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Search Results: 1 - 10 of 191367 matches for " G. Kaur "
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Nevomelanocytic nevus with leukotrichia
Kaur C,Thami G,Kaur S
Indian Journal of Dermatology, Venereology and Leprology , 2003,
Abstract: Nevomelanocytic nevi exhibit clinical variations in morphology, location, texture and number related to age, race and geographical distribution. Development of age related greying of hair over pigmented melanocytic nevus is being discussed.
Glutamate excitotoxicity: Its correlation with neuronal plasticity in young adults and neonate rats.
Kaur G,Basu A,Kaur AP
Journal of Neurological Sciences , 2003,
Abstract: Although axonal growth in the adult mammalian brain is primarily hampered by adverse environmental conditions, the success of regenerative process is dependent on the capability of injured neurons to express the intrinsic molecular machinery required for neurite elongation. In the present study, regional expression of proteins associated with neuronal and glial growth, such as neural cell adhesion molecule (NCAM), polysialylated neural cell adhesion molecule (PSA-NCAM), Growth-associated protein-43 (GAP-43), Glial fibrillary acidic protein (GFAP) and synaptophysin were studied by immunoblotting technique from different brain regions of monosodium glutamate treated neonate rat pups and young adult rats. The animals were sacrificed after 3 hrs of monosodium glutamate treatment in neonates and 45 days in young adult rats. Control animals were injected with saline alone. NCAM, PSA-NCAM, GFAP and GAP-43 levels were significantly increased in majority of brain regions studied both in neonate and adult group of rats. More pronounced increase in the expression of these markers of neuronal plasticity was observed in the neonate rat pups as compared to young adult animals after monosodium glutamate induced excitotoxicity. These findings support the hypothesis that there is a temporary recapitulation of regenerative state by brain following monosodium glutamate induced CNS injury. Understanding and enhancement of such a response may be one therapeutic strategy in treating CNS injury.
Urticarial vasculitis in infancy
Kaur S,Thami G
Indian Journal of Dermatology, Venereology and Leprology , 2003,
Abstract: Urticarial vasculitis is an uncommon manifestation of cutaneous vasculitis closely resembling chronic urticaria. It is an immune complex deposition disorder, which is not commonly observed in children. We report an 9-month-old infant with urticarial vasculitis and discuss its clinical course and differentiation from common urticaria.
Fortification of Transport Layer Security Protocol with Hashed Fingerprint Identity Parameter
Kuljeet Kaur,G. Geetha
International Journal of Computer Science Issues , 2012,
Abstract: Identity over the public links becomes quiet complex as Client and Server needs proper access rights with authentication. For determining clients identity with password Secured Shell Protocol or Public Key Infrastructure is deployed by various organizations. For end to end transport security SSL (Secured Socket Layer) is the de facto standard having Record and Handshake protocol dealing with data integrity and data security respectively. It seems secure but many risks lurk in its use. So focus of the paper would be formulating the steps to be used for the enhancement of SSL. One more tier of security to the transport layer security protocol is added in this research paper by using fingerprints for identity authentication along with password for enhancement of SSL. Bio Hashing which will be done with the help of Minutiae Points at the fingerprints would be used for mutual authentication. New hash algorithm RNA-FINNT is generated in this research paper for converting minutiae points into hashed code. Value of hashed code would be stored at the Database in the Multi Server environment of an organization. Research paper will perform mutual authentication in the multi server environment of an organization with the use of fingerprint and password both as identity authentication parameters. This will strengthen record and handshake protocol which will enhance SSL and further enhancement of SSL will result in the fortification of Transport Layer Security Protocol.
Split hand-foot malformation: a congenital central limb ray deficiency.
