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Search Results: 1 - 10 of 24090 matches for " KeWei Jiang "
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The Research on the Insurance Industry’s Tax Burden in China under the Background of Change from Business Tax to VAT  [PDF]
Kewei Chen
Modern Economy (ME) , 2018, DOI: 10.4236/me.2018.94048
Abstract: The insurance industry plays a key role in the national economy and modern society. The tax policy has a wide range of influences on the development of the insurance industry and the tax burden is the core of tax policy, which is directly related to the business performance and the balance of the insurance market. This paper estimates insurance companies’ effective tax rate of business tax and income tax by selecting insurance companies’ financial statement data from 2008 to 2015. We can see that the effective tax rate of insurance industry is facing a heavy tax burden especially for property insurance industry. By studying these problems, we should pay more attention to the tax burden on the insurance companies in the implementation of value-added tax in order to achieve the basic goal about change from business tax to VAT that does not increase but decreases slightly the tax burden and eliminates the double taxation. We also should solve the problems and challenges of insurance companies, facing change from business Tax to VAT, thus promoting the development of the insurance industry and enhancing its international competitiveness.
BAP31 is frequently overexpressed in patients with primary colorectal cancer and correlates with better prognosis
LingYi Dong,KeWei Jiang,YanBin Zhang,Hui Zhang,HongQing Zhuo,ZhiRong Cui,YingJiang Ye,Shan Wang
Chinese Science Bulletin , 2011, DOI: 10.1007/s11434-011-4610-0
Abstract: We previously showed that B cell receptor associated protein 31(BAP31) was significantly upregulated in colorectal cancer compared with normal mucosa epithelia. However, its expression pattern and pathological role in colorectal cancer are not clearly understood. In this study, we investigated whether the expression of BAP31 was associated with the clinicopathological parameters of colorectal cancer. The expression pattern of BAP31 was detected by immunohistochemistry on a tissue microarray in both primary tumor and paired distant normal mucosa samples from 120 consecutive colorectal cancer patients. Furthermore, BAP31 protein expression was also determined in twenty colorectal adenomas and eight liver metastasis samples. There was positive expression of BAP31 in 64.17% of primary tumors and 6.67% in distant normal mucosa (P=0.000). Negative expression of BAP31 was correlated with distant metastasis (P=0.036) and lower tumor differentiation grade (P=0.001). Patients with BAP31-negative expression showed significantly lower overall survival rate (P=0.003) compared to patients with BAP31-positive expression. Our results demonstrate that BAP31 may serve as a candidate prognostic marker in colorectal cancer and negative BAP31 expression may lead to more aggressive invasion of colorectal cancer.
MSH2 is required for cell proliferation, cell cycle control and cell invasiveness in colorectal cancer cells
Kai Shen,YingJiang Ye,KeWei Jiang,Bin Liang,XiaoDong Yang,Shan Wang
Chinese Science Bulletin , 2012, DOI: 10.1007/s11434-012-5258-0
Abstract: We have investigated the role of MSH2, a mismatch repair gene in cell proliferation, cell cycle control and cell invasiveness in the SW480 human colorectal cancer cell line. RNAi-mediated inhibition of MSH2 expression was achieved using MSH2 shRNA lentiviral expression vectors. Effective knockdown of endogenous MSH2 expression was determined by real-time PCR analysis. The most efficient MSH2 knockdown vector was selected for subsequent studies using SW480 cells. Endogenous MSH2 mRNA levels decreased after lentiviral delivery of the MSH2-RNAi, indicating efficient silencing of MSH2 expression in SW480 cells. Cell proliferation, cell cycle progression and cell invasiveness were quantified by MTT assays, flow cytometry and transwell assays, respectively. RNAi-mediated inhibition of MSH2 expression in SW480 cells resulted in decreased cell proliferation, cell cycle arrest at the G0/G1 phase and decreased cell invasiveness. Taken together, these results provide evidence that MSH2 stimulates cell proliferation, promotes cell cycle progression and positively regulates cell invasiveness.
Some constancy results for harmonic maps from non-contractable domains into spheres
Kewei Zhang
Electronic Journal of Differential Equations , 2000,
Abstract: We use the Pohozaev identity on sub-domains of a Euclidean $r$-neighbourhood for a closed or broken curve to show that harmonic maps from such domains into spheres with constant boundary value remain constant.
Stochastic And-Or Grammars: A Unified Framework and Logic Perspective
Kewei Tu
Computer Science , 2015,
Abstract: Stochastic And-Or grammars (AOG) extend traditional stochastic grammars of language to model other types of data such as images and events. In this paper we propose a representation framework of stochastic AOGs that is agnostic to the type of the data being modeled and thus unifies various domain-specific AOGs. Many existing grammar formalisms and probabilistic models in natural language processing, computer vision, and machine learning can be seen as special cases of this framework. We also propose a domain-independent inference algorithm of stochastic context-free AOGs and show its tractability under a reasonable assumption. Furthermore, we provide an interpretation of stochastic context-free AOGs as a subset of first-order probabilistic logic, which connects stochastic AOGs to the field of statistical relational learning. Based on the interpretation, we clarify the relation between stochastic AOGs and a few existing statistical relational models.
