Dunkern T R, Feurstein D, Rossi G A, et al. Inhibition of TGF-beta induced lung fibroblast to myofibroblast conversion by phosphodiesterase inhibiting drugs and activators of soluble guanylyl cyclase[J]. Eur J Pharmacol, 2007, 572(1):12.
[2]
Maiellaro K, Taylor W R. The role of the adventitia in vascular inflammation[J]. Cardiovasc Res, 2007, 75(4): 640.
Salisbury E, Hipp J, Olmsted-Davis E A, et al. Histologic identification of brown adipose and peripheral nerve involvement in human atherosclerotic vessels[J]. Hum Pathol, 2012, 43(12):2213.
Bot I, de Jager S C, Bot M, et al. The neuropeptide substance P mediates adventitial mast cell activation and induces intraplaque hemorrhage in advanced atherosclerosis[J]. Circ Res, 2010, 106(1): 89.
Drinane M, Mollmark J, Zagorchev L,et al. The antiangiogenic activity of rPAI-1(23) inhibits vasa vasorum and growth of atherosclerotic plaque[J]. Circ Res, 2009,104(3):337.
Watanabe M, Sangawa A, Sasaki Y, et al. Distribution of inflammatory cells in adventitia changed with advancing atherosclerosis of human coronary artery[J]. J Atheroscler Thromb, 2007, 14(6): 325.
Maiellaro K, Taylor W R. The role of the adventitia in vascular inflammation[J]. Cardiovasc Res, 2007, 75(4): 640.
[19]
Cojocaru E, Trandafirescu M, Leon M, et al. Immunohistochemical expression of anti-CD68 antibody in atherosclerotic plaque[J], Rom J Morphol Embryol, 2012,53(1):61.
[20]
秦蒙. 血管损伤后重塑机制及治疗药物开发的研究. 北京:中国协和医科大学,2010.
[21]
Tavora F, Kutys R, Li L, et al. Adventitial lymphocytic inflammation in human coronary arteries with intimal atherosclerosis[J]. Cardiovasc Pathol, 2010,19(3):e61.
[22]
Zubair A, Mubarik A, Jamal S, et al. A comparative analysis of coronary adventitial T-lymphocytes——an autopsy study[J]. J Ayub Med Coll Abbottabad, 2010, 22(1):42.
[23]
刘子懿, 孔炜. 血管外膜在动脉粥样硬化中的作用[J]. 生理科学进展, 2010(3):177.
[24]
Kyaw T, Tipping P, Toh B H, et al. Current understanding of the role of B cell subsets and intimal and adventitial B cells in atherosclerosis[J]. Curr Opin Lipidol, 2011, 22(5):373.
[25]
Kyaw T, Tay C, Khan A, et al. Conventional B2B cell depletion ameliorates whereas its adoptive transfer aggravates atherosclerosis[J]. J Immunol, 2010, 185(7): 4410.
[26]
Koltsova E K, Ley K. How dendritic cells shape atherosclerosis[J]. Trends Immunol, 2011, 32(11): 540.
[27]
Swedenborg J,Mayranpaq M I,Kovanen P T. Mast cells: important players in the orchestrated pathogenesis of abdominal aortic aneurysms[J]. Arterioscler Thromb Vasc Biol,2011, 31(4): 734.
[28]
Shi G P, Lindholt J S. Mast cells in abdominal aortic aneurysms[J]. Curr Vasc Pharmacol, 2012,Jun 22..
[29]
Gossl M, Versari D, Mannheim D, et al. Increased spatial vasa vasorum density in the proximal LAD in hypercholesterolemia-implications for vulnerable plaque-development[J]. Atherosclerosis, 2007, 192:246.