全部 标题 作者
关键词 摘要

OALib Journal期刊
ISSN: 2333-9721
费用:99美元

查看量下载量

相关文章

更多...

血管外膜细胞成分影响动脉粥样硬化发生发展研究概述

Keywords: 动脉粥样硬化,血管外膜,成纤维细胞,炎性细胞,滋养血管,血管周围神经

Full-Text   Cite this paper   Add to My Lib

Abstract:

血管外膜在动脉粥样硬化(atherosclerosis,As)发生发展中的作用不容忽视。血管外膜分布的成纤维细胞、肥大细胞、树突状细胞以及滋养血管、血管相关淋巴组织和血管周围神经等均可参与As发生发展进程。该文就近10年内,血管外膜组织细胞成分在As过程中的变化及对As发生发展的影响方面的研究状况和最新进展做一综述。

References

[1]  Dunkern T R, Feurstein D, Rossi G A, et al. Inhibition of TGF-beta induced lung fibroblast to myofibroblast conversion by phosphodiesterase inhibiting drugs and activators of soluble guanylyl cyclase[J]. Eur J Pharmacol, 2007, 572(1):12.
[2]  Maiellaro K, Taylor W R. The role of the adventitia in vascular inflammation[J]. Cardiovasc Res, 2007, 75(4): 640.
[3]  高琳. P38 MAPK在血管外膜损伤后氧化应激中的作用及药物干预研究. 上海:第二军医大学,2008.
[4]  才晓君. 脂联素通过血管外膜干预脂多糖介导的氮氧化应激的实验研究. 济南:山东大学,2009.
[5]  Salisbury E, Hipp J, Olmsted-Davis E A, et al. Histologic identification of brown adipose and peripheral nerve involvement in human atherosclerotic vessels[J]. Hum Pathol, 2012, 43(12):2213.
[6]  陈玮, 梁春, 吴宗贵. 通心络对动脉粥样硬化中交感神经调节的影响. 上海:第一届全国中西医结合心血管病中青年医师论坛, 2008.
[7]  Bot I, de Jager S C, Bot M, et al. The neuropeptide substance P mediates adventitial mast cell activation and induces intraplaque hemorrhage in advanced atherosclerosis[J]. Circ Res, 2010, 106(1): 89.
[8]  张璐,蒋跃绒,薛梅,等.辛伐他汀对兔动脉粥样硬化斑块稳定性及斑块内血管新生的影响[J].科学通报,2009,15: 2228.
[9]  鹿克风,吕琳,朱兴雷.氟伐他汀对动脉粥样硬化血管外膜炎症的影响[J].中国老年学杂志,2007,8: 718.
[10]  Drinane M, Mollmark J, Zagorchev L,et al. The antiangiogenic activity of rPAI-1(23) inhibits vasa vasorum and growth of atherosclerotic plaque[J]. Circ Res, 2009,104(3):337.
[11]  鹿燕敏,李兰芳,霍海如,等. 4种活血化瘀中药有效成分对血管紧张素Ⅱ诱导的血管外膜成纤维细胞增殖及胶原合成的影响[J]. 中国实验方剂学杂志,2008(11):68.
[12]  任敏. 芩丹胶囊对血管外膜成纤维细胞TGF-β1/Smad信号转导通路影响的研究. 济南:山东大学,2011.
[13]  沈凯, 杨喆娟, 袁国裕, 等. 紫杉醇对血管外膜成纤维细胞表型转化的影响[J]. 中国临床药理学杂志, 2010(8): 615.
[14]  沈伟,范维琥,施海明,等.红景天对兔动脉粥样硬化斑块内血管内皮细胞生长因子表达及血管新生的影响[J].中国中西医结合杂志,2008,28(11):1022.
[15]  Watanabe M, Sangawa A, Sasaki Y, et al. Distribution of inflammatory cells in adventitia changed with advancing atherosclerosis of human coronary artery[J]. J Atheroscler Thromb, 2007, 14(6): 325.
[16]  汤月霞, 梁春, 吴宗贵. 氧化应激在血管外膜损伤致内膜病变形成中的作用及干预研究[J]. 上海医学, 2007(S1):9.
[17]  关启刚,曾定尹,程颖,等. 白介素-1β外膜介导小型猪冠状动脉粥样硬化病变的研究[J]. 天津医药, 2009 (3): 199.
[18]  Maiellaro K, Taylor W R. The role of the adventitia in vascular inflammation[J]. Cardiovasc Res, 2007, 75(4): 640.
[19]  Cojocaru E, Trandafirescu M, Leon M, et al. Immunohistochemical expression of anti-CD68 antibody in atherosclerotic plaque[J], Rom J Morphol Embryol, 2012,53(1):61.
[20]  秦蒙. 血管损伤后重塑机制及治疗药物开发的研究. 北京:中国协和医科大学,2010.
[21]  Tavora F, Kutys R, Li L, et al. Adventitial lymphocytic inflammation in human coronary arteries with intimal atherosclerosis[J]. Cardiovasc Pathol, 2010,19(3):e61.
[22]  Zubair A, Mubarik A, Jamal S, et al. A comparative analysis of coronary adventitial T-lymphocytes——an autopsy study[J]. J Ayub Med Coll Abbottabad, 2010, 22(1):42.
[23]  刘子懿, 孔炜. 血管外膜在动脉粥样硬化中的作用[J]. 生理科学进展, 2010(3):177.
[24]  Kyaw T, Tipping P, Toh B H, et al. Current understanding of the role of B cell subsets and intimal and adventitial B cells in atherosclerosis[J]. Curr Opin Lipidol, 2011, 22(5):373.
[25]  Kyaw T, Tay C, Khan A, et al. Conventional B2B cell depletion ameliorates whereas its adoptive transfer aggravates atherosclerosis[J]. J Immunol, 2010, 185(7): 4410.
[26]  Koltsova E K, Ley K. How dendritic cells shape atherosclerosis[J]. Trends Immunol, 2011, 32(11): 540.
[27]  Swedenborg J,Mayranpaq M I,Kovanen P T. Mast cells: important players in the orchestrated pathogenesis of abdominal aortic aneurysms[J]. Arterioscler Thromb Vasc Biol,2011, 31(4): 734.
[28]  Shi G P, Lindholt J S. Mast cells in abdominal aortic aneurysms[J]. Curr Vasc Pharmacol, 2012,Jun 22..
[29]  Gossl M, Versari D, Mannheim D, et al. Increased spatial vasa vasorum density in the proximal LAD in hypercholesterolemia-implications for vulnerable plaque-development[J]. Atherosclerosis, 2007, 192:246.

Full-Text

Contact Us

service@oalib.com

QQ:3279437679

WhatsApp +8615387084133