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Search Results: 1 - 10 of 3441 matches for " Howard Gardner "
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Inteligencias múltiples veinte a os después
Howard Gardner
Revista de Psicología y Educación , 2005,
Abstract: A menudo me han preguntado cómo tuve la idea dela teoría de las inteligencias múltiples. Probablemente la respuesta más veraz es “no lo sé”. Sin embargo, esta respuesta no satisface ala persona que lo pregunta ni, para ser franco,me satisface a mí. Con la ayuda de la memoria,mencionaría los siguientes factores, unos remotos y otros estrecha y directamente vinculados a mis descubrimientos.
Inteligencias múltiples
Howard Gardner
Revista de Psicología y Educación , 2005,
Abstract: Se define mejor a los seres humanos diciendo que tienen ocho, quizá nueve, tipos de inteligencia, incluyendo la musical, la espacial y la cinestésica que diciendo que sólo tienen una inteligencia definida por el CI.
鲁利亚的一生
Howard Gardner,魏明庠
心理科学进展 , 1985,
Abstract: 当鲁利亚还是一个少年的时候,他就表现出胜任心理学研究的才能。他建立了自己的精神分析小组,创办了一个刊物。在二十岁的时候,他用几种语言掌握了心理学文献,并与他那个时代的伟大学者(包括弗洛伊德)通信。在后四十年里,他在儿童、罪犯、俄国农民、同卵双生子、脑外伤病人、智力落后病人等的思惟、情绪过程方面进行了卓越的研究。到了晚年,毫无疑问,他成了苏联最杰出的心理学家。鲁利亚工作的一个特点是专业领域经常改变,他每选定一个工作领域时,往往被迫放弃,转移到一个他不熟悉的领域上来。
鲁利亚的一生
howard,gardner,魏明庠
心理科学进展 , 1985,
Abstract: ?<正>当鲁利亚还是一个少年的时候,他就表现出胜任心理学研究的才能。他建立了自己的精神分析小组,创办了一个刊物。在二十岁的时候,他用几种语言掌握了心理学文献,并与他那个时代的伟大学者(包括弗洛伊德)通信。在后四十年里,他在儿童、罪犯、俄国农民、同卵双生子、脑外伤病人、智力落后病人等的思惟、情绪过程方面进行了卓越的研究。到了晚年,毫无疑问,他成了苏联最杰出的心理学家。鲁利亚工作的一个特点是专业领域经常改变,他每选定一个工作领域时,往往被迫放弃,转移到一个他不熟悉的领域上来。
A Quantitative Assessment of the Morphology of the Piriform Aperture as an Indicator of Race  [PDF]
Seth Gardner
Forensic Medicine and Anatomy Research (FMAR) , 2015, DOI: 10.4236/fmar.2015.31002
Abstract: Piriform apertures from skulls in the Bass Collection at the University of Tennessee were examined. The morphology of the perform aperture from digital images was captured using Adobe Measuring Tool 9.0 and data analyzed with SPSS 17.0. Twenty-four linear measurements from a central point of the aperture as well as the perimeter were evaluated to quantify a difference between Black and White populations. The statistical analyses employed Discriminate Functional Analysis followed by Stepwise analysis. Discriminate functions were generated to predict to which group a skull belonged. A discriminate function produced an accuracy of 77.4%. Step-wise discriminate function analysis, using only three variables, produced an accuracy of 79.0%.
Unusual Drainage of the Right Testicular Vein: A Case Report  [PDF]
Seth Gardner
Case Reports in Clinical Medicine (CRCM) , 2015, DOI: 10.4236/crcm.2015.46046
Abstract:
Routine dissection was carried out on a 75-year-old male cadaver and the drainage pattern of the testicular veins was identified and photographed. Dissection showed that the right testicular vein demonstrated an abnormal drainage pattern by terminating into the right renal vein. The left testicular vein demonstrated a normal drainage pattern by terminating into the left renal vein. The unusual drainage of the right testicular vein into the right renal vein may complicate hemodynamics thus causing a varicocele. Complications of a varicocele could lead to testis atrophy and/or infertility. Knowing the anatomical variants of the testicular veins drainage pattern may help the surgeon avoid potential complications during routine laparoscopic procedures and may also uncover a reason for male infertility.
Islands Containing Slowly Hydrolyzable GTP Analogs Promote Microtubule Rescues
Carolina Tropini, Elizabeth A. Roth, Marija Zanic, Melissa K. Gardner, Jonathon Howard
PLOS ONE , 2012, DOI: 10.1371/journal.pone.0030103
Abstract: Microtubules are dynamic polymers of GTP- and GDP-tubulin that undergo stochastic transitions between growing and shrinking phases. Rescues, the conversion from shrinking to growing, have recently been proposed to be to the result of regrowth at GTP-tubulin islands within the lattice of growing microtubules. By introducing mixed GTP/GDP/GMPCPP (GXP) regions within the lattice of dynamic microtubules, we reconstituted GXP islands in vitro (GMPCPP is the slowly hydrolyzable GTP analog guanosine-5′-[(α,β)-methyleno]triphospha?te).We found that such islands could reproducibly induce rescues and that the probability of rescue correlated with both the size of the island and the percentage of GMPCPP-tubulin within the island. The islands slowed the depolymerization rate of shortening microtubules and promoted regrowth more readily than GMPCPP seeds. Together, these findings provide new mechanistic insights supporting the possibility that rescues could be triggered by enriched GTP-tubulin regions and present a new tool for studying such rescue events in vitro.
Molecular breakpoint cloning and gene expression studies of a novel translocation t(4;15)(q27;q11.2) associated with Prader-Willi syndrome
Birgitt Schüle, Mohammed Albalwi, Emma Northrop, David I Francis, Margaret Rowell, Howard R Slater, RJ McKinlay Gardner, Uta Francke
BMC Medical Genetics , 2005, DOI: 10.1186/1471-2350-6-18
Abstract: We used metaphase FISH to narrow the breakpoint region and molecular analyses to map the breakpoints on both chromosomes at the nucleotide level. The expression of genes on chromosome 15 on both sides of the breakpoint was determined by RT-PCR analyses.Pertinent clinical features include neonatal hypotonia with feeding difficulties, hypogonadism, short stature, late-onset obesity, learning difficulties, abnormal social behavior and marked tolerance to pain, as well as sticky saliva and narcolepsy. Relative macrocephaly and facial features are not typical for PWS. The translocation breakpoints were identified within SNRPN intron 17 and intron 10 of a spliced non-coding transcript in band 4q27. LINE and SINE sequences at the exchange points may have contributed to the translocation event. By RT-PCR of lymphoblasts and fibroblasts, we find that upstream SNURF/SNRPN exons and snoRNAs HBII-437 and HBII-13 are expressed, but the downstream snoRNAs PWCR1/HBII-85 and HBII-438A/B snoRNAs are not.As part of the PWCR1/HBII-85 snoRNA cluster is highly conserved between human and mice, while no copy of HBII-438 has been found in mouse, we conclude that PWCR1/HBII-85 snoRNAs is likely to play a major role in the PWS- phenotype.Prader-Willi syndrome (PWS) is a complex neurodevelopmental disorder and a classic example for genomic imprinting in humans. The incidence is about 1 in 10–20,000, and the clinical manifestations include decreased fetal activity, neonatal hypotonia, neonatal feeding difficulties, hyperphagia with obesity, hypogonadism, short stature, small hands and feet, characteristic facial features, and mild to moderate mental retardation. Diagnostic criteria have been proposed [1] and revised recently [2].About 70% of individuals clinically diagnosed with PWS have a ~4 Mb interstitial deletion at 15q11-13 of paternal origin, with clustered breakpoints (BP) at either of two proximal sites (BP1 or BP2) and one on the distal site (BP3) (Fig. 1a). The majority of the remai
Use of metformin during pregnancy for women with polycystic ovary syndrome  [PDF]
Howard Fan
Open Journal of Obstetrics and Gynecology (OJOG) , 2013, DOI: 10.4236/ojog.2013.31021
Abstract:

