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Search Results: 1 - 10 of 402558 matches for " M. Gross "
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A Finiteness Theorem for Elliptic Calabi-Yau Threefolds
M. Gross
Mathematics , 1993,
Abstract: We prove that up to birational equivalence, there exists only a finite number of families of Calabi-Yau threefolds (i.e. a threefold with trivial canonical class and factorial terminal singularities) which have an elliptic fibration to a rational surface. This strengthens a result of B. Hunt that there are only a finite number of possible Euler characteristics for such threefolds.
Mulheres budistas como líderes e professoras
Gross, Rita M.;
Revista Estudos Feministas , 2005, DOI: 10.1590/S0104-026X2005000200015
Abstract: in buddhism, the role of the dharma (religious) teacher is the most prestigious role, and dharma teachers have more authority that any other leaders. though the buddhist teachings contain no doctrines that limit this role to men, in practice throughout buddhist history, very few women have been publicly acknowledged as dharma teachers. some people claim that this practice does not harm women because women can, nevertheless, receive teachings, do advanced practices, and attain high states of spiritual realization. however, i claim that the practice of not recognizing women as dharma teachers has been very harmful both to buddhist women and to buddhism itself. it has lead to the demise of the nuns' community in many parts of the buddhist world, to lower standards of education for women, to lower prestige for women practitioners, to the lack of role models for women, and to the loss of women's wisdom from the heritage of buddhist thought. until women dharma teachers are widely recognized and honored, buddhism will continue to be haunted by its patriarchal past, to the loss of everyone concerned.
Simulation of static critical phenomena in non-ideal fluids with the Lattice Boltzmann method
M. Gross,F. Varnik
Physics , 2012, DOI: 10.1103/PhysRevE.85.056707
Abstract: A fluctuating non-ideal fluid at its critical point is simulated with the Lattice Boltzmann method. It is demonstrated that the method, employing a Ginzburg-Landau free energy functional, correctly reproduces the static critical behavior associated with the Ising universality class. A finite-size scaling analysis is applied to determine the critical exponents related to the order parameter, compressibility and specific heat. A particular focus is put on finite-size effects and issues related to the global conservation of the order-parameter.
Quivers, curves, and the tropical vertex
M. Gross,R. Pandharipande
Mathematics , 2009,
Abstract: Elements of the tropical vertex group are formal families of symplectomorphisms of the 2-dimensional algebraic torus. Commutators in the group are related to Euler characteristics of the moduli spaces of quiver representations and the Gromov-Witten theory of toric surfaces. After a short survey of the subject (based on lectures of Pandharipande at the 2009 Geometry summer school in Lisbon), we prove new results about the rays and symmetries of scattering diagrams of commutators (including previous conjectures by Gross-Siebert and Kontsevich). Where possible, we present both the quiver and Gromov-Witten perspectives.
Elliptic Three-folds I: Ogg-Shafarevich Theory
I. Dolgachev,M. Gross
Mathematics , 1992,
Abstract: We calculate the Tate-Shafarevich group of an elliptic three-fold $f:X\rightarrow S$ when $X$ and $S$ are regular and $f$ is flat, relating it to the Brauer group of $X$ and $S$. We show that given certain hypotheses on $f$, the Tate-Shafarevich group has the interpretation of isomorphism classes of elliptic curves over the function field of $S$ which have the same jacobian as the generic fibre of $f$, and for which there exists a relatively minimal model which has no multiple fibres. We use this to give examples of elliptic fibrations with isolated multiple fibres, and also to give a new counterexample to the Luroth problem in dimension three. This is a revised, hopefully improved, version with a few extra theorems and a few errors corrected.
