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Search Results: 1 - 10 of 298944 matches for " Kaur J "
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Nonpathogenic Fusarium as a Biological Control Agent
R. Kaur,J. Kaur,Rama S. Singh
Plant Pathology Journal , 2010,
Abstract: Fusarium oxysporum is an important fungal group among the soil bone microflora. These strains are well-known for inducing wilt or root rots in important agricultural crops worldwide and some occur only as a saprophytes in rhizosphere of plants. There are certain strains which are nonpathogenic and protect plants from pathogenic strains. Based on phenotypic and genetic studies F. oxysporum showed a great diversity among its populations. The nonpathogenic strains, which were first isolated from suppressive soils strains showed several modes of action against pathogenic strains and thus exploited as biocontrol agents. These nonpathogenic strains suppress pathogens by competing for nutrients in the soil, reduce their chlamydospore germination, compete for infection sites on the root and induce systemic resistance in plant when invade host plant species before the pathogen. The nonpathogenic strains are formulated in talc and charcoal based media and commercial formulations are also available. These strains of Fusarium has been successfully combined with other biocontrol agents to obtain a effective biocontrol of plant pathogens. For application of nonpathogenic Fusarium under field condition some additional research is needed in several areas including: field studies and integration into production systems; risk assessment; and genetic improvement of biocontrol agents.
Sedation and analgesia in pediatric intensive care unit
Khilnani P,Kaur J
Indian Journal of Critical Care Medicine , 2003,
Abstract: Common indications of sedation in the PICU (Pediatric intensive care unit) include mechanical Ventilation and various procedures performed in the PICU and in radiology or endoscopy suites. Sedation potentiates the effect of narcotics, thereby ensuring better comfort and analgesia. Sedation is a mandatory prerequisite prior to and during administration of neuromuscular blockers. This review includes practical pharmacology and uses of commonly used agents in the PICU.
Effect of Task-specific Training on Gait Parameters in Hemiparetic Stroke Patients
Kaur J,Kumar A.
Indian Journal of Physical Medicine and Rehabilitation , 2009,
Abstract: Functional mobility is viewed as essential to enabling anindividual to engage in full range of life areas and is centralto enabling the individual to participate in live situations.The walking after stroke is characterized by slow gaitspeed, poor endurance and in the quality and adaptabilityof walking patterns. The instantaneous adaptation tospeed and load changes during over ground locomotionhas major implication for mobility after stroke. Taskspecific training is a therapeutic approach based onSystem theory given by Berstein in 1967 to retrain thepatients with movement disorders.It is a one group pre test post test quasi experiment designwith the objective of to find out the effectiveness of taskspecific training on gait parameters in right hemipareticpatients.10 right hemiparetic patients (n=10) of either sex in theage group of 40-65 yrs (mean age 54.44 yrs) were selectedby convenient sampling method and were assigned inone group. Different tasks in a prefixed pattern aiming atfunctional activities were introduced over a period of 40min duration per day/session, 3 days a week and for totalduration of 4 weeks. i.e. total 12 sessions.Main outcome measures: Changes were measured interms of Cadence, Step length, Stride length and GaitVelocity.Significant statistical improvement was measured in termsof cadence (P= 0.038) step length and gait velocity (P=0.033) whereas there is no significant statisticalimprovement in Step length (P= 0.140) and Stride length(P= 0.162).
