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Search Results: 1 - 10 of 25781 matches for " Yanbin Dong "
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Existence and Uniqueness of Positive Solutions for Discrete Fourth-Order Lidstone Problem with a Parameter
Sang Yanbin,Wei Zhongli,Dong Wei
Advances in Difference Equations , 2010,
Abstract: This work presents sufficient conditions for the existence and uniqueness of positive solutions for a discrete fourth-order beam equation under Lidstone boundary conditions with a parameter; the iterative sequences yielding approximate solutions are also given. The main tool used is monotone iterative technique.
Existence and Uniqueness of Positive Solutions for Discrete Fourth-Order Lidstone Problem with a Parameter
Yanbin Sang,Zhongli Wei,Wei Dong
Advances in Difference Equations , 2010, DOI: 10.1155/2010/971540
Abstract: This work presents sufficient conditions for the existence and uniqueness of positive solutions for a discrete fourth-order beam equation under Lidstone boundary conditions with a parameter; the iterative sequences yielding approximate solutions are also given. The main tool used is monotone iterative technique.
Can novel Apo A-I polymorphisms be responsible for low HDL in South Asian immigrants?
Dodani Sunita,Dong Yanbin,Zhu Haidong,George Varghese
Indian Journal of Human Genetics , 2008,
Abstract: Coronary artery disease (CAD) is the leading cause of death in the world. Even though its rates have decreased worldwide over the past 30 years, event rates are still high in South Asians. South Asians are known to have low high-density lipoprotein (HDL) levels. The objective of this study was to identify Apolipoprotein A-I (Apo A-I) polymorphisms, the main protein component of HDL and explore its association with low HDL levels in South Asians. A pilot study on 30 South Asians was conducted and 12-h fasting samples for C-reactive protein, total cholesterol, HDL, low-density lipoprotein (LDL), triglycerides, Lipoprotein (a), Insulin, glucose levels, DNA extraction, and sequencing of Apo A-I gene were done. DNA sequencing revealed six novel Apo A-I single nucleotide polymorphisms (SNPs) in South Asians, one of which (rs 35293760, C938T) was significantly associated with low (< 40 mg/dl) HDL levels (P = 0.004). The association was also seen with total cholesterol (P = 0.026) and LDL levels (P = 0.032). This pilot work has highlighted some of the gene-environment associations that could be responsible for low HDL and may be excess CAD in South Asians. Further larger studies are required to explore and uncover these associations that could be responsible for excess CAD risk in South Asians.
Relationship between Rad51 G135C and G172T Variants and the Susceptibility to Cancer: A Meta-Analysis Involving 54 Case-Control Studies
Mengmeng Zhao, Pin Chen, Yanbin Dong, Xianji Zhu, Xilong Zhang
PLOS ONE , 2014, DOI: 10.1371/journal.pone.0087259
Abstract: Background The associations between Rad51 gene polymorphisms (G135C and G172T) and risk of cancer have been investigated, but the results were inconclusive. To get a comprehensive evaluation of the association above, we performed a meta-analysis of published studies. Methods A computerized search of PubMed, Embase and Web of Knowledge databases for all relevant studies was performed and the data were analyzed in a meta-analysis. The overall odds ratio (OR) with the 95% confidence interval (95% CI) was calculated to assess the strength of the association between Rad51 polymorphisms and cancer risk. Data were analyzed using fixed- or random-effects model when appropriate. Sensitivity analysis and publication bias test were also estimated. Results Overall, a total of 54 case-control studies were included in the current meta-analysis, among which 42 studies with 19,142 cases and 20,363 controls for RAD51 G135C polymorphism and 12 studies with 6,646 cases and 6,783 controls for G172T polymorphism. For G135C polymorphism, the pooled results indicated that significantly increased risk was found in overall cancers (homozygote model: OR = 1.776, 95% CI = 1.288–2.449; allelic genetic model: OR = 1.169, 95% CI = 1.016–1.345; recessive model: OR = 1.946, 95% CI = 1.336–2.835), especially in breast cancer (homozygote model: OR = 1.498, 95% CI = 1.026–2.189; recessive model: OR = 1.732, 95% CI = 1.170–2.562). For G172T polymorphism, a decreased cancer risk was observed in head and neck cancer (homozygote model: OR = 0.621, 95% CI = 0.460–0.837; allelic genetic model: OR = 0.824, 95% CI = 0.716–0.948; recessive model: OR = 0.639, 95% CI = 0.488–0.837). Conclusions Our results suggested that the Rad51 G135C polymorphism is a candidate for susceptibility to overall cancers, especially to breast cancer, and that the Rad51 G172T might play a protective role in the development of head and neck cancer.
