%0 Journal Article
%T FGF1¦¤HBS ameliorates chronic kidney disease via PI3K/AKT mediated suppression of oxidative stress and inflammation
%A Chao Zheng
%A Dezhong Wang
%A Guanghui Zhu
%A Hui Jin
%A Jie Zhou
%A Lei Ying
%A Lingwei Jin
%A Mengyun Jin
%A Miaojuan Fan
%A Tianyang Zhao
%A Wei Jin
%A Wei Lin
%A Xiaokun Li
%A Xinyu Zhao
%A Yang Wang
%A Yushuo Zhao
%A Zhengle He
%A Zhifeng Huang
%J Archive of "Cell Death & Disease".
%D 2019
%R 10.1038/s41419-019-1696-9
%X C57BL/6J mice after 3 weeks of chronic administration of rFGF1 WT (2£¿mg/kg body weight), rFGF1¦¤HBS (2£¿mg/kg body weight), or control vehicle. a Representative images of PCNA or Ki-67 immunohistochemical staining of renal tissues (left panel) and quantitation using ImageJ software (right panel). Scale bar, 50£¿¦Ìm. b Expression of PCNA and Ki-67 in renal tissue as measured by western blot analyses (left panel) and quantitation using ImageJ software (right panel). Data are presented as the mean£¿¡À£¿SEM (n£¿=£¿6). *p£¿<£¿0.05, ***p£¿<£¿0.00
%K Cell biology
%K Molecular biology
%U https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6561918/