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Search Results: 1 - 10 of 78283 matches for " Jianliang Chen "
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Comparative Efficacy of Different Herbicides for Weed Management and Yield Attributes in Wheat  [PDF]
Shah Fahad, Lixiao Nie, Amjadur Rahman, Chang Chen, Chao Wu, Shah Saud, Jianliang Huang
American Journal of Plant Sciences (AJPS) , 2013, DOI: 10.4236/ajps.2013.46152
Abstract:

Weed competes with crops for water, nutrients and light so weed infestation is one of the major threats to crop. Present investigation was aimed to asses the comparative efficacy of different herbicides for weed management in wheat crop under agro-climatic conditions of Pakistan. This experiment was laid out in a randomized complete block design (RCBD) design with five replications. Different herbicides were used for weed management in wheat crop. The post emergence application of herbicides included Aim 40 DF @ 0.02 kg a.i. ha-1, Agritop 500 GL-1 @ 0.43 kg a.i. ha-1, Isoproturon 50 WP @ 1 kg a.i. ha-1, Puma super 75 EW @ 0.75 kg, Topik 15 WP @ 0.04 kg and Buctril super 60 EC @ 0.45 kg. For comparison hand weeding and weedy check were also included. In each replication six treatments of these six herbicides were kept. The significantly affected parameters were fresh weed biomass (kg·ha-1), thousand grain weight (g), number of tillers m-2, weed control efficiency (%) and grain yield (kg·ha-1). Statistical analysis showed that maximum weed efficiency (84%) was recorded for Isoproturon 50 WP whereas minimum value (37%) was for Aim 40 DF. Similarly maximum number of tillers m-2 (250) was recorded for Isoproturon 50 WP and minimum (133) in weedy check. The herbicide Isoproturon 50 WP @ 1.0 kg a.i. ha-1 was applied at post emergence performed well and exhibited effectively weed control and better yield in wheat.

Edge Colorings of Planar Graphs without 6-Cycles with Two Chords  [PDF]
Ling Xue, Jianliang Wu
Open Journal of Discrete Mathematics (OJDM) , 2013, DOI: 10.4236/ojdm.2013.32016
Abstract:

It is proved here that if a planar graph has maximum degree at least 6 and any 6-cycle contains at most one chord, then it is of class 1.

