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Search Results: 1 - 10 of 3031 matches for " Raimundo Carmona Puerta "
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Trece razones para no abandonar el uso de la quinidina y evitar su retiro del mercado
Raimundo Carmona Puerta
Revista Argentina de Cardiología , 2010,
Abstract:
SíNDROME DE BRUGADA: DESDE LOS GENES HASTA LA TERAPéUTICA / Brugada syndrome: from genes to therapeutics
Raimundo Carmona Puerta
CorSalud , 2010,
Abstract: The increasing number of ion channelopathies discovered in the heart, with fatal consequences, implies that the specialists involved in the management of these patients must strive to reach a better understanding of basic cardiac electrophysiology. In the Brugada syndrome, up to six genotypes have already been described with affectations in the sodium, calcium and potassium (Ito type) channels. In all cases there is a typical electrocardiogram which shows right precordial leads due to the transmural dispersion of repolarization, more pronounced in the region of the outflow tract of the right ventricle. The disease may be asymptomatic or have sudden death as its first manifestation. The implantable defibrillator is considered the most effective treatment, but it can be combined with quinidine to space the shocks and abort electrical storms.
BASES ELECTROFISIOLóGICAS DE LA ESTIMULACIóN CARDíACA / Electrophysiological basis of cardiac pacing
Ginner Odorico Rizo Rivera,Raimundo Carmona Puerta
CorSalud , 2009,
Abstract: The search for a physiological cardiac pacing has made the functioning of the pacemakers increasingly complex. This complexity ranges from the detection of the cardiac signal, its fitting-outand the response of the pacemaker by means of algorithms, to the electrical pacing of the heart. It is a must for us, as professionals devoted to cardiology, to know the basic principles of this process.The electrophysiological basis of electrical cardiac pacing is treated in this article, trying to achieve a general understanding of it, and in consequence facilitating a better comprehension of a topic that we hear or deal with, over and over, in our medical practice.
PREDICCIóN DEL RIESGO ARRíTMICO MEDIANTE ELECTROCARDIOGRAFíA. VALOR DEL íNDICE TPICO-TFINAL Y OTRA PROPUESTA POR CONFIRMAR / Arrhythmic risk prediction by means of electrocardiography. The value of T peak-T end index, and another proposal yet to be confirmed
Raimundo Carmona Puerta,Ginner O. Rizo Rivera
CorSalud , 2010,
Abstract:
DISPERSIóN DE LA ONDA P, USO DEL MéTODO MANUAL DE MEDICIóN COMO RECOMENDACIóN PARA LA PRáCTICA MéDICA. EXPERIENCIA DEL CARDIOCENTRO ERNESTO CHE GUEVARA / P- wave dispersion, use of the manual method of measurement as a recommendation for medical practice. Experience at the Ernesto Che Guevara Cardiology Hospital
Elibet Chávez González,Raimundo Carmona Puerta
CorSalud , 2011,
Abstract: Since the discovery of atrial fibrillation, this disease hasbecome a huge potential market, which attracts considerable resources for its research. In the multivariatemodels, the established cardiovascular disease, hypertension,age and left ventricular hypertrophy had the strongest association with atrial fibrillation. In the pathophysiologic basis of the P-wave dispersion, an emphasisis made on the alterations of atrial actionpotential, delayed impulse propagation through theatria and electrical remodeling of the walls of these cardiac chambers. The methodology for the measurementof the P-wave has been extensively studied anddiscussed at international symposia. It is known thatthe most reliable method is to use a digital signal withhigh resolution screen. In daily medical practice, theuse of this technology is being limited, and the use ofthe ECG is a routine practice that we should not forgetfor the measurement of the P-wave and the calculationof its dispersion. The digital method can be well used inthe design of studies, but the everyday use of the electrocardiogram should strengthen the practice ofeach electrocardiographist to identify risk patients with higher values of dispersion of the wave.
