全部 标题 作者
关键词 摘要

OALib Journal期刊
ISSN: 2333-9721
费用:99美元

查看量下载量

相关文章

更多...
PLOS Genetics  2015 

Signaling from Within: Endocytic Trafficking of the Robo Receptor Is Required for Midline Axon Repulsion

DOI: 10.1371/journal.pgen.1005441

Full-Text   Cite this paper   Add to My Lib

Abstract:

References

[1]  Chance RK, Bashaw GJ. Slit-dependent endocytic trafficking of the Robo receptor is required for Son of Sevenless recruitment and midline axon repulsion. PLoS Genet. 2015; 11(9): e1005371.
[2]  Piper M, Salih S, Weinl C, Holt C, Harris W (2005) Endocytosis-dependent desensitization and protein synthesis-dependent resensitization in retinal growth cone adaptation. Nat Neurosci 8: 179–186. pmid:15643427 doi: 10.1038/nn1380
[3]  Yang L, Bashaw GJ (2006) Son of sevenless directly links the Robo receptor to rac activation to control axon repulsion at the midline. Neuron 52: 595–607. pmid:17114045 doi: 10.1016/j.neuron.2006.09.039
[4]  Piper M, Anderson R, Dwivedy A, Weinl C, van Horck F, et al. (2006) Signaling mechanisms underlying Slit2-induced collapse of Xenopus retinal growth cones. Neuron 49: 215–228. pmid:16423696 doi: 10.1016/j.neuron.2005.12.008
[5]  Jen JC, Chan W-M, Bosley TM, Wan J, Carr JR, et al. (2004) Mutations in a human ROBO gene disrupt hindbrain axon pathway crossing and morphogenesis. Science 304: 1509–1513. pmid:15105459 doi: 10.1126/science.1096437
[6]  Anitha A, Nakamura K, Yamada K, Suda S, Thanseem I, et al. (2008) Genetic analyses of roundabout (ROBO) axon guidance receptors in autism. Am J Med Genet B Neuropsychiatr Genet 147B: 1019–1027. doi: 10.1002/ajmg.b.30697. pmid:18270976
[7]  Potkin SG, Turner JA, Guffanti G, Lakatos A, Fallon JH, et al. (2009) A genome-wide association study of schizophrenia using brain activation as a quantitative phenotype. Schizophr Bull 35: 96–108. doi: 10.1093/schbul/sbn155. pmid:19023125
[8]  Hannula-Jouppi K, Kaminen-Ahola N, Taipale M, Eklund R, Nopola-Hemmi J, et al. (2005) The axon guidance receptor gene ROBO1 is a candidate gene for developmental dyslexia. PLoS Genet 1: e50. pmid:16254601 doi: 10.1371/journal.pgen.0010050.eor

Full-Text

Contact Us

service@oalib.com

QQ:3279437679

WhatsApp +8615387084133