Zinc Deficiency and Zinc Therapy Efficacy with Reduction of Serum Free Copper in Alzheimer’s Disease
George J. Brewer,Sukhvir Kaur
International Journal of Alzheimer's Disease , 2013, DOI: 10.1155/2013/586365
Abstract: We are in the midst of an epidemic of Alzheimer’s disease (AD) in developed countries. We have postulated that ingestion of inorganic copper from drinking water and taking supplement pills and a high fat diet are major causative factors. Ingestion of inorganic copper can directly raise the blood free copper level. Blood free copper has been shown by the Squitti group to be elevated in AD, to correlate with cognition, and to predict cognition loss. Secondly, we have shown that AD patients are zinc deficient compared to age matched controls. Zinc is important in neuronal protection. We carried out a 6-month small double blind trial of a new zinc formulation on AD patients. We found that in patients 70 years and older, zinc therapy protected against cognition decline compared to placebo controls. We also found that zinc therapy significantly lowered blood free copper levels. So zinc efficacy could be due to restoring neuronal zinc levels, to lowering blood free copper levels, or to both. 1. Introduction We are in the midst of an epidemic of Alzheimer’s disease (AD), particularly in developed countries [1]. We have previously hypothesized that ingestion of inorganic copper from drinking water and supplement pills and a high fat diet are main causal factors in AD [2–5]. These two factors interact synergistically because copper oxidizes fats to molecules that are toxic, particularly to neurons. The epidemic correlates temporally with the use of copper plumbing in developed countries, and there is a great amount of additional data that draws the net tighter around a causative role for inorganic copper. Inorganic copper must not be confused with organic copper, the copper in food that is safely bound to protein. Inorganic copper is a simple salt of copper, not bound to anything, and is in part handled differently by the intestinal absorption mechanism, such that some of it contributes immediately to the serum free copper pool [6]. This pool has been shown by Squitti and her group to be expanded in AD [7], to correlate negatively with cognition [8], and to predict deterioration in cognition [9]. So, our inorganic copper hypothesis fits well with the work of the Squitti et al. group. While we believe ingestion of inorganic copper and a high fat diet are major causal factors, there are a number of other known risk factors in AD. These include having an apolipoprotein E4 allele [10], having elevated homocysteine levels [11], or having certain alleles of the iron management genes, hemochromatosis [12] and transferrin [13]. These latter fit with the copper hypothesis
The effect of prostaglandin synthase inhibitor, aspirin on the rat intestinal membrane structure and function
Kaur,G.; Kaur,J.; Mittal,N.; Nath Sanyal,S.;
Nutrición Hospitalaria , 2010,
Abstract: aspirin at a dose of 50 mg/kg body weight was found to decrease the activity of the rat intestinal brush border membrane (bbm) - associated enzymes such as the sucrase, lactase, maltase and alkaline phosphatase. aspirin treatment also led to a decrease in the microviscosity in the native as well as the benzyl alcohol treated membrane which might be due to the lipid peroxidative damage in the membrane. physical correlation of the membrane oxidative damage was evident as the fourier transformation infra red (ftir) study of the aspirin treated membrane, which include an increased proportion of gauche to trans conformer, shift in the methylene c-h asymmetric and symmetric stretching frequencies, c = o double bond stretching, nh bending, antisymmetric (n)-ch3 bending, c-n stretching and antisymmetric cnc stretching while there was no change in the ch2 wagging and twisting as well as in nh-bending amide bond i and ii. aspirin treatment also caused an alteration in the glucose and histidine transport, as evident by a decreased vmax value while the apparent km remaining unchanged in the control and aspirin-treated animals confirming that there was no change in the substrate affinity constant of the membrane transport proteins for the glucose and the basic amino acid, although the rate of transport decreased considerably. there was a decrease noted in the energy of activation of glucose and histidine transport when studied at different temperature but no change in the temperature of phase transition in the bbm with aspirin treatment, thus implying that perhaps the thermotropic phase transition in the membrane may have relatively little effect on the transport processes. the result suggests an underlying molecular mechanism indicating the implied membrane damage by aspirin, an important member of the non-steroidal antiinflammatory drug (nsaid) family which could possibly through an oxidative damage may lead to an altered molecular structure, physical state and biological func