Thami G,Kaur S
Journal of Postgraduate Medicine , 2002,
Abstract:
Idiopathic Hypogonadotropic Hypogonadism— An Update on the Aetiopathogenesis, Management of IHH in Both Males and Females—An Exhaustive Review  [PDF]
Kochar Kaur Kulvinder, G. N. Allahbadia, M. Singh
Advances in Sexual Medicine (ASM) , 2016, DOI: 10.4236/asm.2016.64007
Abstract: Methods: Asystematic literature search was performed using PUBMED for all English articles up to April 2014. Although this review mainly focuses on published human studies, it also draws attention to where future research should be directed based on animal studies. Results: Besides the 9 known mutations widely quoted for KS namely KAL1, Fibroblast growth factor 8 (FGF8), fibroblast growth factor receptor 1 (FGFR1), prokineticin 2 (PROK2), PROK receptor 2 (PROKR2), WDR11, heparin sulfate-6-O-Transferase (HS6T1), chromodomain helicase DNA binding protein 7 (CHD7) and semaphorin 3A (SEMA 3A), additional mutations in “FGF8 synexpression” group e.g., FGF 17, ILRD, DUSP 6, SPRY4 and FLRT3 have been shown to be involved in CHH, mostly KS besides SEMA 7A. Although traditionally division has been based on anosmic/normosnic criteria, further genes found to cause so called nIHH like Gonadotropin releasing hormone receptor (GNRHR). KISS1, TAC3, TACR3 have also been found to be associated with hyposmia on detailed testing on UPSIT and MRI for olfactory structures revealed absent OB. Further detailed examination of transcription factor genes have revealed involvement of HESX1, TSHZ1, AXL, SOX10 with a strong overlap of in transcription factors in development of septooptic dysplasia (SOD), combined pituitary hormone deficiency (CHPD) and KS. Treatment with rFSH/-hCG gives almost similar results to pulsatile GnRH therapy and should be based on cost factor, availability and in occasional cases specific treatment like kisspeptin therapy. Conclusions: Contrary to the traditional thinking, one shoud reconsider classifying cases of IHH simply on basis of anosmia/normosmia. Deafness calls for looking for mutations in Sox 10/CHD7/ILRD7 considering 38% association of former. Therapy should be individualized based on availability of pulsatile GnRH, cost factor and in recalcitrant cases kp therapy may be of use with kp mutations and NKB mutations.
Wei Hua's Four Parameter Potential Comments and Computation of Moleculer Constants α_e and ω_e x_e
Sarvpreet Kaur,C. G. Mahajan
Physics , 1998, DOI: 10.1007/BF02830107
Abstract: The value of adjustable parameter $C$ and the four-parameter potential $U(r) = D_{e}\left [ \frac{1-{exp}[-b(r-r_{e})]}{1-C{exp} [-b(r-r_{e})]} \right ]^{2}$ has been expressed in terms of molecular parameters and its significance has been brought out. The potential so constructed, with $C$ derived from the molecular parameters, has been applied to ten electronic states in addition to the states studied by Wei Hua. Average mean deviation has been found to be 3.47 as compared to 6.93, 6.95 and 9.72 obtained from Levine2, Varshni and Morse potentials, respectively. Also Dunham's method has been used to express rotation-vibration interaction constant $(\alpha_{e})$ and anharmonocity constant $(\omega_{e}x_{e})$ in terms of $C$ and other molecular constants. These relations have been employed to determine these quantities for 37 electronic states. For $\alpha_{e}$, the average mean deviation is 7.2% compared to 19.7% for Lippincott's potential which is known to be the best to predict the values. Average mean deviation for $(\omega_{e}x_{e})$ turns out to be 17.4% which is almost the same as found from Lippincott's potential function.