Expanded progenitor populations, vitreo-retinal abnormalities, and Müller glial reactivity in the zebrafish leprechaun/patched2 retina
Jonathan Bibliowicz, Jeffrey M Gross
BMC Developmental Biology , 2009, DOI: 10.1186/1471-213x-9-52
Abstract: To gain further insight into Hh pathway function in the retina, we have analyzed retinal development in leprechaun/patched2 mutant zebrafish. While lep/ptc2 mutants possessed more cells in their retinas, all cell types, except for Müller glia, were present at identical ratios as those observed in wild-type siblings. lep/ptc2 mutants possessed a localized upregulation of GFAP, a marker for 'reactive' glia, as well as morphological abnormalities at the vitreo-retinal interface, where Müller glial endfeet terminate. In addition, analysis of the over-proliferation phenotype at the ciliary marginal zone (CMZ) revealed that the number of proliferating progenitors, but not the rate of proliferation, was increased in lep/ptc2 mutants.Our results indicate that Patched2-dependent Hh signaling does not likely play an integral role in neuronal cell fate decisions in the zebrafish retina. ptc2 deficiency in zebrafish results in defects at the vitreo-retinal interface and Müller glial reactivity. These phenotypes are similar to the ocular abnormalities observed in human patients suffering from Basal Cell Naevus Syndrome (BCNS), a disorder that has been linked to mutations in the human PTCH gene (the orthologue of the zebrafish ptc2), and point to the utility of the lep/ptc2 mutant line as a model for the study of BCNS-related ocular pathologies. Our findings regarding CMZ progenitor proliferation suggest that, in the zebrafish retina, Hh pathway activity may not affect cell cycle kinetics; rather, it likely regulates the size of the retinal progenitor pool in the CMZ.During retinal development, proliferation and differentiation must be tightly coordinated in order to produce a tissue of the proper size and containing the correct cell types [1]. The Hh pathway has been shown to play a critical role in controlling these two seemingly opposite processes [2]. Early in retinal development, the optic vesicle is composed of a population of proliferating neuroepithelial cells that will u
How does the estrogen receptor work?
Jennifer M Gross, Douglas Yee
Breast Cancer Research , 2002, DOI: 10.1186/bcr424
Abstract: It has long been known that ovarian production of the sex steroid hormone, estrogen, affects growth, differentiation, and function of the mammary gland. Estrogen mediates its effects by binding to its receptors, estrogen receptor (ER)-α and ER-β. Because ER-α was the first receptor subtype to be identified in the breast, most studies have focused on the biological role of ER-α in the mammary gland. It has recently been shown that ER-β is also expressed in breast cancer, but its function remains elusive [1]. Classically, ER-α is thought to function as a ligand-activated transcription factor. By interacting with estrogen-response elements contained in the promoter region of specific genes, modulation of gene expression ultimately results in the biological effects of estrogen. Extracellular signals can also stimulate ER-α-mediated transcription in the absence of estrogen. In recent years, emerging evidence has revealed that a role for ER-α is to affect gene expression in the absence of direct DNA binding. For example, liganded ER-α can influence gene expression by associating with other transcription factors without binding directly to DNA. Despite the clear understanding of the genomic mechanism of estrogen action, it is also postulated that estradiol can exert nongenomic effects on cell biology by interacting with other proteins, including a putative membrane estrogen receptor, growth factor receptors, and intermediate cell signaling molecules [2-4].Clinically, breast cancers have been classified as either ER-α-positive or ER-α-negative. While patients with ER-α-positive tumors have a slightly better survival rate than patients with ER-α-negative tumors, expression of ER-α is more useful as a predictive factor for response to endocrine therapy. Over half of patients with tumors positive for both ER-α and the progesterone receptor respond to therapy, such as tamoxifen, that is designed to interrupt the function of ER-α [5].Despite the clear clinical benefit for the in
Discovery of the Calcium Isotopes
J. L. Gross,M. Thoennessen
Physics , 2010,
Abstract: Twenty four calcium isotopes have so far been observed; the discovery of these isotopes is discussed. For each isotope a brief summary of the first refereed publication, including the production and identification method, is presented.
Discovery of Gallium, Germanium, Lutetium, and Hafnium Isotopes
J. L. Gross,M. Thoennessen
Physics , 2011, DOI: 10.1016/j.adt.2011.09.004
Abstract: Currently, twenty-eight gallium, thirty-one germanium, thirty-five lutetium, and thirty-six hafnium isotopes have been observed and the discovery of these isotopes is discussed here. For each isotope a brief synopsis of the first refereed publication, including the production and identification method, is presented.
Collaborative Regression
Samuel M. Gross,Robert Tibshirani
Quantitative Biology , 2014,
Abstract: We consider the scenario where one observes an outcome variable and sets of features from multiple assays, all measured on the same set of samples. One approach that has been proposed for dealing with this type of data is ``sparse multiple canonical correlation analysis'' (sparse mCCA). All of the current sparse mCCA techniques are biconvex and thus have no guarantees about reaching a global optimum. We propose a method for performing sparse supervised canonical correlation analysis (sparse sCCA), a specific case of sparse mCCA when one of the datasets is a vector. Our proposal for sparse sCCA is convex and thus does not face the same difficulties as the other methods. We derive efficient algorithms for this problem, and illustrate their use on simulated and real data.
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