Chemopreventive response of diclofenac, a non-steroidal anti-inflammatory drug in experimental carcinogenesis
Kaur Saini,M.; Kaur,J.; Sharma,P.; Nath Sanyal,S.;
Nutrición Hospitalaria , 2009,
Abstract: the chemopreventive response was evaluated of nonsteroidal anti-inflammatory drug, diclofenac, a preferential cyclooxygenase-2 (cox-2) inhibitor in 1,2-dimethyhydrazine (dmh)-induced colon cancer in rat model. the signs of neoplasm were evident in the animals receiving 30mg of dmh per kg body weight in a weekly s.c injection for six weeks. the putative biomarker of carcinogenesis was visible in the form of multiple plaque lesions in dmh treatment and then regression seen in those animals which also received an oral dose of diclofenac, 8 mg/kg body weight whereas no such macroscopic neoplastic lesions were seen in the animals receiving diclofenac only or the control animals receiving the vehicle of the drug. histopathological results showed the presence of early aberrant changes in the form of severe dysplasia and also numerous crypt fissions in the apical surface of the colonic mucosa. a very high expression of cox-2 was seen in the colonic epithelium of dmh-treated rats, as analyzed by immunohistochemistry. also, the apoptotic events were assessed by terminal deoxynucleotidyl dutp nick end labeling (tunel) assay, where the dmh group shows few number of tunel positive cells which dramatically increased in the diclofenac treatment. the results suggest that diclofenac could be an effective chemopreventive agent in colon cancer, where perhaps apoptosis plays a very dominant end effect in cancer cell killings.
Lichen Aureus : Atypcial Presentation Showing Remarkable Therapeutic Response
Kaur Charandeep,kaur Sukhjot,Thami G . P,Kanwar A . J
Indian Journal of Dermatology , 2002,
Abstract: A typical patient with zosteriform distribution of rust-coloured to yellowish brown patches over the right leg is reported. The histopathology was consistent with lichen aureus.
Chemopreventive response of diclofenac, a non-steroidal anti-inflammatory drug in experimental carcinogenesis Respuesta quimiopreventiva del diclofenaco, un fármaco antiinflamatorio no esteroideo en la carcinogénesis de colon experimental
M. Kaur Saini,J. Kaur,P. Sharma,S. Nath Sanyal
Nutrición Hospitalaria , 2009,
Abstract: The chemopreventive response was evaluated of nonsteroidal anti-inflammatory drug, Diclofenac, a preferential cyclooxygenase-2 (COX-2) inhibitor in 1,2-dimethyhydrazine (DMH)-induced colon cancer in rat model. The signs of neoplasm were evident in the animals receiving 30mg of DMH per kg body weight in a weekly s.c injection for six weeks. The putative biomarker of carcinogenesis was visible in the form of multiple plaque lesions in DMH treatment and then regression seen in those animals which also received an oral dose of Diclofenac, 8 mg/kg body weight whereas no such macroscopic neoplastic lesions were seen in the animals receiving Diclofenac only or the control animals receiving the vehicle of the drug. Histopathological results showed the presence of early aberrant changes in the form of severe dysplasia and also numerous crypt fissions in the apical surface of the colonic mucosa. A very high expression of COX-2 was seen in the colonic epithelium of DMH-treated rats, as analyzed by immunohistochemistry. Also, the apoptotic events were assessed by terminal deoxynucleotidyl dUTP nick end labeling (TUNEL) assay, where the DMH group shows few number of TUNEL positive cells which dramatically increased in the Diclofenac treatment. The results suggest that Diclofenac could be an effective chemopreventive agent in colon cancer, where perhaps apoptosis plays a very dominant end effect in cancer cell killings. Se evaluó la respuesta quimiopreventiva del fármaco antiinflamatorio no esteroideo, diclofenaco, un inhibidor preferente de la ciclooxigenasa-2 (Cox-2), en el cáncer de colon inducido por 1,2-dimetilhidracina (DMH) en un modelo de rata. Los signos de neoplasia fueron evidentes en los animales que recibieron 30 mg de DMH por kg de peso corporal mediante inyecciones s.c. semanales durante 6 semanas. El biomarcador putativo de la carcinogénesis fue visible en la forma de múltiples lesiones en placas con el tratamiento de DMH y la posterior regresión apreciada en los animales que también recibieron una dosis oral de diclofenaco, 8 mg/kg de peso corporal, mientras que no se vieron tales lesiones neoplásicas macroscópicas en los animales que recibieron solamente el diclofenaco o en los animales control que recibieron el vehículo del fármaco. Los resultados histopatológicos mostraron la presencia de cambios aberrantes precoces en la forma de una displasia intensa y también numerosas fisiones de las criptas en la superficie apical de la mucosa colónica. Se vio una expresión muy elevada de Cox-2 en el epitelio colónico de las ratas tratadas con DMH, al analiza
