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Search Results: 1 - 10 of 9035 matches for " Farah Michel Eid "
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Inje??o intravítrea de acetato de triancinolona no tratamento da síndrome de Vogt-Koyanagi-Harada
Andrade, Rafael Ernane Almeida;Muccioli, Cristina;Farah, Michel Eid;
Arquivos Brasileiros de Oftalmologia , 2004, DOI: 10.1590/S0004-27492004000300005
Abstract: purpose: to report the use of intravitreal injection of triamcinolone acetonide in the acute phase of vogt-koyanagi-harada's disease. methods: nine eyes from five patients in the acute phase of vogt-koyanagi-harada's disease with serous retinal detachments were treated with a single 4-mg intravitreal injection of triamcinolone acetonide. the following parameters were evaluated: visual acuity, intraocular pressure, as well as the height of the serous retinal detachment using optical coherence tomography. results: optical coherence tomography images showed a marked decrease in the retinal detachment in the first week after the injection with subsequent return to normal retinal thickness in all eyes. follow-up ranged from 5 to 12 months with a mean of 7.8 months. no complications were observed. conclusions: intravitreal triamcinolone acetonide provides short-term improvement in visual acuity and serous retinal detachments associated with vogt-koyanagi-harada's disease. these findings should be followed by further studies to evaluate long-term effects.
Pigmentos maculares
Canovas, Renata;Cypel, Marcela;Farah, Michel Eid;Belfort Jr, Rubens;
Arquivos Brasileiros de Oftalmologia , 2009, DOI: 10.1590/S0004-27492009000600021
Abstract: lutein and zeaxanthin are yellow pigments located at the macula. because of your location macular pigments decrease and filter the amount of blue light that reach photoreceptors, protect the outer retina from oxidative stress and may improve the vision quality. this is a review regarding incorporation mechanism, function and knowledge update.
Transscleral diode laser retinopexy in retinal reattachment surgery
Gon?alves, Jo?o Carlos de Miranda;Farah, Michel Eid;
Arquivos Brasileiros de Oftalmologia , 2004, DOI: 10.1590/S0004-27492004000100004
Abstract: purpose: transscleral diode retinal photocoagulation (diopexy) is becoming an accepted technique in the treatment of selected retinal diseases. the objective of this study is to evaluate diopexy technique in the production of adhesive chorioretinal lesions during the surgical treatment of the rhegmatogenous retinal detachment. methods: 25 patients with rhegmatogenous retinal detachment were enrolled in a prospective clinical-surgical study to evaluate the technique of transscleral diode laser photocoagulation to obtain adhesive chorioretinal lesions during retinal reattachment surgery. the surgery consisted of the placement of an exoplant silicon to produce a buckle effect combined with a drainage of subretinal fluid in most cases. results: by a mean follow-up of 10 months, 21 of 25 eyes had their retinas reattached after only one surgery with diopexy used in all cases. conclusion: transscleral diode laser photocoagulation was a technically easy, controlled, effective, reproducible and safe means of obtaining chorioretinal adhesion in retinal reattachment surgery.
Heterogeneidade genética em atrofia óptica autoss?mica dominante
Sallum, Juliana Maria Ferraz;Farah, Michel Eid;Maumenee, Irene Hussels;
Arquivos Brasileiros de Oftalmologia , 2002, DOI: 10.1590/S0004-27492002000400005
Abstract: purpose: autosomal dominant optic atrophy is a hereditary optic neuropathy characterized by progressive visual loss in childhood, color vision anomalies, visual field defects and temporal pallor of the optic disc. this disease has been mapped to a 1.4 cm interval in chromosome 3q28-29 between markers d3s3669 and d3s3562. one family was mapped to chromosome 18q12.2-12.3. linkage analysis in three families with autosomal dominant optic atrophy with polymorphic dna markers for chromosome 3q28-29 and 18q12.2-12.3. methods: 57 individuals from three families underwent ophthalmological examination. genomic dna was extracted from blood samples. linkage analysis was performed between the disease and 11 polymorphic markers around 3q28-qter and 18q12.2-12.3. polymerase chain reaction (pcr) fragments sizes were identified in a scanner gel using a 373 dna sequencer. these numbers were used as alleles for pedigree analysis. the lod scores were calculated using the mlink program. results: all three families presented optic atrophy with autosomal dominant pattern of inheritance, variable expression and high penetrance. two families were linked to 3q28-29 markers. a maximal lod score of 3.56, at a recombination fraction of zero, was obtained using the marker d3s3669 in one family. the linkage area was defined in a 2 cm interval by haplotype analysis between markers d3s2418 and d3s1305, because patients iii.4 and iii.14 showed crossing-overs. the third family was not linked to 3q28-29 neither to 18q12.2-12.3. conclusions: there is genetic heterogeneity in autosomal dominant optic atrophy, because the third family did not map to any known locus. and a third locus for this disease may exist.
