1 National Institutes of Health. Clinical guidelines on the identification, evaluation, and treatment of overweight and obesity in adults-the evidence report. Obes Res, 1998, 6 (Suppl 2): 51S-209S
[2]
2 Kopelman P G. Obesity as a medical problem. Nature, 2000, 404: 635-643
[3]
3 Dulloo A G, Seydoux J, Girardier L, et al. Green tea and thermogenesis: interactions between catechin-polyphenols, caffeine and sympathetic activity. Int J Obes Relat Metab Disord, 2000, 24: 252-258
[4]
4 Ashida H, Furuyashiki T, Nagayasu H, et al. Anti-obesity actions of green tea: possible involvements in modulation of the glucose uptake system and suppression of the adipogenesis-related transcription factors. Biofactors, 2004, 22: 135-140
[5]
5 Furuyashiki T, Nagayasu H, Aoki Y, et al. Tea catechin suppresses adipocyte differentiation accompanied by down-regulation of PPARgamma2 and C/EBPalpha in 3T3-L1 cells. Biosci Biotechnol Biochem, 2004, 68: 2353-2359
[6]
6 Tokimitsu I. Effects of tea catechins on lipid metabolism and body fat accumulation. Biofactors, 2004, 22: 141-143
[7]
7 Dufresne C J, Farnworth E R. A review of latest research findings on the health promotion properties of tea. J Nutr Biochem, 2001, 12: 404-421
[8]
24 DeFronzo R A. Dysfunctional fat cells, lipotoxicity and type 2 diabetes. Int J Clin Pract Suppl, 2004, (143): 9-21
[9]
25 Spiegelman B M, Flier J S. Adipogenesis and obesity: rounding out the big picture. Cell, 1996, 87: 377-389
[10]
26 Tiraby C, Tavernier G, Lefort C, et al. Acquirement of brown fat cell features by human white adipocytes. J Biol Chem, 2003, 278: 33370- 33376
[11]
27 Abe K, Okada N, Tanabe H, et al. Effects of chronic ingestion of catechin-rich green tea on hepatic gene expression of gluconeogenic enzymes in rats. Biomed Res, 2009, 30: 25-29
[12]
28 Zhao B L, Li X J, He R G, et al. Scavenging effect of extracts of green tea and natural antioxidants on active oxygen radicals. Cell Biophys, 1989, 14: 175-185
[13]
29 Guo Q, Zhao B, Li M, et al. Studies on protective mechanisms of four components of green tea polyphenols against lipid peroxidation in synaptosomes. Biochim Biophys Acta, 1996, 1304: 210-222
[14]
30 Guo Q, Zhao B, Shen S, et al. ESR study on the structure-antioxidant activity relationship of tea catechins and their epimers. Biochim Biophys Acta, 1999, 1427: 13-23
[15]
31 Zhao B, Guo Q, Xin W. Free radical scavenging by green tea polyphenols. Methods Enzymol, 2001, 335: 217-231
[16]
32 Furukawa S, Fujita T, Shimabukuro M, et al. Increased oxidative stress in obesity and its impact on metabolic syndrome. J Clin Invest, 2004, 114: 1752-1761
[17]
33 Bondia-Pons I, Ryan L, Martinez J A. Oxidative stress and inflammation interactions in human obesity. J Physiol Biochem, 2012, 68: 701- 711
[18]
34 Keaney J F, Larson M G, Vasan R S, et al. Obesity and systemic oxidative stress: clinical correlates of oxidative stress in the Framingham Study. Arterioscler Thromb Vasc Biol, 2003, 23: 434-439
[19]
35 Nakai M, Fukui Y, Asami S, et al. Inhibitory effects of oolong tea polyphenols on pancreatic lipase in vitro. J Agric Food Chem, 2005, 53: 4593-4598
[20]
8 Yang M, Wang C, Chen H. Green, oolong and black tea extracts modulate lipid metabolism in hyperlipidemia rats fed high-sucrose diet. J Nutr Biochem, 2001, 12: 14-20
[21]
9 Klaus S, Pultz S, Thone-Reineke C, et al. Epigallocatechin gallate attenuates diet-induced obesity in mice by decreasing energy absorption and increasing fat oxidation. Int J Obes (Lond), 2005, 29: 615-623
[22]
10 Nagao T, Komine Y, Soga S, et al. Ingestion of a tea rich in catechins leads to a reduction in body fat and malondialdehyde-modified LDL in men. Am J Clin Nutr, 2005, 81: 122-129
[23]
11 Wang X, Song K S, Guo Q X, et al. The galloyl moiety of green tea catechins is the critical structural feature to inhibit fatty-acid synthase. Biochem Pharmacol, 2003, 66: 2039-2047
[24]
12 Mori M, Hasegawa N. Superoxide dismutase activity enhanced by green tea inhibits lipid accumulation in 3T3-L1 cells. Phytother Res, 2003, 17: 566-567
[25]
13 Choo J J. Green tea reduces body fat accretion caused by high-fat diet in rats through beta-adrenoceptor activation of thermogenesis in brown adipose tissue. J Nutr Biochem, 2003, 14: 671-676
[26]
14 Kersten S, Desvergne B, Wahli W. Roles of PPARs in health and disease. Nature, 2000, 405: 421-424
[27]
15 Rosen E D, Spiegelman B M. PPARgamma: a nuclear regulator of metabolism, differentiation, and cell growth. J Biol Chem, 2001, 276: 37731-37734
[28]
16 Wang Y X, Lee C H, Tiep S, et al. Peroxisome-proliferator-activated receptor delta activates fat metabolism to prevent obesity. Cell, 2003, 113: 159-170
[29]
17 Mukherjee R, Jow L, Croston G E, et al. Identification, characterization, and tissue distribution of human peroxisome proliferator-activated receptor (PPAR) isoforms PPARgamma2 versus PPARgamma1 and activation with retinoid X receptor agonists and antagonists. J Biol Chem, 1997, 272: 8071-8076
[30]
18 Walczak R, Tontonoz P. Setting fat on fire. Nat Med, 2003, 9: 1348-1349
[31]
19 Lee K. Transactivation of peroxisome proliferator-activated receptor alpha by green tea extracts. J Vet Sci, 2004, 5: 325-330
[32]
20 Shimabukuro M, Koyama K, Chen G, et al. Direct antidiabetic effect of leptin through triglyceride depletion of tissues. Proc Natl Acad Sci USA, 1997, 94: 4637-4641
[33]
21 Pappachan J M, Chacko E C, Arunagirinathan G, et al. Management of hypertension and diabetes in obesity: non-pharmacological measures. Int J Hypertens, 2011, 2011: 398065
[34]
22 Kao Y H, Hiipakka R A, Liao S. Modulation of endocrine systems and food intake by green tea epigallocatechin gallate. Endocrinology, 2000, 141: 980-987
[35]
23 Chen N, Bezzina R, Hinch E, et al. Green tea, black tea, and epigallocatechin modify body composition, improve glucose tolerance, and differentially alter metabolic gene expression in rats fed a high-fat diet. Nutr Res, 2009, 29: 784-793