The effect of prostaglandin synthase inhibitor, aspirin on the rat intestinal membrane structure and function El efecto del inhibidor de la sintasa de prostaglandina, aspirina, sobre la estructura y función de la membrana intestinal de la rata
G. Kaur,J. Kaur,N. Mittal,S. Nath Sanyal
Nutrición Hospitalaria , 2010,
Abstract: Aspirin at a dose of 50 mg/kg body weight was found to decrease the activity of the rat intestinal brush border membrane (BBM) - associated enzymes such as the sucrase, lactase, maltase and alkaline phosphatase. Aspirin treatment also led to a decrease in the microviscosity in the native as well as the benzyl alcohol treated membrane which might be due to the lipid peroxidative damage in the membrane. Physical correlation of the membrane oxidative damage was evident as the Fourier Transformation Infra Red (FTIR) study of the Aspirin treated membrane, which include an increased proportion of gauche to trans conformer, shift in the methylene C-H asymmetric and symmetric stretching frequencies, C = O double bond stretching, NH bending, antisymmetric (N)-CH3 bending, C-N stretching and antisymmetric CNC stretching while there was no change in the CH2 wagging and twisting as well as in NH-bending amide bond I and II. Aspirin treatment also caused an alteration in the glucose and histidine transport, as evident by a decreased Vmax value while the apparent Km remaining unchanged in the control and Aspirin-treated animals confirming that there was no change in the substrate affinity constant of the membrane transport proteins for the glucose and the basic amino acid, although the rate of transport decreased considerably. There was a decrease noted in the energy of activation of glucose and histidine transport when studied at different temperature but no change in the temperature of phase transition in the BBM with Aspirin treatment, thus implying that perhaps the thermotropic phase transition in the membrane may have relatively little effect on the transport processes. The result suggests an underlying molecular mechanism indicating the implied membrane damage by Aspirin, an important member of the non-steroidal antiinflammatory drug (NSAID) family which could possibly through an oxidative damage may lead to an altered molecular structure, physical state and biological functions of the intestinal membrane. Se encontró que la aspirina a una dosis de 50 mg/kg de peso corporal disminuye la actividad de las enzimas asociadas a la membrana con borde en cepillo (MBC) del intestino de la rata como la sucrasa, lactasa, maltasa y fosfata alcalina. El tratamiento con aspirina también produjo una disminución de la microviscosidad en la membrana nativa así como en la membrana tratada con alcohol bencílico, lo que podría deberse a la lesión de peroxidación lipídica de la membrana. La correlación física de la lesión oxidativa de la membrana fue evidente como mostró el estudio
Advances in Intrusion Detection System for WLAN  [PDF]
Ravneet Kaur
Advances in Internet of Things (AIT) , 2011, DOI: 10.4236/ait.2011.13007
Abstract: A wireless network is not as secure as compare the wired network because the data is transferred on air so any intruder can use hacking techniques to access that data. Indeed it is difficult to protect the data and provide the user a secure information system for lifetime. An intrusions detection system aim to detect the different attacks against network and system. An intrusion detection system should be capable for detecting the misuse of the network whether it will be by the authenticated user or by an attacker. Cross layer based technique help to make decision based on two layer physical layer where we compute RSS value and on MAC layer where one compute RTS-CTS time taken. This will reduce the positive false rate.They detect attempts and active misuse either by legitimate users of the information systems or by external. The paper has higlighted the advances in intrusion detection in wireless local area network.
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