%0 Journal Article
%T Gain-of-Function Mutations of SLC16A11 Contribute to the Pathogenesis of Type 2 Diabetes
%J -
%D 2019
%R https://doi.org/10.1016/j.celrep.2018.12.100
%X £¿ Slc16a11 knockout mice have minimal metabolic consequences £¿ Mice carrying SLC16A11 mutations have disrupted lipid metabolism and fatty liver £¿ Mutant SLC16A11 upregulates lipin 1 and increases liver triglyceride accumulatio
%U https://www.cell.com/cell-reports/fulltext/S2211-1247(18)32075-8