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Search Results: 1 - 10 of 3249 matches for " Yves Palau "
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La transformation des relations entre état et associations au centre d’une nouvelle dynamique institutionnelle : le cas de la politique de la ville en France
Yves Palau
Pyramides : Revue du Centre d'Etudes et de Recherches en Administration Publique , 2011,
Abstract: Les associations uvrant pour la politique de la ville constituent le principal mode d’intervention de l’état dans cette politique publique. Il serait pourtant erroné de ne considérer ces associations que comme de simples auxiliaires de la puissance publique. On assiste en fait à une dynamique de plus grande ampleur qui tout en inscrivant les associations dans une nouvelle configuration institutionnelle participe aussi de l’émergence d’un état médiateur . Celui-ci emprunte à l’idée associative son positionnement au sein de la société caractérisé par la valorisation de la proximité et de la cohésion sociale qui en fait une sorte de corps intermédiaire entre l’état et la société civile. Réciproquement, l’état médiateur intègre à lui, par des dispositifs partenariaux, certaines associations, celles notamment qui de petite taille, peuvent justifier de la plus grande proximité avec les populations des quartiers de la politique de la ville. L’état médiateur ne désigne donc en aucune manière une phase de retrait de l’état mais un redéploiement de celui-ci, marqué par un fort interventionnisme sur la société en même temps que l’adoption d’un discours et de pratiques qui furent longtemps l’expression exclusive des associations.
El Significado de la Negación Paraconsistente
Gladys Palau,Cecilia Duran
Principia : an International Journal of Epistemology , 2009,
Abstract: This work agrees and supports the I. Hacking’s thesis regarding the meaningof the logical constants accordingly with Gentzen’s Introduction and Elimination Rules of Sequent Calculus, corresponding with the abstract conception of the notion of logical consequence. We would like to ask for the minimum rules that must satisfy a connective in order to be considered as a genuine negation. Mainly, we will refer to both da Costa’s C-Systems and Priest’s LP system. Finally, we will analyze the presentations of these systems within the Sequent Logic to show that paraconsistent negation lacks of pure rules of negation-elimination and negation-introduction rules or that they involve other connectives, thus making difficult to assign an univocal meaning to paraconsistent negation.
Friedensreich (Fritz) Hundertwasser: Viena, 1928 - Queen Elizabeth II, 2000
JJ Noguera Palau
Archivos de la Sociedad Espa?ola de Oftalmología , 2002,
Joseph-Antoine Ferdinand Plateau: (Bruselas, 1801 - Gante, 1883)
JJ Noguera Palau
Archivos de la Sociedad Espa?ola de Oftalmología , 2003,
JJ Noguera Palau
Archivos de la Sociedad Espa?ola de Oftalmología , 2003,
El rol de las fuerzas armadas en la continuidad de los regímenes democráticos y en la vigencia del respeto a los Derechos Humanos.
Juan Carlos Palau.
Colombia Internacional , 1993,
H2 and CH4 Sorption on Cu-BTC Metal Organic Frameworks at Pressures up to 15 MPa and Temperatures between 273 and 318 K  [PDF]
Yves Gensterblum
Journal of Surface Engineered Materials and Advanced Technology (JSEMAT) , 2011, DOI: 10.4236/jsemat.2011.12004
Abstract: Sorption isotherms of methane and hydrogen on Cu3(BTC)2 have been measured in the temperature range from 273 to 318 K and at pressures up to 15 MPa. H2 excess sorption capacities of the Cu3(BTC)2 amounted to 3.9 mg/g at 14 MPa. Promising maximum CH4 excess sorption capacities on the same sample were reached at approximately 5 MPa. They amounted to 101, 100, 92 and 80 mg/g at 273, 278, 293 and 318 K, respectively. The sorbed phase density was essestially the same for all temperatures and amounted to ~600 kg/m3. Structural changes of the Cu3(BTC)2 samples after thermal activation and treatment with high pressure H2 and CH4 were tested. It was found that the initial micropore structure has virtually disappeared as evidenced by a decrease of the Langmuir specific surface area by a factor ~3 and CO2 micropore volume by a factor of ~4 for H2 and ~3 for CH4. This is in line with an increase in the average pore diameter from initially 9.2 to 15.7 for H2 and 12.8 for CH4.
Minkowskian Solution of General Relativity with Cosmological Constant and the Accelerating Universe  [PDF]
Yves Pierseaux
Journal of Modern Physics (JMP) , 2014, DOI: 10.4236/jmp.2014.516172
Abstract: A Minkowskian solution of the equation of General Relativity (as written by Einstein in 1915) is trivial because it simply means that both members of the equation are equal to zero. However, if alternatively, one considers the complete equation with a non-zero constant Λ (Einstein 1917), a Minkowskian solution is no longer trivial because it amounts to impose a constraint on the right hand side of the equation (i.e. a non-null stress-energy tensor). If furthermore one identifies (as usual) this tensor to the one of a perfect fluid, one finds that this fluid has a positive energy density and a negative pressure that depend on the three constants of the equation (i.e. gravitational constant G, cosmological constant Λ and velocity of light c). When doing that (§1), one has to consider the “Minkowskian Vacuum” as a physical object of GR (an enigmatic non-baryonic Minkowskian fluid). Can one build a model of this object on the basis of a dynamical equilibrium between the effective gravitational attraction due to the positive energy density versus the negative pressure repulsion? We propose to study such a model, where the (enigmatic) fluid is assumed to exist only in a limited sphere whose surface acts like a “test body” sensitive to the gravitational field created by the fluid. No static equilibrium exists, but a pseudoNewtonian “dynamical equilibrium” (§2) can be reached if the pseudoEuclidean fluid is in state of expansion. Up to there, we have simply constructed a model of an “abstract Universe” (i.e. the limited sphere: There is no fluid outside this sphere!) that gives to a (purely mathematical) constant Λ a concrete physical meaning. We discover finally that our expanding fluid has not only dynamical (gravitational) properties (§3) but also optical properties that are connected with Doppler Redshift (§4). Remembering that recent observations in Cosmology indicate that the “real Universe” seems to be “Flat” and in “Accelerated Expansion”; remembering also (after all) that the archetypal Flat Universe is simply a Minkowskian Universe, we logically wonder if the unexpected Minkowskian global solution, could not be also a significant cosmological model (conclusion).
