In this paper, a multi label variant of CLUBAS  algorithm, ML-CLUBAS (Multi Label-Classification of software Bugs Using Bug Attribute Similarity) is presented. CLUBAS is a hybrid algorithm, and is designed by using text clustering, frequent term calculations and taxonomic terms mapping techniques, and is an example of classification using clustering technique. CLUBAS is a single label algorithm, where one bug cluster is exactly mapped to a single bug category. However a bug cluster can be mapped into the more than one bug category in case of cluster label matches with the more than one category term, for this purpose ML-CLUBAS a multi label variant of CLUBAS is presented in this work. The designed algorithm is evaluated using the performance parameters F-measures and accuracy, number of clusters and purity. These parameters are compared with the CLUBAS and other multi label text clustering algorithms.
Endotoxic shock was induced in five apparently healthy male buffalo calves by i.v infusion of Escherichia coli endotoxin at 5microgram/kilogram (μg/Kg) body weight/hour (BW/hr) for 3 hours. Endotoxin infusion caused clinical signs of restlessness,
respiratory distress, snoring, diarrhoea, profuse
salivation along with the significant
hypoproteinemia, hypoalbuminemia and hypokalemia in all the animals. The
animals were observed up to day 4 or death, whichever was earlier. The treatment
with one time intravenous infusion of hypertonic
saline solution @ 4milliliter/Kilogram body weight (ml/Kg?BW), dextran-40
@ 10 ml/Kg?BW, flunixin meglumine @ 1.1 milligram/Kg?BW
(mg/Kg?BW) and blood @ 20 ml/Kg?BW to these animals alleviated the clinical
signs and significantly raised the circulating glucose level at 4.5 and 5.5 hrs.
The treatment led to survival of three of the
five endotoxemic buffalo calves. The significant hypoproteinemia, hypoalbuminemia,
hypokalemia and hypoglobulinemia continued even after treatment. Gross and histopathologic
findings of congestion, haemorrhage, necrosis in vital organs viz., lungs, liver,
kidneys, brain and intestines were suggestive of endotoxin induced hypoxia and multi-organ
failure. Additionally, emphysema and fibrinous thrombi in microvasculature of lungs
were salient histopathological findings indicating terminal respiratory failure
in the remaining two dead endotoxemic buffalo calves. From clinical signs, plasma
chemistry and pathological lesions, it was concluded that endotoxemia led to a disruption
of critical life processes, but a timely and effective treatment could counter these
deleterious effects and save precious lives.