Objective: The purpose of this article is to review the literature assessing foetal and maternal pregnancy outcomes in women with PCOS who used metformin during pregnancy. Study Design: A literature search was conducted using MEDLINE, with analysis of 25 studies that recorded neonatal and maternal outcomes in women who used metformin during pregnancy. The outcomes assessed in this review include congenital deformities, miscarriages, preterm labour, gestational diabetes (GDM) and pregnancy induced hypertension (PIH). Results: We found that the use of metformin throughout pregnancy correlated with decreased rates of preterm labour, GDM and PIH. Conflicting evidence exists over whether metformin use during pregnancy reduced miscarriage rates. The use of metformin during pregnancy did not increase teratogenicity risks. Conclusion: The use of metformin throughout pregnancy is associated with decreased rates of preterm labour, GDM, and PIH. However, more randomised controls involving larger numbers of participants are required for more definitive results.

The cytokine hypothesis: A neurodevelopmental explanation for the emergence of schizophrenia later in life  [PDF]
Julia Howard
Advances in Bioscience and Biotechnology (ABB) , 2013, DOI: 10.4236/abb.2013.48A2011
Abstract:

There is increasing evidence for the cytokine hypothesis, which states that exposure to elevated cytokines in utero due to maternal immune activation is a major risk factor for the development of schizophrenia later in life. This is supported by numerous epidemicologic studies that connect multiple infections with schizophrenia emergence. Furthermore, cytokines are critically involved in early neurodevelopment and deviations from the norm can result in abnormal neuroanatomy and brain chemistry. Animal models of schizophrenia also support the critical role of developmental neuroinflammation in predisposing the brain to anatomical and behavioral abnormalities. Although there is strong evidence for the critical role of cytokines, they most likely work with other contributing risk factors such as genetic predisposition. New evidence indicates that cytokine exposure in utero may prime the brain and that a second stressor during adolescence, referred to as a second hit, may activate existing developmental vulnerabilities resulting in the emergence of clinical schizophrenia. Further knowledge of these pathogenic processes and risk factors could be very instrumental in reducing risk and slowing emergence of schizophrenia.

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