Neural and Molecular Features on Charcot-Marie-Tooth Disease Plasticity and Therapy
Paula Juárez,Francesc Palau
Neural Plasticity , 2012, DOI: 10.1155/2012/171636
Abstract: In the peripheral nervous system disorders plasticity is related to changes on the axon and Schwann cell biology, and the synaptic formations and connections, which could be also a focus for therapeutic research. Charcot-Marie-Tooth disease (CMT) represents a large group of inherited peripheral neuropathies that involve mainly both motor and sensory nerves and induce muscular atrophy and weakness. Genetic analysis has identified several pathways and molecular mechanisms involving myelin structure and proper nerve myelination, transcriptional regulation, protein turnover, vesicle trafficking, axonal transport and mitochondrial dynamics. These pathogenic mechanisms affect the continuous signaling and dialogue between the Schwann cell and the axon, having as final result the loss of myelin and nerve maintenance; however, some late onset axonal CMT neuropathies are a consequence of Schwann cell specific changes not affecting myelin. Comprehension of molecular pathways involved in Schwann cell-axonal interactions is likely not only to increase the understanding of nerve biology but also to identify the molecular targets and cell pathways to design novel therapeutic approaches for inherited neuropathies but also for most common peripheral neuropathies. These approaches should improve the plasticity of the synaptic connections at the neuromuscular junction and regenerate cell viability based on improving myelin and axon interaction. 1. Introduction Charcot-Marie-Tooth disease (CMT) is a clinical and genetic heterogeneous group of inherited motor and sensory peripheral neuropathies (HMSN) that affect 17–40 per 100,000 inhabitants [1, 2]. Mendelian segregation in families may follow either autosomal dominant, autosomal recessive, or X-linked patterns. Autosomal recessive forms are described more frequently in specific populations and geographical areas such as the Mediterranean basin. Molecular genetic studies and positional cloning, and more recently exome sequencing approaches, have unraveled a wide number of genes involved in the etiology of CMT disease [3–5]. Molecular genetic studies have been very successful for defining the gene nosology and classification of inherited peripheral neuropathies; more than 40 genes have so far been identified to be associated with CMT and related neuropathies, including rare clinical variants (http://www.molgen.ua.ac.be/CMTmutations/). As an immediate consequence, genetic testing has become an important tool in clinical practice of CMT, and patients and families have been beneficiated of a more specific genetic counseling.
Autosomal recessive cerebellar ataxias
Francesc Palau, Carmen Espinós
Orphanet Journal of Rare Diseases , 2006, DOI: 10.1186/1750-1172-1-47
Abstract: Autosomal recessive cerebellar ataxias (ARCA)Early onset cerebellar ataxias (EOCA)Autosomal recessive spinocerebellar ataxiasAutosomal recessive cerebellar ataxias (ARCA) belong to the wider group disorders known as inherited ataxias [1-3]. ARCA are neurological disorders characterized by degeneration or abnormal development of cerebellum and spinal cord, autosomal recessive inheritance and, in most cases, early onset occurring before the age of 20. This group encompasses a large number of rare diseases, the most frequent being Friedreich ataxia.Different criteria have been used to classify ARCA and issues of classification still remain controversial. In the 80s of the last century, Harding proposed a clinical classification for inherited ataxias based on two criteria: the age of onset and pathological mechanisms [4,5]. Most of early onset ataxias (before age of 20 years) show autosomal recessive inheritance and may be classified as ARCA. Some of them are not progressive disorders and are associated with impaired development of the cerebellum and its connections; they were considered congenital ataxias. Other have a metabolic cause and show either progressive or intermittent natural history. The most frequent ataxias in Harding classification were degenerative ataxias with unknown cause. However, in the last ten years molecular genetic studies have changed the global panorama of inherited ataxias. Koenig has used topographical and pathophysiological criteria for ARCA; he distinguished sensory and spinocerebellar ataxias, cerebellar ataxias with sensory-motor peripheral neuropathy and pure cerebellar ataxia [6]. Recently, the group of Filla suggested a pathogenic approach to classify hereditary ataxias [7]. These authors did not consider any genetic, pathological or natural history-based criteria, and divided the disorders in mitochondrial ataxias (including Friedreich ataxia), metabolic ataxias, ataxias associated with defective DNA repair, ataxias with abnormal pro
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