The effect of prostaglandin synthase inhibitor, aspirin on the rat intestinal membrane structure and function
Kaur,G.; Kaur,J.; Mittal,N.; Nath Sanyal,S.;
Nutrición Hospitalaria , 2010,
Abstract: aspirin at a dose of 50 mg/kg body weight was found to decrease the activity of the rat intestinal brush border membrane (bbm) - associated enzymes such as the sucrase, lactase, maltase and alkaline phosphatase. aspirin treatment also led to a decrease in the microviscosity in the native as well as the benzyl alcohol treated membrane which might be due to the lipid peroxidative damage in the membrane. physical correlation of the membrane oxidative damage was evident as the fourier transformation infra red (ftir) study of the aspirin treated membrane, which include an increased proportion of gauche to trans conformer, shift in the methylene c-h asymmetric and symmetric stretching frequencies, c = o double bond stretching, nh bending, antisymmetric (n)-ch3 bending, c-n stretching and antisymmetric cnc stretching while there was no change in the ch2 wagging and twisting as well as in nh-bending amide bond i and ii. aspirin treatment also caused an alteration in the glucose and histidine transport, as evident by a decreased vmax value while the apparent km remaining unchanged in the control and aspirin-treated animals confirming that there was no change in the substrate affinity constant of the membrane transport proteins for the glucose and the basic amino acid, although the rate of transport decreased considerably. there was a decrease noted in the energy of activation of glucose and histidine transport when studied at different temperature but no change in the temperature of phase transition in the bbm with aspirin treatment, thus implying that perhaps the thermotropic phase transition in the membrane may have relatively little effect on the transport processes. the result suggests an underlying molecular mechanism indicating the implied membrane damage by aspirin, an important member of the non-steroidal antiinflammatory drug (nsaid) family which could possibly through an oxidative damage may lead to an altered molecular structure, physical state and biological func
Lichen Aureus : Atypcial Presentation Showing Remarkable Therapeutic Response
Kaur Charandeep,kaur Sukhjot,Thami G . P,Kanwar A . J
Indian Journal of Dermatology , 2002,
Abstract: A typical patient with zosteriform distribution of rust-coloured to yellowish brown patches over the right leg is reported. The histopathology was consistent with lichen aureus.
The effect of prostaglandin synthase inhibitor, aspirin on the rat intestinal membrane structure and function El efecto del inhibidor de la sintasa de prostaglandina, aspirina, sobre la estructura y función de la membrana intestinal de la rata
G. Kaur,J. Kaur,N. Mittal,S. Nath Sanyal
Nutrición Hospitalaria , 2010,
Abstract: Aspirin at a dose of 50 mg/kg body weight was found to decrease the activity of the rat intestinal brush border membrane (BBM) - associated enzymes such as the sucrase, lactase, maltase and alkaline phosphatase. Aspirin treatment also led to a decrease in the microviscosity in the native as well as the benzyl alcohol treated membrane which might be due to the lipid peroxidative damage in the membrane. Physical correlation of the membrane oxidative damage was evident as the Fourier Transformation Infra Red (FTIR) study of the Aspirin treated membrane, which include an increased proportion of gauche to trans conformer, shift in the methylene C-H asymmetric and symmetric stretching frequencies, C = O double bond stretching, NH bending, antisymmetric (N)-CH3 bending, C-N stretching and antisymmetric CNC stretching while there was no change in the CH2 wagging and twisting as well as in NH-bending amide bond I and II. Aspirin treatment also caused an alteration in the glucose and histidine transport, as evident by a decreased Vmax value while the apparent Km remaining unchanged in the control and Aspirin-treated animals confirming that there was no change in the substrate affinity constant of the membrane transport proteins for the glucose and the basic amino acid, although the rate of transport decreased considerably. There was a decrease noted in the energy of activation of glucose and histidine transport when studied at different temperature but no change in the temperature of phase transition in the BBM with Aspirin treatment, thus implying that perhaps the thermotropic phase transition in the membrane may have relatively little effect on the transport processes. The result suggests an underlying molecular mechanism indicating the implied membrane damage by Aspirin, an important member of the non-steroidal antiinflammatory drug (NSAID) family which could possibly through an oxidative damage may lead to an altered molecular structure, physical state and biological functions of the intestinal membrane. Se encontró que la aspirina a una dosis de 50 mg/kg de peso corporal disminuye la actividad de las enzimas asociadas a la membrana con borde en cepillo (MBC) del intestino de la rata como la sucrasa, lactasa, maltasa y fosfata alcalina. El tratamiento con aspirina también produjo una disminución de la microviscosidad en la membrana nativa así como en la membrana tratada con alcohol bencílico, lo que podría deberse a la lesión de peroxidación lipídica de la membrana. La correlación física de la lesión oxidativa de la membrana fue evidente como mostró el estudio
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