Classifica??o diagnóstica dos portadores de doen?as degenerativas de retina, integrantes dos grupos Retina S?o Paulo e Retina Vale do Paraíba
Unonius, Nichard;Farah, Michel Eid;Sallum, Juliana M. Ferraz;
Arquivos Brasileiros de Oftalmologia , 2003, DOI: 10.1590/S0004-27492003000400009
Abstract: purpose: to organize a regional data bank of all individuals that have retinal degenerative diseases, with the aim to classify each patient according to the type of distrophy and pattern of inheritance. methods: during the meeting of the s?o paulo retina group on may 5th, 2001, two hundred and forty-three persons were registered, part of whom provided information concerning ocular, personal and family history and family tree. ninety-three patients were asked about age, origin, type of dystrophy, family history and family tree information, type of inheritance, other systemic abnormalities and complementary examination. they were classified according to the diagnosis and pattern of inhe-ritance. results: the distrophies found in the registered two hundred and forty-three patients, were: retinitis pigmentosa, stargardt disease, usher syndrome, leber congenital amaurosis and choroideremia. of the ninety-three patients examined on the same day, sixty-two had retinitis pigmentosa, thirteen had stargardt disease, thirteen had usher syndrome, three had leber congenital amaurosis and two had choroideremia. the inheritance pattern of the patients with retinitis pigmentosa was autosomal dominant in 4 cases (7%), autosomal recessive in twenty cases (32%), x-linked recessive in 7 cases (11%). twenty-nine cases were isolated (47%) and two had an indeterminate pattern of inheritance (3%). of the stargardt disease patients, three (23%) were autosomal recessive and ten (77%) were isolated cases. of the thirteen patients with usher syndrome, eight (61.5%) were autosomal recessive, four (31%) were isolated cases and one (7.5%) did not have a determined inheritance pattern. the two patients with choroideremia were x-linked recessive. in leber congenital amaurosis one (33.5%) was autosomal recessive and two (66.5%) were isolated cases. conclusion: this study highlights the importance of this classification as being the first reference of inheritance patterns of retinal distrophies in ou
Transscleral diode laser retinopexy in retinal reattachment surgery
Gon?alves Jo?o Carlos de Miranda,Farah Michel Eid
Arquivos Brasileiros de Oftalmologia , 2004,
Abstract: PURPOSE: Transscleral diode retinal photocoagulation (diopexy) is becoming an accepted technique in the treatment of selected retinal diseases. The objective of this study is to evaluate diopexy technique in the production of adhesive chorioretinal lesions during the surgical treatment of the rhegmatogenous retinal detachment. METHODS: 25 patients with rhegmatogenous retinal detachment were enrolled in a prospective clinical-surgical study to evaluate the technique of transscleral diode laser photocoagulation to obtain adhesive chorioretinal lesions during retinal reattachment surgery. The surgery consisted of the placement of an exoplant silicon to produce a buckle effect combined with a drainage of subretinal fluid in most cases. RESULTS: By a mean follow-up of 10 months, 21 of 25 eyes had their retinas reattached after only one surgery with diopexy used in all cases. CONCLUSION: Transscleral diode laser photocoagulation was a technically easy, controlled, effective, reproducible and safe means of obtaining chorioretinal adhesion in retinal reattachment surgery.
