%0 Journal Article
%T Redefining environmental exposure for disease etiology
%A Stephen M. Rappaport
%J Archive of "NPJ Systems Biology and Applications".
%D 2018
%R 10.1038/s41540-018-0065-0
%X a Inputs to the blood exposome from endogenous sources (G, genome; GE, epigenome; R, transcriptome; P, proteome; M, metabolome), exogenous exposures (E), post-translational modifications (PTMs) and gene¨Cenvironment interactions (G£¿¡Á£¿E). b Pathways connecting the blood exposome to disease processes (causal pathways) and subsequent feedback to G, GE, R, and P (via reactive pathways
%U https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6119193/