Initiating, growing and cracking of hydrogen blisters
Ren Xuechong,Shan Guangbin,Chu Wuyang,Su Yanjing,Gao Kewei,Qiao Lijie,Jiang Bo,Chen Gang,Cui Yinhui
Chinese Science Bulletin , 2005, DOI: 10.1360/982004-581
Abstract: The growing process of a hydrogen blister in a wheel steel was observed in situ with an optical microscope, and the fracture surfaces formed from broken blisters on a wheel steel and bulk metallic glass were investigated. The initiating, growing, cracking and breaking of hydrogen blisters are as follows. Supersaturated vacancies can increase greatly during charging and gather together into a vacancy cluster (small cavity). Hydrogen atoms become hydrogen molecules in the vacancy cluster and hydrogen molecules can stabilize the vacancy cluster. The small cavity becomes the nucleus of hydrogen blister. The blister will grow with entering of vacancies and hydrogen atoms. With increasing hydrogen pressure, plastic deformation occurs first, the hydrogen blister near the surface extrudes, and then cracks initiate along the wall of the blister with further increasing hydrogen pressure. A cracked blister can grow further through propagating of cracks until it breaks.
Multi-Scale Object Perception with Embedding Textural Space  [PDF]
Kewei Wu, Zhao Xie, Jun Gao
International Journal of Intelligence Science (IJIS) , 2012, DOI: 10.4236/ijis.2012.22005
Abstract: This paper mainly focuses on the issues about generic multi-scale object perception for detection or recognition. A novel computational model in visually-feature space is presented for scene & object representation to purse the underlying textural manifold statistically in nonparametric manner. The associative method approximately makes perceptual hierarchy in human-vision biologically coherency in specific quad-tree-pyramid structure, and the appropriate scale-value of different objects can automatically be selected by evaluating from well-defined scale function without any priori knowledge. The sufficient experiments truly demonstrate the effectiveness of scale determination in textural manifold with object localization rapidly.
Adhesive contact of a rigid sphere to finitely stretched substrates
LingHui He,KeWei Ding
Chinese Science Bulletin , 2009, DOI: 10.1007/s11434-009-0328-7
Abstract: Exemplified by adhesive contact of a rigid sphere with equiaxially stretched elastomeric substrates, the effect of finite pre-strain on adhesion behavior is studied based on the JKR model. The analysis indicates that the pre-strain reduces the contact area between the sphere and the substrate, but does not alter the adhesion strength.
Research on the Parameters of Environment-Friendly Recycled Road Materials
Yuanchen Guo,Kewei Sun
Journal of Sustainable Development , 2009, DOI: 10.5539/jsd.v2n3p102
Abstract: The feasibility of propagating road materials in the use of contracture wastes especially contented great amount waste bricks were researched in three aspects: Performance of recycled aggregate, mechanical properties of renewable road materials, compression - splitting indexes. Conclusions are as following: recycled aggregates contenting great amount waste bricks in place of natural aggregates are feasible from the point of view on technical and technological. The performances of recycled aggregates have a significant impact on properties of renewable road material. Whether it is cement stabilized RCA material or cements stabilized RCBA material, age and cement in the age-dose studies, with the increase in cement dose, mechanical properties growth trends are similar. Compared to unconfined compressive strength and resilient modulus, splitting strength is much closed in terms of growth trends or Growth value at the cement doses or ages. Good Relevance correlation are existed among resilient modulus, compressive strength and splitting strength: ,RCA: .
Pathophysiological Significance of Hepatic Apoptosis
Kewei Wang,Bingliang Lin
ISRN Hepatology , 2013, DOI: 10.1155/2013/740149
Abstract: Apoptosis is a classical pathological feature in liver diseases caused by various etiological factors such as drugs, viruses, alcohol, and cholestasis. Hepatic apoptosis and its deleterious effects exacerbate liver function as well as involvement in fibrosis/cirrhosis and carcinogenesis. An imbalance between apoptotic and antiapoptotic capabilities is a prominent characteristic of liver injury. The regulation of apoptosis and antiapoptosis can be a pivotal step in the treatment of liver diseases. 1. Apoptosis Apoptosis is a process of programmed cell death. Apoptotic cells are characterized by energy-dependent biochemical mechanisms and obvious morphological changes [1, 2]. These features include membrane blebbing, cell shrinkage, nuclear chromatin condensation, and chromosomal DNA fragmentation. The apoptotic process deletes single cell or small clusters of cells without inflammatory response [3]. Apoptotic cells die in a controlled and regulated fashion. This makes apoptosis distinct from other uncontrolled modes of cell death such as necrosis, necroptosis, autophagy, and cornification [4]. Uncontrolled cell death leads to cell lysis, inflammatory response, and serious health problems [5]. Apoptosis is associated with multiple pathophysiological functions. During the embryological stage of mammals, apoptosis is important for the normal development of organs [6]. In adults, apoptosis regulates physiological processes (e.g., removing aged cells) and maintains tissue homeostasis [7]. Dysfunction or dysregulation of the apoptotic program is implicated in a variety of congenital anomalies and pathological conditions such as tumorigenesis, autoimmune diseases, neurodegenerative disorders, and others [8]. 2. Hepatic Apoptosis Hepatic apoptosis, as name indicated, means cell suicide in liver. The hepatic apoptosis is different from hepatocyte apoptosis. The hepatocyte apoptosis describes the apoptotic cell death in only hepatocytes (one type of liver cells), but the hepatic apoptosis reflects the interaction of manifold cells in liver and represents a comprehensive outcome of multiple effects. The liver is an organ consisting of several phenotypically distinct cell types, for example, hepatocytes, cholangiocytes, stellate cells, sinusoidal endothelial cells, Kupffer cells, oval cells, and so forth [9]. Predominant hepatocytes make up 70–80% of the liver cells [10]. Hepatocytes manufacture critical circulating proteins, generate bile acid-dependent bile flow, detoxify endo- and xenobiotics, and regulate intermediary metabolism [11]. Hepatocyte injury results
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