Genotypic Variations in Terms of NH3 Volatilization in Four Rice (Oryza sativa L.) Cultivars
Mingxia Chen,Jianliang Huang,Kehui Cui,Lixiao Nie
Asian Journal of Plant Sciences , 2009,
Abstract: NH3 volatilization from the cultivated rice (Oryza sativa L.) canopy is one of the major contributing factors of the N loss in the rice production. The objectives of this study are to examine the genotypic variations in NH3 volatilization from canopy among the cultivars and to determine the mechanisms responsible for NH3 volatilization from rice. With these aims, two pot experiments were conducted using Yoshida culture solution and four rice genotypes (the Yangdao-6, the Wuyujing-3, the BG34-8 and the Zhenshan-97) in greenhouse at Huazhong Agricultural University, Wuhan City, China. The amount of ammonia volatilized from different rice cultivars was evaluated at booting, heading and milking stages. At booting stage, inhibitors of glutamine synthetase (methionine sulfoximine (MSO)) and photorespiration (isonicotinyl hydrazine (INH)) were sprayed on leaves at 0.1 mmol L-1 and 2 g L-1, respectively. Genotypic variations in amount of NH3 volatilized from canopy existed among the four rice cultivars at different growth stages and N treatments. According to results, spraying of the MSO at 0.1 mmol L-1 was significantly increased the NH3 volatilization from the rice canopy, while spraying INH at 2 g L-1 decreased the NH3 volatilization. Obtained results were showed that rate of ammonia volatilization was negatively correlated with the leaf GS activity, while the leaf GO activity related positively to the rice ammonia volatilization.
Late gestational lung hypoplasia in a mouse model of the Smith-Lemli-Opitz syndrome
Yu Hongwei,Wessels Andy,Chen Jianliang,Phelps Aimee L
BMC Developmental Biology , 2004,
Abstract: Background Normal post-squalene cholesterol biosynthesis is important for mammalian embryonic development. Neonatal mice lacking functional dehydrocholesterol Δ7-reductase (Dhcr7), a model for the human disease of Smith-Lemli-Opitz syndrome, die within 24 hours of birth. Although they have a number of biochemical and structural abnormalities, one cause of death is from apparent respiratory failure due to developmental pulmonary abnormalities. Results In this study, we characterized further the role of cholesterol deficiency in lung development of these mice. Significant growth retardation, beginning at E14.5~E16.5, was observed in Dhcr7-/- embryos. Normal lobation but smaller lungs with a significant decrease in lung-to-body weight ratio was noted in Dhcr7-/- embryos, compared to controls. Lung branching morphogenesis was comparable between Dhcr7-/- and controls at early stages, but delayed saccular development was visible in all Dhcr7-/- embryos from E17.5 onwards. Impaired pre-alveolar development of varying severity, inhibited cell proliferation, delayed differentiation of type I alveolar epithelial cells (AECs) and delayed vascular development were all evident in knockout lungs. Differentiation of type II AECs was apparently normal as judged by surfactant protein (SP) mRNAs and SP-C immunostaining. A significant amount of cholesterol was detectable in knockout lungs, implicating some maternal transfer of cholesterol. No significant differences of the spatial-temporal localization of sonic hedgehog (Shh) or its downstream targets by immunohistochemistry were detected between knockout and wild-type lungs and Shh autoprocessing occurred normally in tissues from Dhcr7-/- embryos. Conclusion Our data indicated that cholesterol deficiency caused by Dhcr7 null was associated with a distinct lung saccular hypoplasia, characterized by failure to terminally differentiate alveolar sacs, a delayed differentiation of type I AECs and an immature vascular network at late gestational stages. The molecular mechanism of impaired lung development associated with sterol deficiency by Dhcr7 loss is still unknown, but these results do not support the involvement of dysregulated Shh-Patched-Gli pathway in causing this defect.
Incremental Nonnegative Matrix Factorization for Face Recognition
Wen-Sheng Chen,Binbin Pan,Bin Fang,Ming Li,Jianliang Tang
Mathematical Problems in Engineering , 2008, DOI: 10.1155/2008/410674
Abstract: Nonnegative matrix factorization (NMF) is a promising approach for local feature extraction in face recognition tasks. However, there are two major drawbacks in almost all existing NMF-based methods. One shortcoming is that the computational cost is expensive for large matrix decomposition. The other is that it must conduct repetitive learning, when the training samples or classes are updated. To overcome these two limitations, this paper proposes a novel incremental nonnegative matrix factorization (INMF) for face representation and recognition. The proposed INMF approach is based on a novel constraint criterion and our previous block strategy. It thus has some good properties, such as low computational complexity, sparse coefficient matrix. Also, the coefficient column vectors between different classes are orthogonal. In particular, it can be applied to incremental learning. Two face databases, namely FERET and CMU PIE face databases, are selected for evaluation. Compared with PCA and some state-of-the-art NMF-based methods, our INMF approach gives the best performance.
VEGF Promotes the Transcription of the Human PRL-3 Gene in HUVEC through Transcription Factor MEF2C
Jianliang Xu, Shaoxian Cao, Lu Wang, Rui Xu, Gong Chen, Qiang Xu
PLOS ONE , 2011, DOI: 10.1371/journal.pone.0027165
Abstract: Phosphatase of regenerating liver 3 (PRL-3) is known to be overexpressed in many tumors, and its transcript level is high in the vasculature and endothelial cells of malignant tumor tissue. However, the mechanism(s) underlying its enhanced expression and its function in endothelial cells remain unknown. Here, we report that vascular endothelial growth factor (VEGF) can induce PRL-3 transcription in human umbilical vein endothelial cells (HUVEC). An analysis of its 5′UTR revealed that PRL-3 transcription is initiated from two distinct sites, which results in the formation of the two transcripts, PRL-3-iso1 and PRL-3-iso2, but only the latter is up-regulated in HUVEC by VEGF. The PRL-3-iso2 promoter region includes two functional MEF2 (myocyte enhancer factor2) binding sites. The over-expression of the constitutively active form of MEF2C promotes the abundance of the PRL-3-iso2 transcript in a number of human cell lines. The siRNA-induced knockdown of MEF2C abolished the stimulative effect of VEGF on PRL-3 transcript in HUVEC, indicating that the VEGF-induced promotion of PRL-3 expression requires the presence of MEF2C. Finally, blocking PRL-3 activity or expression suppresses tube formation by HUVEC. We suggest that PRL-3 functions downstream of the VEGF/MEF2C pathway in endothelial cells and may play an important role in tumor angiogenesis.
Selective reconstitution of liver cholesterol biosynthesis promotes lung maturation but does not prevent neonatal lethality in Dhcr7 null mice
Hongwei Yu, Man Li, G Stephen Tint, Jianliang Chen, Guorong Xu, Shailendra B Patel
BMC Developmental Biology , 2007, DOI: 10.1186/1471-213x-7-27
Abstract: We generated mice that carried a liver-specific human DHCR7 transgene whose expression was driven by the human apolipoprotein E (ApoE) promoter and its associated liver-specific enhancer. These mice were then crossed with Dhcr7+/- mutants to generate Dhcr7-/- mice bearing a human DHCR7 transgene. Robust hepatic transgene expression resulted in significant improvement of cholesterol homeostasis with cholesterol concentrations increasing to 80~90 % of normal levels in liver and lung. Significantly, cholesterol deficiency in brain was not altered. Although late gestational lung sacculation defect reported previously was significantly improved, there was no parallel increase in postnatal survival in the transgenic mutant mice.The reconstitution of DHCR7 function selectively in liver induced a significant improvement of cholesterol homeostasis in non-brain tissues, but failed to rescue the neonatal lethality of Dhcr7 null mice. These results provided further evidence that CNS defects caused by Dhcr7 null likely play a major role in the lethal pathogenesis of Dhcr7-/- mice, with the peripheral organs contributing the morbidity.The role of cholesterol in embryonic development is an important question in biology, with significant ramifications for human disease [1,2]. Defects in post-squalene cholesterol biosynthesis, such as in Smith-Lemli-Opitz syndrome (SLOS, MIM 270400) or desmosterolosis (MIM 603398), disrupt the synthesis of cholesterol and cause a variety of severe developmental abnormalities [3-8]. SLOS is a complex inborn error of cholesterol biosynthesis caused by mutations of the 3β-hydroxysterol-Δ7 reductase gene (DHCR7) [9-11]. The lack of Dhcr7 expression in mouse mimics the early postnatal lethality observed in severely affected individuals (those with the condition formerly referred to as SLOS type II). The biological changes in development caused by disruption of normal cholesterol biosynthesis that result in this early postnatal lethality remain obscure. A
Study on the international resonance of nonlinear vibration of axially moving beams
轴向运动梁非线性振动内共振研究

