Lozano R, Naghavi M, Foreman K, et al. Global and regional mortality from 235 causes of death for 20 age groups in 1990 and 2010: a systematic analysis for the global burden of disease study 2010[J]. Lancet,2012,380(9859): 2095.
[4]
Brook R D, Rajagopalan S, Pope C A, et al. Particulate matter air pollution and cardiovascular disease: an update to the scientific statement from the American heart association[J]. Circulation,2010,121(21): 2331.
[5]
Pope C A, Burnett R T, Thurston G D, et al. Cardiovascular mortality and long-term exposure to particulate air pollution: epidemiological evidence of general pathophysiological pathways of disease[J]. Circulation,2004,109(1): 71.
[6]
Pope C A, Muhlestein J B, May H T, et al. Ischemic heart disease events triggered by short-term exposure to fine particulate air pollution[J]. Circulation,2006,114(23): 2443.
Poursafa P, Kelishadi R. Air pollution, platelet activation and atherosclerosis[J]. Inflamm Allergy Drug Targets,2010,9(5): 387.
[10]
Sun Q, Yue P, Kirk R I, et al. Ambient air particulate matter exposure and tissue factor expression in atherosclerosis[J]. Inhal Toxicol,2008,20(2): 127.
[11]
Franchini M, Mannucci P M. Thrombogenicity and cardiovascular effects of ambient air pollution[J]. Blood,2011,118(9): 2405.
[12]
Sun Q, Wang A, Jin X, et al. Long-term air pollution exposure and acceleration of atherosclerosis and vascular inflammation in an animal model[J]. J Amer Med Assoc, 2005,294(23): 3003.
[13]
Nadadur S S, Haykal-Coates N, Mudipalli A, et al. Endothelial effects of emission source particles: acute toxic response gene expression profiles[J]. Toxicol In Vitro, 2009,23(1): 67.
[14]
Montiel-Davalos A, Ibarra-Sanchez M J, Ventura-Gallegos J L, et al. Oxidative stress and apoptosis are induced in human endothelial cells exposed to urban particulate matter[J]. Toxicol In Vitro, 2010,24(1): 135.
[15]
Shannahan J H, Kodavanti U P, Brown J M. Manufactured and airborne nanoparticle cardiopulmonary interactions: a review of mechanisms and the possible contribution of mast cells[J]. Inhal Toxicol,2012,24(5): 320.
[16]
Barbieri S S, Weksler B B. Tobacco smoke cooperates with interleukin-1beta to alter beta-catenin trafficking in vascular endothelium resulting in increased permeability and induction of cyclooxygenase-2 expression in vitro and in vivo[J]. FASEB J, 2007,21(8): 1831.
Li R, Navab M, Pakbin P, et al. Ambient ultrafine particles alter lipid metabolism and HDL anti-oxidant capacity in LDLR-null mice[J]. J Lipid Res,2013,54(6): 1608.
[19]
Li R, Ning Z, Majumdar R, et al. Ultrafine particles from diesel vehicle emissions at different driving cycles induce differential vascular pro-inflammatory responses: implication of chemical components and NF-kappaB signaling[J]. Part Fibre Toxicol,2010(7): 6.
[20]
Totlandsdal A I, Skomedal T, Lag M, et al. Pro-inflammatory potential of ultrafine particles in mono- and co-cultures of primary cardiac cells[J]. Toxicol,2008,247(1): 23.
[21]
Sutterwala F S, Ogura Y, Flavell R A. The inflammasome in pathogen recognition and inflammation[J]. J Leukoc Biol,2007,82(2): 259.
[22]
Rajamaki K, Lappalainen J, Oorni K, et al. Cholesterol crystals activate the NLRP3 inflammasome in human macrophages: a novel link between cholesterol metabolism and inflammation[J]. PLoS ONE,2010,5(7): e11765.
[23]
Duewell P, Kono H, Rayner K J, et al. NLRP3 inflammasomes are required for atherogenesis and activated by cholesterol crystals[J]. Nature,2010,464(7293): 1357.
[24]
Li Y, Xu S, Jiang B, et al. Activation of sterol regulatory element binding protein and NLRP3 inflammasome in atherosclerotic lesion development in diabetic pigs[J]. PLoS ONE,2013,8(6): e67532.
[25]
Dunne A. Inflammasome activation: from inflammatory disease to infection[J]. Biochem Soc Trans,2011,39(2): 669.
[26]
Houston M C. The role of mercury and cadmium heavy metals in vascular disease, hypertension, coronary heart disease, and myocardial infarction[J]. Altern Ther Health Med,2007, 13(2): S128.
[27]
Nogueira J B. Air pollution and cardiovascular disease[J]. Rev Port Cardiol, 2009,28(6): 715.
[28]
Watterson T L, Sorensen J, Martin R, et al. Effects of PM2.5 collected from Cache Valley Utah on genes associated with the inflammatory response in human lung cells[J]. J Toxicol Environ Health A,2007,70(20): 1731.
[29]
Painschab M S, Davila-Roman V G, Gilman R H, et al. Chronic exposure to biomass fuel is associated with increased carotid artery intima-media thickness and a higher prevalence of atherosclerotic plaque[J]. Heart,2013,99(14): 984.
[30]
Tian L, Zhang W, Lin Z Q, et al. Impact of traffic emissions on local air quality and the potential toxicity of traffic-related particulates in Beijing, China[J]. Biomed Environ Sci,2012,25(6): 663.
[31]
Chen T, Jia G, Wei Y, et al. Beijing ambient particle exposure accelerates atherosclerosis in ApoE knockout mice[J]. Toxicol Lett,2013,223(2): 146.
[32]
Wan Q, Cui X, Shao J, et al. Beijing ambient particle exposure accelerates atherosclerosis in ApoE knockout mice by upregulating visfatin expression[J]. Cell Stress Chaperones,2014,PMID:24523034.
Sandanger O, Ranheim T, Vinge L E, et al. The NLRP3 inflammasome is up-regulated in cardiac fibroblasts and mediates myocardial ischaemia-reperfusion injury[J]. Cardiovasc Res,2013,99(1): 164.
[36]
Menu P, Pellegrin M, Aubert J F, et al. Atherosclerosis in ApoE-deficient mice progresses independently of the NLRP3 inflammasome[J]. Cell Death Dis,2011,2: e137.
[37]
Hirota J A, Hirota S A, Warner S M, et al. The airway epithelium nucleotide-binding domain and leucine-rich repeat protein 3 inflammasome is activated by urban particulate matter[J]. J Allergy Clin Immunol,2012, 129(4): 1116.
[38]
Bengalli R, Molteni E, Longhin E, et al. Release of IL-1 beta triggered by Milan summer PM10: molecular pathways involved in the cytokine release[J]. Biomed Res Int,2013,doi: 158093.
