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Search Results: 1 - 8 of 8 matches for " Gerová "
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Angelite: Paziteli na vhoda
Gerov Georgi
Zbornik Radova Vizantolo?kog Instituta , 2009, DOI: 10.2298/zrvi0946435g
Abstract: (bugarski) Statijata prosledjava ikonografskata evoljucija na izobra enijata na arhangelite Mihail i Gavriil, pomesteni pri vhoda na pravoslavnija hram. Povraten moment v neja e 13 vek. Togava arhangel Mihail zapo va da se izobrazjava kato voin. S tova apotropejnite mu funkcii namirat adekvaten vizualen izraz. K'm kraja na stoletieto arhangel Gavriil zapo va da se izobrazjava kato pisar - ikonografija, kojato e b'de dorazvita i utv'rdena prez 14 vek. Prez postvizantijskata epoha v obraza na Mihail se pojavjavat elementi, koito akcentirat v'rhu roljata mu na psihopomp.
La nouvelle-métropole (l’église Saint-Stephane) à Nessébar: Résultats d’une recherche
Gerov Georgi
Zograf , 2012, DOI: 10.2298/zog1236075g
Abstract: This paper presents the most important conclusions regarding the architecture and wall paintings of the New Metropolis (Church of St. Stephen) in Nesebar, adopted during the recent restoration works in this church, which were financed by the Leventis Foundation.
Hypotrophy of conduit artery walls of the offspring of nitric oxide-defective rats
Kristek, F.;Gerová, M.;
Brazilian Journal of Medical and Biological Research , 2004, DOI: 10.1590/S0100-879X2004000400018
Abstract: the objective of the present study was to investigate the structure of the arterial walls of the offspring stemming from nitric oxide (no)-defective hypertensive parents. the parents were treated with ng-nitro-l-arginine methyl ester (40 mg kg-1 day-1) for 5 weeks. blood pressure was measured noninvasively in six 30-day-old rats and nine age-matched controls. the cardiovascular system was perfused with glutaraldehyde at 120 mmhg. the thoracic aorta and carotid artery were processed for electron microscopy, and geometry was determined by light microscopy. endothelial cells, smooth muscle cells (smc) and extracellular matrix (ecm) were determined by the point counting method in electron micrographs of the carotid artery. the blood pressure of experimental offspring was 150.0 ± 2.3 vs 104.6 ± 2.1 mmhg (p < 0.01) for the controls and their heart/body weight ratio of 3.9 ± 0.1 vs 4.4 ± 0.2 (p < 0.05) for the controls indicated cardiac hypotrophy. the wall thickness (tunica intima and media) of the thoracic aorta and carotid artery of experimental offspring was decreased to 78.9% (p < 0.01) and 83.8% (p < 0.01), respectively, compared to controls, as confirmed by a respective cross-sectional area of 85.3% (p < 0.01) and 84.1% (p < 0.01). the wall thickness/inner diameter ratio was reduced to 75% (p < 0.01) in the thoracic artery and to 81.5% (p < 0.01) in the carotid artery. no change in endothelial cell volume density or ecm was observed in the tunica intima of the carotid artery, and smc volume density was lower in the tunica media (37.6 ± 0.9 vs 44.7 ± 1.1% for controls, p < 0.01), indicating compromised smc development. interference with arginine metabolism, a decrease in no, and other factors are possible mechanisms underlying the structural alterations of the cardiovascular system of offspring from no-defective hypertensive rats.
Hypotrophy of conduit artery walls of the offspring of nitric oxide-defective rats
Kristek F.,Gerová M.
Brazilian Journal of Medical and Biological Research , 2004,
Abstract: The objective of the present study was to investigate the structure of the arterial walls of the offspring stemming from nitric oxide (NO)-defective hypertensive parents. The parents were treated with N G-nitro-L-arginine methyl ester (40 mg kg-1 day-1) for 5 weeks. Blood pressure was measured noninvasively in six 30-day-old rats and nine age-matched controls. The cardiovascular system was perfused with glutaraldehyde at 120 mmHg. The thoracic aorta and carotid artery were processed for electron microscopy, and geometry was determined by light microscopy. Endothelial cells, smooth muscle cells (SMC) and extracellular matrix (ECM) were determined by the point counting method in electron micrographs of the carotid artery. The blood pressure of experimental offspring was 150.0 ± 2.3 vs 104.6 ± 2.1 mmHg (P < 0.01) for the controls and their heart/body weight ratio of 3.9 ± 0.1 vs 4.4 ± 0.2 (P < 0.05) for the controls indicated cardiac hypotrophy. The wall thickness (tunica intima and media) of the thoracic aorta and carotid artery of experimental offspring was decreased to 78.9% (P < 0.01) and 83.8% (P < 0.01), respectively, compared to controls, as confirmed by a respective cross-sectional area of 85.3% (P < 0.01) and 84.1% (P < 0.01). The wall thickness/inner diameter ratio was reduced to 75% (P < 0.01) in the thoracic artery and to 81.5% (P < 0.01) in the carotid artery. No change in endothelial cell volume density or ECM was observed in the tunica intima of the carotid artery, and SMC volume density was lower in the tunica media (37.6 ± 0.9 vs 44.7 ± 1.1% for controls, P < 0.01), indicating compromised SMC development. Interference with arginine metabolism, a decrease in NO, and other factors are possible mechanisms underlying the structural alterations of the cardiovascular system of offspring from NO-defective hypertensive rats.
