J]. 海南医学院学报, 2012, 18(1): 12-14. [11]王萌, 任国峰. 雌马酚健康效应的研究进展[J]. 卫生研究, 2012, 41(5): 874-876. [12]Marin-Juez R, Rovira M, Crespo D,et al. GLUT2-mediated glucose uptake and availability are required for embryonic brain development in zebrafish[J]. J Cereb Blood Flow Metab, 2014, [Epub ahead of print]. [13]Al-Haggar M, Sakamoto O, Shaltout A,et al. Mutation analysis of the GLUT2 gene in three unrelated Egyptian families with Fanconi-Bickel syndrome: revisited gene atlas for renumbering[J]. Clin Exp Nephrol, 2012, 16(4): 604-610. [14]Chankiewitz E, Peschke D, Herberg L,et al. Did the gradual loss of GLUT2 cause a shift to diabetic disorders in the New Zealand obese mouse (NZO/Hl)?[J]. Exp Clin Endocrinol Diabetes, 2006, 114(5): 262-269. [15]Kiraly M A, Bates H E, Kaniuk N A,et al. Swim training prevents hyperglycemia in ZDF rats: mechanisms involved in the partial maintenance of beta-cell function[J]. Am J Physiol Endocrinol Metab, 2008, 294(2): E271-E283. [16]Brand M D, Parker N, Affourtit C,et al. Mitochondrial uncoupling protein 2 in pancreatic beta-cells[J]. Diabetes Obes Metab, 2010, 12 Suppl 2: 134-140. [17]Affourtit C, Jastroch M, Brand M D. Uncoupling protein-2 attenuates glucose-stimulated insulin secretion in INS-1E insulinoma cells by lowering mitochondrial reactive oxygen species[J]. Free Radic Biol Med, 2011, 50(5): 609-616. [18]Lee S C, Robson-Doucette C A, Wheeler M B. Uncoupling protein 2 regulates reactive oxygen species formation in islets and influences susceptibility to diabetogenic action of streptozotocin[J]. J Endocrinol, 2009, 203(1): 33-43.
[2]
姜友昭,张玲,郑江,等.不同葡萄糖浓度对INS-1细胞PPARδ表达的影响[J].第三军医大学学报,2007,29(03):247. JIANG You-zhao,ZHANG Ling,ZHENG Jiang,et al.Effects of different concentrations of glucose on PPARδ expression in INS-1 cells[J].J Third Mil Med Univ,2007,29(05):247. [2]姜友昭,张玲,周艳,等.小鼠PPARδ腺病毒载体的构建及其在INS-1细胞中的表达[J].第三军医大学学报,2007,29(01):68. JIANG You-zhao,ZHANG Ling,ZHOU Yan,et al.Construction of mouse PPARδ adenovirus vector and its expression in INS-1 cells[J].J Third Mil Med Univ,2007,29(05):68.