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- 2019
Gain-of-Function Mutations of SLC16A11 Contribute to the Pathogenesis of Type 2 DiabetesDOI: https://doi.org/10.1016/j.celrep.2018.12.100 Abstract: ？ Slc16a11 knockout mice have minimal metabolic consequences ？ Mice carrying SLC16A11 mutations have disrupted lipid metabolism and fatty liver ？ Mutant SLC16A11 upregulates lipin 1 and increases liver triglyceride accumulatio
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