The possibilities of heat pumps utilisation for family houses and flats fumigation
Ján Pinka,Beáta Jágerová
Acta Montanistica Slovaca , 2006,
Abstract: Heat pumps (HPs) with the help of electricity use a renewable energy source to supply heat for homes or industrial buildings and to heat tap water. HP is a heating unit that will provide us with heat for our home for some 20 to 30 years to come and has a potential to replace traditional heating systems powered by gas, oil or coal. At this time, there is no other heating system that supplies clean heat with the help of up to 80 per cent of the renewable solar energy during all year.
Acetylcholine and bradykinin enhance hypotension and affect the function of remodeled conduit arteries in SHR and SHR treated with nitric oxide donors
Gerová, M.;Kristek, F.;Cacányiová, S.;Cebová, M.;
Brazilian Journal of Medical and Biological Research , 2005, DOI: 10.1590/S0100-879X2005000600019
Abstract: discrepancy was found between enhanced hypotension and attenuated relaxation of conduit arteries in response to acetylcholine (ach) and bradykinin (bk) in nitric oxide (no)-deficient hypertension. the question is whether a similar phenomenon occurs in spontaneously hypertensive rats (shr) with a different pathogenesis. wistar rats, shr, and shr treated with no donors [molsidomine (50 mg/kg) or pentaerythritol tetranitrate (100 mg/kg), twice a day, by gavage] were studied. after 6 weeks of treatment systolic blood pressure (bp) was increased significantly in experimental groups. under anesthesia, the carotid artery was cannulated for bp recording and the jugular vein for drug administration. the iliac artery was used for in vitro studies and determination of geometry. compared to control, shr showed a significantly enhanced (p < 0.01) hypotensive response to ach (1 and 10 μg, 87.9 ± 6.9 and 108.1 ± 5.1 vs 35.9 ± 4.7 and 64.0 ± 3.3 mmhg), and bk (100 μg, 106.7 ± 8.3 vs 53.3 ± 5.2 mmhg). shr receiving no donors yielded similar results. in contrast, maximum relaxation of the iliac artery in response to ach was attenuated in shr (12.1 ± 3.6 vs 74.2 ± 8.6% in controls, p < 0.01). iliac artery inner diameter also increased (680 ± 46 vs 828 ± 28 μm in controls, p < 0.01). wall thickness, wall cross-section area, wall thickness/inner diameter ratio increased significantly (p < 0.01). no differences were found in this respect among shr and shr treated with no donors. these findings demonstrated enhanced hypotension and attenuated relaxation of the conduit artery in response to no activators in shr and in shr treated with no donors, a response similar to that found in no-deficient hypertension.
Underground coal gasification and the ways of their admission
Ján Pinka,Beáta Jágerová,Mariana Miho?ová
Acta Montanistica Slovaca , 2006,
Abstract: Underground Coal Gasification (UCG) is the conversion of coal in the seam into a combustible gas. UCG is conceptually very simple but controlling the reaction and producing a consistent gas quality under a variety of geological and coal conditions is difficult to achieve. The basic concept has two boreholes, one for the injection of oxidants and the other for the removal of the product gas.UCG requires boreholes to access the coal, and three methods have been developed to connect them, namely:1. Air pressurisation between vertical holes2. Man – built galleries in the coal3. Directional drilling in the coal seam with a controlled injection.The most utilised method is the directional drilling in coal. This technology is well advanced in the hydrocarbons industry. The conventional steerable downhole motors are used for this purpose.
Acetylcholine and bradykinin enhance hypotension and affect the function of remodeled conduit arteries in SHR and SHR treated with nitric oxide donors
Gerová M.,Kristek F.,Cacányiová S.,Cebová M.
Brazilian Journal of Medical and Biological Research , 2005,
Abstract: Discrepancy was found between enhanced hypotension and attenuated relaxation of conduit arteries in response to acetylcholine (ACh) and bradykinin (BK) in nitric oxide (NO)-deficient hypertension. The question is whether a similar phenomenon occurs in spontaneously hypertensive rats (SHR) with a different pathogenesis. Wistar rats, SHR, and SHR treated with NO donors [molsidomine (50 mg/kg) or pentaerythritol tetranitrate (100 mg/kg), twice a day, by gavage] were studied. After 6 weeks of treatment systolic blood pressure (BP) was increased significantly in experimental groups. Under anesthesia, the carotid artery was cannulated for BP recording and the jugular vein for drug administration. The iliac artery was used for in vitro studies and determination of geometry. Compared to control, SHR showed a significantly enhanced (P < 0.01) hypotensive response to ACh (1 and 10 μg, 87.9 ± 6.9 and 108.1 ± 5.1 vs 35.9 ± 4.7 and 64.0 ± 3.3 mmHg), and BK (100 μg, 106.7 ± 8.3 vs 53.3 ± 5.2 mmHg). SHR receiving NO donors yielded similar results. In contrast, maximum relaxation of the iliac artery in response to ACh was attenuated in SHR (12.1 ± 3.6 vs 74.2 ± 8.6% in controls, P < 0.01). Iliac artery inner diameter also increased (680 ± 46 vs 828 ± 28 μm in controls, P < 0.01). Wall thickness, wall cross-section area, wall thickness/inner diameter ratio increased significantly (P < 0.01). No differences were found in this respect among SHR and SHR treated with NO donors. These findings demonstrated enhanced hypotension and attenuated relaxation of the conduit artery in response to NO activators in SHR and in SHR treated with NO donors, a response similar to that found in NO-deficient hypertension.
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