CONTRADICCIóN DE ABLACIONAR O NO A PACIENTES ASINTOMáTICOS CON SíNDROME DE WOLFF PARKINSON WHITE / Contradiction of whether or not ablating asymptomatic patients with Wolff Parkinson White syndrome
Elibet Chávez González,Raimundo Carmona Puerta
CorSalud , 2012,
Abstract:
RAZONES Y MéTODO PARA PREVENIR LA FIBRILACIóN AURICULAR CUANDO PUEDE LOGRARSE / Reasons and method to prevent, when possible, atrial fibrillation
Elibet Chávez González,Raimundo Carmona Puerta
CorSalud , 2012,
Abstract:
RIESGO DE ARRITMIAS E HIPERTENSIóN ARTERIAL / Risk of Arrhythmias and hypertension
Elibet Chávez González,Raimundo Carmona Puerta
CorSalud , 2012,
Abstract: Resumen La mayor prevalencia de fibrilación auricular se duplica en pacientes con cardiopatía hipertensiva. En el estudio Framingham se encontraron como factores de riesgo: la hipertensión, la máxima duración y dispersión de la onda P del electrocardiograma, el tama o de la aurícula y la masa ventricular izquierda. El sistema renina-angiotensina-aldosterona es responsable de la inflamación y los cambios estructurales que justifican la aparición de arritmias en el paciente hipertenso. La presencia de arritmias ventriculares, desde complejos ventriculares prematuros hasta la taquicardia ventricular, se ha demostrado entre un 10 - 27 % en los pacientes afectados, porcentaje que disminuye mientras más grave es la arritmia. Se han resumido los siguientes marcadores incruentos de riesgo para presentar arritmias ventriculares: dispersión y variabilidad del intervalo QT, presencia de potenciales tardíos, variabilidad de la frecuencia cardíaca, y morfología anormal y alternancia de la onda T. Todos ellos relacionados con cambios anátomo-estructurales de la pared ventricular, que afectan el potencial de acción. La importancia de conocer la epidemiología, fisiopatología, y los marcadores incruentos de riesgo de presentar arritmias en el paciente hipertenso, ofrece un camino para la aplicación de una correcta terapéutica en el control de las cifras de presión arterial y regresión de la hipertrofia, como lo han demostrado los fármacos inhibidores del sistema renina-angiotensina-aldosterona, que disminuyen el riesgo de arritmias. / Abstract The higher prevalence of atrial fibrillation is doubled in patients with hypertensive heart disease. In the Framingham study, the following risk factors were found: hypertension, maximum P wave duration and dispersion of the electrocardiogram, atrial size and left ventricular mass. The renin-angiotensin-aldosterone system is responsible for the inflammation and the structural changes that justify the development of arrhythmias in hypertensive patients. The presence of ventricular arrhythmias, from premature ventricular complexes to ventricular tachycardia, has been shown in a 10 to 27% of affected patients, a percentage that decreases with the severity of the arrhythmia. Noninvasive risk markers for ventricular arrhythmias have been summarized as follows: dispersion and variability of the QT interval, presence of late potentials, heart rate variability, abnormal morphology and T-wave alternans. All of these are related to anatomic-structural changes of the ventricular wall, which affect the action potential. The importance of know
FIBRILACIóN AURICULAR POR VíA ACCESORIA EN UN ADULTO. CONTRADICCIóN DE ABLACIONAR O NO A PACIENTES ASINTOMáTICOS / Atrial fibrillation through accessory pathway in an adult. Contradiction in whether to ablate or not asymptomatic patients
Elibet Chávez González,Raimundo Carmona Puerta
CorSalud , 2011,
Abstract:
DISPERSIóN DE LA ONDA P, UN TRASTORNO DE CONDUCCIóN INTRA E INTERAURICULAR / P-wave dispersion, an intra and interauricular conduction disorder
Elibet Chávez González,Raimundo Carmona Puerta,Ramiro R. Ramos Ramírez
CorSalud , 2011,
Abstract:
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