A MAC Scheme with QoS Guarantee for MANETs  [PDF]
Yanbin YANG, Yulin WEI
Int'l J. of Communications, Network and System Sciences (IJCNS) , 2009, DOI: 10.4236/ijcns.2009.28088
Abstract: IEEE 802.11 distributed coordination function (DCF) can alleviate the collision and hidden station problem, but it doesn’t differentiate traffic categories (TC). Therefore, it can’t provide sufficient QoS support for different traffic categories. Recently, a new contention-based enhanced distributed channel access (EDCA) scheme was proposed which provides a probabilistic QoS support. In this paper, an adaptive EDCA scheme with QoS guarantee for mobile ad hoc networks (MANETs) is proposed. In this scheme, the EDCA scheme and the token bucket algorithm (TBA) are combined to adjust the contention window (CW). Our scheme provides the traffic differentiation.
Deviations of Steady States of the Traveling Wave to a Competition Diffusion System with Random Perturbation  [PDF]
Xiaorong Hu, Yanbin Tang
Journal of Applied Mathematics and Physics (JAMP) , 2015, DOI: 10.4236/jamp.2015.35062
Abstract: This paper considers the asymptotic dynamics of steady states to the Lotka-Volterra competition diffusion systems with random perturbations by two-parameter white noise on the whole real line. By the fundamental solution of heat equation, we get the asymptotic fluctuating behaviors near the stable states respectively. That is, near the steady state (u,v)=(0,1), the mean value Eu(x,t) is shifted above the equilibrium u=0 and Ev(x,t) is shifted below the equilibrium v=1. However, near the steady state (u,v)=(1,0), the mean value Eu(x,t) is shifted below the equilibrium u =1 and Eu(x,t)=0.
Differential Impact of Stress Reduction Programs upon Ambulatory Blood Pressure among African American Adolescents: Influences of Endothelin-1 Gene and Chronic Stress Exposure
Mathew J. Gregoski,Vernon A. Barnes,Martha S. Tingen,Yanbin Dong,Haidong Zhu,Frank A. Treiber
International Journal of Hypertension , 2012, DOI: 10.1155/2012/510291
Abstract: Stress-activated gene × environment interactions may contribute to individual variability in blood pressure reductions from behavioral interventions. We investigated effects of endothelin-1 (ET-1) LYS198ASN SNP and discriminatory stress exposure upon impact of 12-week behavioral interventions upon ambulatory BP (ABP) among 162 prehypertensive African American adolescents. Following genotyping, completion of questionnaire battery, and 24-hour ABP monitoring, participants were randomized to health education control (HEC), life skills training (LST), or breathing awareness meditation (BAM). Postintervention ABP was obtained. Significant three-way interactions on ABP changes indicated that among ET-1 SNP carriers, the only group to show reductions was BAM from low chronic stress environments. Among ET-1 SNP noncarriers, under low chronic stress exposure, all approaches worked, especially BAM. Among high stress exposure noncarriers, only BAM resulted in reductions. If these preliminary findings are replicated via ancillary analyses of archival databases and then via efficacy trials, selection of behavioral prescriptions for prehypertensives will be edging closer to being guided by individual's underlying genetic and environmental factors incorporating the healthcare model of personalized preventive medicine.
Cardiometabolic Biomarkers in Young Black Girls: Relations to Body Fatness and Aerobic Fitness, and Effects of a Randomized Physical Activity Trial
Bernard Gutin,Ryan A. Harris,Cheryl A. Howe,Maribeth H. Johnson,Haidong Zhu,Yanbin Dong
International Journal of Pediatrics , 2011, DOI: 10.1155/2011/219268
Abstract: There is little evidence from randomized trials showing that physical activity alone influences biomarker profiles in youths. This study tested two hypotheses: (i) that elevated body fatness and poor fitness would be associated with unfavorable levels of cardiometabolic biomarkers in 8–12-y-old black girls (=242) and (ii) that a 10-mo PA intervention would have favorable effects on the fatness-related cardiometabolic biomarkers. At baseline, all fatness indices (i.e., percent body fat, visceral adipose tissue, BMI, and waist circumference) were significantly (<0.05) associated with unfavorable levels of insulin, glucose, systolic BP, diastolic BP, triglycerides, C-reactive protein (CRP), and fibrinogen. Aerobic fitness was significantly (<0.05) associated with favorable levels of insulin, CRP, fibrinogen, and HDL2. The PA intervention had significant and favorable effects on fitness, fatness, and two biomarkers—resting heart rate and LDL cholesterol. More research is needed to clarify what types of interventions can enhance the cardiometabolic health of youths.
