Qiu G, Yan P, Shao W, et al. Two new sesquiterpenoids including a sesquiterpenoid lactam from Curcuma wenyujin[J]. Chem Pharm Bull (Tokyo), 2013,61(9):983.
[5]
Yin G P, An Y W, Hu G, et al. Three new guaiane sesquiterpene lactones from rhizomes of Curcuma wenyujin[J]. J Asian Nat Prod Res, 2013, 15(7):723.
Rungseesantivanon S, Thenchaisri N. Curcumin supplementation could improve diabetes-induced endothelial dysfunction associated with decreased vascular superoxide production and PKC inhibition[J]. BMC Complement Alternat, 2010, 10:57.
[9]
Fang X D, Yang F, Zhu L, et al. Curcumin ameliorates high glucose-induced acute vascular endothelial dysfunction in rat thoracic aorta [J]. Clin Exp Pharmacol Physiol, 2009,36(12):1177.
[10]
Xu P, Yao Y, Guo P, et al. Curcumin protects rat heart mitochondria against anoxia-reoxygenation induced oxidative injury [J]. Can J Physiol Pharmacol, 2013,91(9):715.
[11]
Akazawa N, Choi Y, Miyaki A, et al. Curcumin ingestion and exercise training improve vascular endothelial function in postmenopausal women[J]. Nutr Res, 2012, 32(10):795.
[12]
Charoenwanthanang P, Lawanprasert S, Phivthong-Ngam L, et al. Effects of Curcuma comosa on the expression of atherosclerosis-related cytokine genes in rabbits fed a high-cholesterol diet[J]. J Ethnopharmacol, 2011,134(3):608.
[13]
Panicker S R, Kartha C C. Curcumin attenuates glucose-induced monocyte chemoattractant protein-1 synthesis in aortic endothelial cells by modulating the nuclear factor-kappaB pathway[J]. Pharmacology, 2010, 85(1):18.
[14]
Wang J, Dong S. ICAM-1 and IL-8 are expressed by DEHP and suppressed by curcumin through ERK and p38 MAPK in human umbilical vein endothelial cells[J]. Inflammation, 2012, 35(3):859.
[15]
Angel-Morales G, Noratto G, Mertens-Talcott S U. Standardized curcuminoid extract (Curcuma longa l.) decreases gene expression related to inflammation and interacts with associated micro RNAs in human umbilical vein endothelial cells (HUVEC) [J]. Food Funct, 2012, 3(12):1286.
[16]
Lyss G, Schmidt T J, Merfort I, et al. Helenalin, an antiinflammatory sesquiterpene lactone from Arnica, selectively inhibits transcription factor NF-κB[J]. Biol Chem, 1997, 378:951.
Liu Y, Dolence J, Ren J, et al. Inhibitory effect of dehydrozingerone on vascular smooth muscle cell function[J]. J Cardiovasc Pharmacol,2008,52(5):422.
[19]
Shen M J, Lin H Y, Yang Y H, et at. Demethoxycurcumin, a major active curcuminoid from Curcuma longa, suppresses balloon injury induced vascular smooth muscle cell migration and neointima formation:an in vitro and in vivo study [J]. Mol Nutr Food Res, 2013, 57(9):1586.
[20]
Zhao F H, Liu J G, Wang X, et al. Long-term effect of stent coating with zedoary essential components on neointimal formation in the porcine coronary artery[J]. Chin J Integr Med, 2013,19(10):771.
[21]
Inayama S, Gao J F, Harimaya K, et al. The absolute stereostructure of curcumol isolated from Curcuma wenyujin[J]. Chem Pharm Bull (Tokyo), 1984,32(9):3783.
Jiang H S, Nam H Y, Kim J M, et al. Effects of curcumin for preventing restenosis in a hypercholesterolemic rabbit iliac artery stent model[J]. Catheter Cardiovasc Interv, 2009,74(6):881.
Xia Q, Wang X, Xu D J, et al. Inhibition of platelet aggregation by curdione from Curcuma wenyujin essential oil [J]. Thromb Res, 2012, 130(3):409.
[27]
Chen C C, Chen Y, Hsi Y T, et al. Chemical constituents and anticancer activity of Curcuma zedoaria roscoe essential oil against non-small cell lung carcinoma cells in vitro and in vivo[J]. J Agric Food Chem, 2013,61(47):11418.
[28]
Shi H, Tan B, Ji G, et al. Zedoary oil (Ezhu You) inhibits proliferation of AGS cells[J]. Chin Med, 2013,8(1):13.
Lim C B, Ky N, Ng H M, et al. Curcuma wenyujin extract induces apoptosis and inhibits proliferation of human cervical cancer cells in vitro and in vivo[J]. Integr Cancer Ther, 2010,9(1):36.
[31]
Singh V, Jain M, Misra A, et al. Curcuma oil ameliorates hyperlipidaemia and associated deleterious effects in golden Syrian hamsters[J]. Br J Nutr, 2013, 110(3):437.
[32]
Lee H S, Lee M J, Kim H,et al. Curcumin inhibits TNFalpha-induced lectin-like oxidised LDL receptor-1 (LOX-1) expression and suppresses the inflammatory response in human umbilical vein endothelial cells (HUVECs) by an antioxidant mechanism[J]. J Enzyme Inhib Med Chem, 2010,25(5):720.