Chen Shuhui,Huang Jianliang,
陈树辉
,黄建亮

力学学报 , 2005,
Abstract: 采用多元L-P方法分析轴向运动梁横向非线性振动的内共振,首先根据哈密顿原理建立轴向运动梁的横向振动微分方程,然后利用Galerkin方法分离时间和空间变量,再采用多元L-P方法进行求解,推导了内共振条件下频率-振幅方程的求根判别式,理论分析发现内共振与强迫力的振幅有关,而且可以从理论上决定这一界乎不同内共振的强迫力振幅的临界值,典型算例获得了轴向运动梁横向非线性振动内共振复杂的频率一振幅响应曲线,揭示了很多复杂而有趣的非线性振动特有的现象,多元L-P方法的数值结果,在小振幅时与IHB法的结果一致。
A note for coriolis acceleration of axially moving beams
关于轴向运动梁科氏加速度的注释

CHEN Shuhui,LIU Shougui,HUANG Jianliang,
陈树辉
,刘守圭,黄建亮

力学与实践 , 2006,
Abstract: 对轴向运动梁横向非线性振动方程中出现的科氏加速度从理论力学的角度加以注释.
Late gestational lung hypoplasia in a mouse model of the Smith-Lemli-Opitz syndrome
Hongwei Yu, Andy Wessels, Jianliang Chen, Aimee L Phelps, John Oatis, G Stephen Tint, Shailendra B Patel
BMC Developmental Biology , 2004, DOI: 10.1186/1471-213x-4-1
Abstract: In this study, we characterized further the role of cholesterol deficiency in lung development of these mice. Significant growth retardation, beginning at E14.5~E16.5, was observed in Dhcr7-/- embryos. Normal lobation but smaller lungs with a significant decrease in lung-to-body weight ratio was noted in Dhcr7-/- embryos, compared to controls. Lung branching morphogenesis was comparable between Dhcr7-/- and controls at early stages, but delayed saccular development was visible in all Dhcr7-/- embryos from E17.5 onwards. Impaired pre-alveolar development of varying severity, inhibited cell proliferation, delayed differentiation of type I alveolar epithelial cells (AECs) and delayed vascular development were all evident in knockout lungs. Differentiation of type II AECs was apparently normal as judged by surfactant protein (SP) mRNAs and SP-C immunostaining. A significant amount of cholesterol was detectable in knockout lungs, implicating some maternal transfer of cholesterol. No significant differences of the spatial-temporal localization of sonic hedgehog (Shh) or its downstream targets by immunohistochemistry were detected between knockout and wild-type lungs and Shh autoprocessing occurred normally in tissues from Dhcr7-/- embryos.Our data indicated that cholesterol deficiency caused by Dhcr7 null was associated with a distinct lung saccular hypoplasia, characterized by failure to terminally differentiate alveolar sacs, a delayed differentiation of type I AECs and an immature vascular network at late gestational stages. The molecular mechanism of impaired lung development associated with sterol deficiency by Dhcr7 loss is still unknown, but these results do not support the involvement of dysregulated Shh-Patched-Gli pathway in causing this defect.Cholesterol is a necessary membrane constituent of all mammalian, reptile and avian cells, as well as a few other organisms, but not all eukaryotic organisms. In the latter cases, such as plants and fungi, cholesterol-lik
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