Classifica o diagnóstica dos portadores de doen as degenerativas de retina, integrantes dos grupos Retina S o Paulo e Retina Vale do Paraíba
Unonius Nichard,Farah Michel Eid,Sallum Juliana M. Ferraz
Arquivos Brasileiros de Oftalmologia , 2003,
Abstract: OBJETIVO:Organizar um banco de dados regional de todos os indivíduos portadores de doen as degenerativas da retina, com o objetivo de classificar cada paciente de acordo com o tipo de distrofia e padr o de heran a. MéTODOS: Durante o encontro do Grupo Retina S o Paulo no dia 5 de maio de 2001, duzentas e quarenta e três pessoas foram registradas, sendo que parte forneceu dados de antecedentes oculares, pessoais e familiares e árvore genealógica. Noventa e três pacientes foram questionados quanto a idade, origem, tipo de distrofia, história familiar e árvore genealógica, tipo de heran a, outras anomalias sistêmicas e exames complementares. Foram classificados quanto ao diagnóstico e padr o de heran a. RESULTADOS: Dos duzentos e quarenta e três pacientes registrados, as distrofias encontradas foram retinose pigmentária, doen a de Stargardt, síndrome de Usher, amaurose congênita de Leber e coroideremia. Quanto à divis o por doen a dos 93 pacientes argüidos, havia 62 pacientes com retinose pigmentária, 13 com doen a de Stargardt, 13 com síndrome de Usher, três com amaurose congênita de Leber e dois com coroideremia. Dos pacientes com retinose pigmentária, o padr o de heran a detectado foi autoss mico dominante em quatro casos (7%), autoss mico recessivo em vinte casos (32%), ligado ao cromossomo X recessivo em sete casos (11%), caso isolado em vinte e nove (47%) e padr o indeterminado em dois (3%). Para a doen a de Stargardt três indivíduos (23%) seguiam o padr o de heran a autoss mico recessivo e dez (77%) eram casos isolados. Dos treze pacientes com síndrome de Usher, oito (61,5%) apresentavam heran a autoss mica recessiva, quatro (31%) eram casos isolados e um (7,5%) tinha o padr o de heran a indeterminado. Os dois pacientes com coroideremia seguiam o padr o de heran a ligado ao X recessivo. Para amaurose congênita de Leber, um paciente (33,5%) tinha padr o autoss mico recessivo de heran a e dois (66,5%) eram casos isolados. CONCLUS O: Destaca-se assim a importancia desta classifica o como a primeira referência nacional dos padr es de hereditariedade das distrofias retinianas do país. Este é o primeiro passo para se proceder em seguida a classifica o genético-molecular baseada no seqüenciamento de cada gene responsável por cada um dos padr es de heran a. A freqüência de cada tipo específico é semelhante à encontrada em outros trabalhos epidemiológicos de outros países.
Late-onset persistent retinal microvascular changes after bone marrow transplantation: 3-year follow-up
Muccioli Cristina,Belfort Jr Rubens,Jorge Rodrigo,Farah Michel Eid
Arquivos Brasileiros de Oftalmologia , 2002,
Abstract: Purpose: To describe a case of persistent retinopathy after bone marrow transplantation in the absence of radiation therapy. Methods: Case Report. Results: A 42 year-old man developed bilateral visual loss 15 months after receiving a bone marrow transplant for acute leukemia. The patient was treated with a high dose of cyclosporin A and oral corticosteroids. No radiation therapy was given. Late-onset, multiple, bilateral cotton-wool spots developed 15 months after the bone marrow transplantation and still persist. After three years other cotton-wool spots arose in the absence of any immunosuppressive therapy. Conclusions: Bone marrow transplantation microvasculopathy of the retina may be related to certain combinations of chemotherapy drugs or immunosuppression itself and may persist in the absence of these immunosuppressive drugs.
