Interaction of the Yersinia pestis type III regulatory proteins LcrG and LcrV occurs at a hydrophobic interface: correction

This error changes our interpretation of the experiment that tested the trans-complementation of an lcrG strain of Yersinia pestis with PcrG. We reported that our clone of pcrG on plasmid pJM132 could not complement an lcrG strain of Y. pestis (Fig. 5). Discovery of the sequencing error led us to re-sequence several of our pcrG constructs. We found that our original subclone, pJM132, contained a deletion of an adenine residue between the ribosome-binding site and the initiating ATG of pcrG. Our complementing "mutant" on pJM133 (Fig. 5) contains the deleted adenine. This result suggests that the failure of pJM132 to complement is due to an expression problem. Therefore, we now conclude that pcrG from P. aeruginosa PAO1 complements an lcrG strain of Y. pestis. We apologize for the error.