%0 Journal Article
%T 一氧化氮合酶与缺血缺氧性视网膜病变<br>Nitric Oxide Synthase and Ischemic Hypoxic Retinopathy
%A 郭玉洁
%A 李娜
%J Advances in Clinical Medicine
%P 9946-9952
%@ 2161-8720
%D 2022
%I Hans Publishing
%R 10.12677/ACM.2022.12111434
%X 缺血、缺氧是糖尿病视网膜病变(diabetic retinopathy, DR)、视网膜静脉阻塞(retinal vein occlusion, RVO)、高海拔视网膜病变(high altitude retinopathy, HAR)和年龄相关性黄斑病变(Age-related Macular Degeneration, AMD)及早产儿视网膜病变(retinopathy of prematurity, ROP)等常见视网膜病变的重要病理生理因素。近年来，关于缺血缺氧性视网膜病变机制的研究热点集中于一氧化氮合酶(Nitric oxide synthases, NOS)。本文主要从NOS在不同种类缺血、缺氧性视网膜病变中的作用机制角度进行综述。<br />
Ischemia and hypoxia are important pathophysiological factors of common retinopathy of diabetic retinopathy (DR), retinal vein occlusion (RVO) and high altitude retinopathy retinopathy (HAR), Age-related Macular Degeneration (AMD) and retinopathy of prematurity ROP), etc. In recent years, the research focus on the mechanism of ischemic hypoxic retinopathy is on Nitric oxide synthases (NOS). This article mainly reviews the mechanism of NOS in different kinds of ischemic and hypoxic retinopathy.
%K 一氧化氮合酶，低氧，视网膜<br>Nitric Oxide Synthase
%K Hypoxia
%K Retina
%U http://www.hanspub.org/journal/PaperInformation.aspx?PaperID=57583