Heterogeneidade genética em atrofia óptica autoss mica dominante
Sallum Juliana Maria Ferraz,Farah Michel Eid,Maumenee Irene Hussels
Arquivos Brasileiros de Oftalmologia , 2002,
Abstract: Objetivos: A atrofia óptica autoss mica dominante, tipo Kjer ou juvenil, é neuropatia óptica hereditária que causa perda de acuidade visual, anormalidades da vis o de cores e defeitos do campo visual, caracterizada por palidez do disco óptico. O gene desta doen a foi mapeado por análise de liga o genética em um intervalo de 1,4 cM no cromossomo 3q28-29 entre os marcadores microssatélites D3S3669 e D3S3562. Embora a maioria das famílias estudadas tenha mostrado liga o para a regi o cromoss mica 3q28-29, uma família foi mapeada no cromossomo 18q12.2-12.3. Este trabalho analisa a liga o da atrofia óptica em três famílias com marcadores polimórficos para os cromossomos 3q28-29 e 18q12.2-12.3. Métodos: Cinqüenta e sete indivíduos de três famílias foram submetidos a exame oftalmológico e coleta de sangue. O DNA foi extraído e amplificado em rea es de polimerase em cadeia (PCR) com marcadores polimórficos para os cromossomos 3q28-29 e 18q12.2-12.3. Os fragmentos de PCR foram mensurados em seqüenciador automático (373 DNA sequencer). Estes números foram utilizados como alelos para análise de haplótipos. Os "lod scores" foram calculados pelo programa MLINK. Resultados: Na primeira família houve suspeita da atrofia óptica mapear para o cromossomo 3q28-29, mas sem significancia estatística no valor do "lod score". Na segunda família a atrofia óptica apresentou liga o para este locus. Os eventos de recombina o nesta família localizaram o gene num intervalo de 2 cM entre os marcadores D3S3669 e D3S2305. O "lod score" máximo obtido foi de 3,56 no theta de 0,00 com o marcador D3S3669. A terceira família n o apresentou liga o nos cromossomos 3q28-29 e 18q12.2-12.3. Conclus o: O fato da terceira família n o mapear para nenhum dos dois loci já descritos é indicativo de que existe heterogeneidade genética na atrofia óptica autoss mica dominante e levanta a possibilidade de existir um terceiro locus para esta doen a.
Inje o intravítrea de acetato de triancinolona no tratamento da síndrome de Vogt-Koyanagi-Harada
Andrade Rafael Ernane Almeida,Muccioli Cristina,Farah Michel Eid
Arquivos Brasileiros de Oftalmologia , 2004,
Abstract: OBJETIVO: Avaliar o uso da inje o intravítrea do acetato de triancinolona no tratamento da fase aguda da síndrome de Vogt-Koyanagi-Harada, demonstrando a rápida resolu o do descolamento seroso de retina. MéTODOS: Nove olhos de cinco pacientes apresentando descolamento seroso de retina associado à síndrome de Vogt-Koyanagi-Harada foram tratados com uma única inje o intravítrea de 4 mg de acetato triancinolona. Os seguintes parametros foram avaliados: melhor acuidade visual, press o intra-ocular e a altura do descolamento de acordo com a tomografia de coerência óptica. RESULTADOS: Em todos os olhos a tomografia de coerência óptica revelou diminui o marcada no descolamento seroso de retina na primeira semana após a inje o intravítrea do acetato de triancinolona, com subseqüente recupera o da acuidade visual e das características anat micas retinianas normais. N o foram observadas complica es durante o seguimento, que variou de 5 a 12 meses (média de 7,8 meses). CONCLUS ES: A inje o intravítrea do acetato de triancinolona pode proporcionar em curto tempo a resolu o do processo inflamatório e exsudativo intra e sub-retiniano na síndrome de Vogt-Koyanagi-Harada cursando com melhora da acuidade visual. S o necessários novos estudos para avaliar a eficácia e a seguran a deste tipo de procedimento a longo prazo.
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