BAP31 is frequently overexpressed in patients with primary colorectal cancer and correlates with better prognosis
LingYi Dong,KeWei Jiang,YanBin Zhang,Hui Zhang,HongQing Zhuo,ZhiRong Cui,YingJiang Ye,Shan Wang
Chinese Science Bulletin , 2011, DOI: 10.1007/s11434-011-4610-0
Abstract: We previously showed that B cell receptor associated protein 31(BAP31) was significantly upregulated in colorectal cancer compared with normal mucosa epithelia. However, its expression pattern and pathological role in colorectal cancer are not clearly understood. In this study, we investigated whether the expression of BAP31 was associated with the clinicopathological parameters of colorectal cancer. The expression pattern of BAP31 was detected by immunohistochemistry on a tissue microarray in both primary tumor and paired distant normal mucosa samples from 120 consecutive colorectal cancer patients. Furthermore, BAP31 protein expression was also determined in twenty colorectal adenomas and eight liver metastasis samples. There was positive expression of BAP31 in 64.17% of primary tumors and 6.67% in distant normal mucosa (P=0.000). Negative expression of BAP31 was correlated with distant metastasis (P=0.036) and lower tumor differentiation grade (P=0.001). Patients with BAP31-negative expression showed significantly lower overall survival rate (P=0.003) compared to patients with BAP31-positive expression. Our results demonstrate that BAP31 may serve as a candidate prognostic marker in colorectal cancer and negative BAP31 expression may lead to more aggressive invasion of colorectal cancer.
Differential Impact of Stress Reduction Programs upon Ambulatory Blood Pressure among African American Adolescents: Influences of Endothelin-1 Gene and Chronic Stress Exposure
Mathew J. Gregoski,Vernon A. Barnes,Martha S. Tingen,Yanbin Dong,Haidong Zhu,Frank A. Treiber
International Journal of Hypertension , 2012, DOI: 10.1155/2012/510291
Abstract: Stress-activated gene × environment interactions may contribute to individual variability in blood pressure reductions from behavioral interventions. We investigated effects of endothelin-1 (ET-1) LYS198ASN SNP and discriminatory stress exposure upon impact of 12-week behavioral interventions upon ambulatory BP (ABP) among 162 prehypertensive African American adolescents. Following genotyping, completion of questionnaire battery, and 24-hour ABP monitoring, participants were randomized to health education control (HEC), life skills training (LST), or breathing awareness meditation (BAM). Postintervention ABP was obtained. Significant three-way interactions on ABP changes indicated that among ET-1 SNP carriers, the only group to show reductions was BAM from low chronic stress environments. Among ET-1 SNP noncarriers, under low chronic stress exposure, all approaches worked, especially BAM. Among high stress exposure noncarriers, only BAM resulted in reductions. If these preliminary findings are replicated via ancillary analyses of archival databases and then via efficacy trials, selection of behavioral prescriptions for prehypertensives will be edging closer to being guided by individual's underlying genetic and environmental factors incorporating the healthcare model of personalized preventive medicine. 1. Introduction Essential hypertension (EH) is a major risk factor for cardiovascular disease (CVD), and EH incidence among youth is increasing [1]. African Americans (AAs) experience a higher prevalence, earlier onset, and greater severity of EH-related complications than other ethnic groups [2]. From late childhood onward, AAs display increased levels of resting and ambulatory blood pressure (ABP) compared to other ethnic groups [3–5]. BP levels are monotonically associated with future CVD morbidity and mortality [6]. Stage I prehypertensive adults (i.e., SBP/DBP 121–129/81–84?mmHg) have a 40% increased risk and adults with stage II prehypertension (i.e., SBP/DBP 130–139/85–89?mmHg) are twice as likely to develop CVD compared to those with optimal BP (<120/<80?mmHg) [6–8]. BP percentile ranking tracks from late childhood into adulthood [9–11] placing AA adolescents with BP between the 50th and 95th percentiles for age and sex at an increased risk of future EH and CVD development [9]. EH, like other multifactorial chronic diseases, results from a complex interplay between an individual’s genetic underpinnings, lifestyle behaviors, psychosocial factors, and exposures to various environmental toxins. Over time, this dynamic interplay eventuates in adverse
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